Osteogenic differentiation of hypertrophic chondrocytes involves asymmetric cell divisions and apoptosis. - Wikidata - wikimedia - TriplyDB

Osteogenic differentiation of hypertrophic chondrocytes involves asymmetric cell divisions and apoptosis.

Osteogenic differentiation of hypertrophic chondrocytes involves asymmetric cell divisions and apoptosis.

http://www.wikidata.org/entity/Q42952953

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Articular cartilage and changes in arthritis. An introduction: cell biology of osteoarthritis A pathway to bone: signaling molecules and transcription factors involved in chondrocyte development and maturation The multifaceted role of the vasculature in endochondral fracture repair Control of apoptosis by asymmetric cell division Apoptotic cell death is not a widespread phenomenon in normal aging and osteoarthritis human articular knee cartilage: a study of proliferation, programmed cell death (apoptosis), and viability of chondrocytes in normal and osteoarthritic human knee Hypertrophic chondrocytes can become osteoblasts and osteocytes in endochondral bone formation.Substrate stiffness and oxygen as regulators of stem cell differentiation during skeletal tissue regeneration: a mechanobiological model.Chondrocytes transdifferentiate into osteoblasts in endochondral bone during development, postnatal growth and fracture healing in mice.Role of Fas and Treg cells in fracture healing as characterized in the fas-deficient (lpr) mouse model of lupus.Hypertrophic chondrocytes in the rabbit growth plate can proliferate and differentiate into osteogenic cells when capillary invasion is interposed by a membrane filter Dual pathways to endochondral osteoblasts: a novel chondrocyte-derived osteoprogenitor cell identified in hypertrophic cartilage Epiphyseal chondrocyte secondary ossification centers require thyroid hormone activation of Indian hedgehog and osterix signaling Synergistic effect of IGF-1 and OP-1 on matrix formation by normal and OA chondrocytes cultured in alginate beads.Endochondral Ossification Is Accelerated in Cholinesterase-Deficient Mice and in Avian Mesenchymal Micromass Cultures.Regulated production of mineralization-competent matrix vesicles in hypertrophic chondrocytes Defective endochondral ossification-derived matrix and bone cells alter the lymphopoietic niche in collagen X mouse models Apoptosis in bone physiology and disease.Comparative analysis of signaling pathways in peripheral blood from patients with Kashin-Beck disease and osteoarthritis.Abl expression, tumour grade, and apoptosis in chondrosarcoma.Bone circulatory disturbances in the development of spontaneous bacterial chondronecrosis with osteomyelitis: a translational model for the pathogenesis of femoral head necrosis.The chondrocytic journey in endochondral bone growth and skeletal dysplasia.Chondrocyte hypertrophy in skeletal development, growth, and disease.Collagenase 3 is a target of Cbfa1, a transcription factor of the runt gene family involved in bone formation.Fate of growth plate hypertrophic chondrocytes: death or lineage extension?Signaling pathways regulating cartilage growth plate formation and activity.Roles of Chondrocytes in Endochondral Bone Formation and Fracture Repair.Lessons on skeletal cell plasticity from studying jawbone regeneration in zebrafish.The use of rats and mice as animal models in ex vivo bone growth and development studies.A murine osteosarcoma cell line with a potential to develop ossification upon transplantation.Kinesin-1 promotes chondrocyte maintenance during skeletal morphogenesis.Fragmentation of the distal portion of Tomes' processes of secretory ameloblasts in the forming enamel of rat incisors.Differential requirements for IKKalpha and IKKbeta in the differentiation of primary human osteoarthritic chondrocytes.Altered cellular kinetics in growth plate according to alterations in weight bearing Chondrocytes derived from mouse embryonic stem cells.Phenotypic diversity of neoplastic chondrocytes and extracellular matrix gene expression in cartilaginous neoplasms.Localization of mRNAs and proteins in methyl methacrylate-embedded tissues.The role of apoptosis in mineralizing murine versus avian micromass culture systems.5-Aza-2'-deoxycytidine acts as a modulator of chondrocyte hypertrophy and maturation in chick caudal region chondrocytes in culture.Cell differentiation and matrix gene expression in mesenchymal chondrosarcomas Rapid new bone tissue remodeling during distraction osteogenesis is associated with apoptosis.

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P2860

Osteogenic differentiation of hypertrophic chondrocytes involves asymmetric cell divisions and apoptosis.

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Aigner T

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Erenpreisa J

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Roach HI

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P2860

Identification of programmed cell death in situ via specific labeling of nuclear DNA fragmentation

The fine structure of the proximal growth plate and metaphysis of the avian tibia: endochondral osteogenesis

Detection of DNA fragmentation in apoptosis: application of in situ nick translation to cell culture systems and tissue sections.

Symmetric and asymmetric cell division in rat corneal epithelium.

The origins of DNA breaks: a consequence of DNA damage, DNA repair, or apoptosis?

Apoptosis and the regulation of cell numbers in normal and neoplastic tissues: an overview.

Cartilage ultrastructure after high pressure freezing, freeze substitution, and low temperature embedding. I. Chondrocyte ultrastructure--implications for the theories of mineralization and vascular invasion.

Gene expression during osteogenic differentiation in mandibular condyles in vitro.

Mechanisms of asymmetric cell division: two Bs or not two Bs, that is the question.

Condensation of the chromatin at the membrane of an apoptotic nucleus is not associated with activation of an endonuclease.

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