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Fibrin clot structure in patients with congenital dysfibrinogenaemia.Factor XIIIa-dependent retention of red blood cells in clots is mediated by fibrin α-chain crosslinkingRelease of TGFβig-h3 by gastric myofibroblasts slows tumor growth and is decreased with cancer progression.Increased expression of the urokinase plasminogen activator system by Helicobacter pylori in gastric epithelial cells.The role of matrix metalloproteinase-7 in redefining the gastric microenvironment in response to Helicobacter pylori.Gastrin stimulates expression of plasminogen activator inhibitor-1 in gastric epithelial cells.Fibrinogen splice variation and cross-linking: Effects on fibrin structure/function and role of fibrinogen γ' as thrombomobulin II.Factor XIII A-Subunit V34L Variant Affects Thrombus Cross-Linking in a Murine Model of Thrombosis.Effects of EGFR Inhibitor on Helicobacter pylori Induced Gastric Epithelial Pathology in Vivo.The effect of blood coagulation factor XIII on fibrin clot structure and fibrinolysis.The 95RGD97 sequence on the Aα chain of fibrinogen is essential for binding to its erythrocyte receptor.Thrombin and fibrinogen γ' impact clot structure by marked effects on intrafibrillar structure and protofibril packing.Roles of fibrin α- and γ-chain specific cross-linking by FXIIIa in fibrin structure and function.A fibrin biofilm covers the blood clot and protects from microbial invasion.Partial deletion of the αC-domain in the Fibrinogen Perth variant is associated with thrombosis, increased clot strength and delayed fibrinolysisGastrin regulates the urokinase plasminogen activator system587 EGFR Inhibitor Reduces Helicobacter-Induced Upregulation of Gastric Activation-Induced Cytidine Deaminase in the Murine INS-GAS Cancer ModelS1658 Long Term Infection of Helicobacter pylori 48gx, and Output Strains 12.2 and 12.3, Results in Hypergastrinaemia, Gastric Carcinoids and Dysplasia in Mongolian GerbilsInsulin-Like Growth Factor Binding Protein-5 Is a Target of Matrix Metalloproteinase-7: Implications for Epithelial-Mesenchymal SignalingRecurrent venous thromboembolism patients form clots with lower elastic modulus than those formed by patients with non-recurrent diseaseFibrinogen αC-regions are not directly involved in fibrin polymerization as evidenced by a "Double-Detroit" recombinant fibrinogen mutant and knobs-mimic peptidesCoagulation Factor XIII-A Subunit Missense Mutation in the Pathobiology of Autosomal Dominant Multiple Dermatofibromas
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