Bladder outlet obstruction: progression from inflammation to fibrosis.
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Research Findings on Overactive BladderSuppression of the PI3K pathway in vivo reduces cystitis-induced bladder hypertrophy and restores bladder capacity examined by magnetic resonance imagingPhenotypic switching induced by damaged matrix is associated with DNA methyltransferase 3A (DNMT3A) activity and nuclear localization in smooth muscle cells (SMC)A mouse model of urofacial syndrome with dysfunctional urinationLL-37 induced cystitis and the receptor for advanced glycation end-products (RAGE) pathway.Increased detrusor collagen is associated with detrusor overactivity and decreased bladder compliance in men with benign prostatic obstruction.Bladder dysfunction in 2015: Novel findings continue to challenge researchers and clinicians.Obstruction-induced alterations within the urinary bladder and their role in the pathophysiology of lower urinary tract symptomatology.The urinary bladder of spontaneously hypertensive rat demonstrates bladder hypertrophy, inflammation, and fibrosis but not hyperplasiaMast Cell Accumulation and Degranulation in Rat Bladder with Partial Outlet Obstruction.Adjustable passive stiffness in mouse bladder: regulated by Rho kinase and elevated following partial bladder outlet obstructionBenefits of mesenchymal stem cells after partial bladder outlet obstruction.The NLRP3 Inflammasome Mediates Inflammation Produced by Bladder Outlet Obstruction.A murine model of inflammatory bladder disease: cathelicidin peptide induced bladder inflammation and treatment with sulfated polysaccharidesCharacterization of miRNA-regulated networks, hubs of signaling, and biomarkers in obstruction-induced bladder dysfunctionNecessity of routine histopathological evaluation subsequent to bladder neck contracture resection.TGF-β1 induces EMT reprogramming of porcine bladder urothelial cells into collagen producing fibroblasts-like cells in a Smad2/Smad3-dependent manner.Evaluation of the effects of omega-3 & interferon alpha-2b administration on partial bladder outlet obstruction in a rat model.From urgency to frequency: facts and controversies of TRPs in the lower urinary tract.Advanced therapeutic directions to treat the underactive bladder.Stem Cell Therapy: Current Applications and Potential for Urology.Inflammasomes in the urinary tract: a disease-based review.Management of Urethral Stricture and Bladder Neck Contracture Following Primary and Salvage Treatment of Prostate Cancer.Pathophysiology of the Underactive Bladder: Evolving New Concepts.A mucosal imprint left by prior Escherichia coli bladder infection sensitizes to recurrent disease.Alteration of Urothelial Inflammation, Apoptosis, and Junction Protein in Patients with Various Bladder Conditions and Storage Bladder Symptoms Suggest Common Pathway Involved in Underlying Pathophysiology.Proteomic profile of an acute partial bladder outlet obstruction.Role of PAR2 in the Development of Lower Urinary Tract Dysfunction.Deletion of neuropilin 2 enhances detrusor contractility following bladder outlet obstruction.Urodynamic improvements following oral medical therapy for partial bladder outlet obstruction in an animal model.Acute length adaptation and adjustable preload in the human detrusor.A pilot study to measure dynamic elasticity of the bladder during urodynamics.Benefits and limitations of animal models in partial bladder outlet obstruction for translational research.Effects of varying degrees of partial bladder outlet obstruction on urinary bladder function of rats: A novel link to inflammation, oxidative stress and hypoxia.Bladder fibrosis during outlet obstruction is triggered through the NLRP3 inflammasome and the production of IL-1β.Effect of Naftopidil on Bladder Microcirculation in a Rat Model of Bladder Outlet Obstruction.NLRP3/IL-1β mediates denervation during bladder outlet obstruction in rats.Collagen content in the bladder of men with LUTS undergoing open prostatectomy: A pilot study.Preventative effects of a HIF inhibitor, 17-DMAG, on partial bladder outlet obstruction-induced bladder dysfunction.Hypoxia-increased expression of genes involved in inflammation, dedifferentiation, pro-fibrosis, and extracellular matrix remodeling of human bladder smooth muscle cells.
P2860
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P2860
Bladder outlet obstruction: progression from inflammation to fibrosis.
description
2010 nî lūn-bûn
@nan
2010年の論文
@ja
2010年学术文章
@wuu
2010年学术文章
@zh
2010年学术文章
@zh-cn
2010年学术文章
@zh-hans
2010年学术文章
@zh-my
2010年学术文章
@zh-sg
2010年學術文章
@yue
2010年學術文章
@zh-hant
name
Bladder outlet obstruction: progression from inflammation to fibrosis.
@en
Bladder outlet obstruction: progression from inflammation to fibrosis.
@nl
type
label
Bladder outlet obstruction: progression from inflammation to fibrosis.
@en
Bladder outlet obstruction: progression from inflammation to fibrosis.
@nl
prefLabel
Bladder outlet obstruction: progression from inflammation to fibrosis.
@en
Bladder outlet obstruction: progression from inflammation to fibrosis.
@nl
P2093
P1433
P1476
Bladder outlet obstruction: progression from inflammation to fibrosis
@en
P2093
Edward E Tredget
Haiyan Jiao
JianFei Wang
Keijiro Hori
P304
P356
10.1111/J.1464-410X.2010.09445.X
P577
2010-12-01T00:00:00Z