Effects of EGFR Inhibitor on Helicobacter pylori Induced Gastric Epithelial Pathology in Vivo.

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Proteolysis in Helicobacter pylori-Induced Gastric Cancer.Epidemiology of Helicobacter pylori and CagA-Positive Infections and Global Variations in Gastric Cancer.

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Q39234154-45C60901-5288-432B-B417-597A18F3CF2B Q52801849-82E947A2-FC18-42A5-978B-86FBE5128997

P2860

Effects of EGFR Inhibitor on Helicobacter pylori Induced Gastric Epithelial Pathology in Vivo.

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2013 nî lūn-bûn

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2013年の論文

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2013年学术文章

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2013年学术文章

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2013年学术文章

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2013年學術文章

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Effects of EGFR Inhibitor on H ...... Epithelial Pathology in Vivo.

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Effects of EGFR Inhibitor on H ...... Epithelial Pathology in Vivo.

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Effects of EGFR Inhibitor on H ...... Epithelial Pathology in Vivo.

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Effects of EGFR Inhibitor on H ...... Epithelial Pathology in Vivo.

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Effects of EGFR Inhibitor on H ...... Epithelial Pathology in Vivo.

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Effects of EGFR Inhibitor on H ...... Epithelial Pathology in Vivo.

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Effects of EGFR Inhibitor on H ...... Epithelial Pathology in Vivo.

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Effects of EGFR Inhibitor on H ...... Epithelial Pathology in Vivo.

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Effects of EGFR Inhibitor on H ...... Epithelial Pathology in Vivo.

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Pathogens

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Effects of EGFR Inhibitor on H ...... Epithelial Pathology in Vivo.

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Anthony H T Jeremy

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D Mark Pritchard

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Jean E Crabtree

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Kazuma Danjo

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Michael F Dixon

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Philip A Robinson

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P2860

Analysis of Relative Gene Expression Data Using Real-Time Quantitative PCR and the 2−ΔΔCT Method

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EGF receptor transactivation by G-protein-coupled receptors requires metalloproteinase cleavage of proHB-EGF

A mutant epidermal growth factor receptor targeted to lung epithelium inhibits asbestos-induced proliferation and proto-oncogene expression.

Helicobacter pylori counteracts the apoptotic action of its VacA toxin by injecting the CagA protein into gastric epithelial cells.

Activation of EGFR on monocytes is required for human cytomegalovirus entry and mediates cellular motility.

Activation of beta-catenin by carcinogenic Helicobacter pylori

Blocking airway mucous cell metaplasia by inhibiting EGFR antiapoptosis and IL-13 transdifferentiation signals.

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571-590

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scholarly article

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10.3390/PATHOGENS2040571

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Ian M. Carr

Cedric Duval

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Helicobacter pylori

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