Regulation of HSP60 and the role of MK2 in a new model of severe experimental pancreatitis. - Wikidata - wikimedia - TriplyDB

Regulation of HSP60 and the role of MK2 in a new model of severe experimental pancreatitis.

Regulation of HSP60 and the role of MK2 in a new model of severe experimental pancreatitis.

http://www.wikidata.org/entity/Q43054672

about

Ascitic fluid and serum from rats with acute pancreatitis injure rat pancreatic tissues and alter the expression of heat shock protein 60.Molecular and cellular mechanisms of pancreatic injury.Down-regulation of HSP60 expression by RNAi increases lipopolysaccharide- and cerulein-induced damages on isolated rat pancreatic tissues.An NF-κB pathway-mediated positive feedback loop amplifies Ras activity to pathological levels in mice.Socs1 and Socs3 degrades Traf6 via polyubiquitination in LPS-induced acute necrotizing pancreatitis.Deletion Of XIAP reduces the severity of acute pancreatitis via regulation of cell death and nuclear factor-κB activity The role of p38 mitogen-activated protein kinase in the pathogenesis of inflammatory bowel disease.Anti-inflammatory effect of cannabinoid agonist WIN55, 212 on mouse experimental colitis is related to inhibition of p38MAPK.The Role of Mitogen-Activated Protein Kinase-Activated Protein Kinases (MAPKAPKs) in Inflammation.Long-lasting effect of infant rats endotoxemia on heat shock protein 60 in the pancreatic acinar cells: involvement of toll-like receptor 4 Activation of cannabinoid receptor 2 reduces inflammation in acute experimental pancreatitis via intra-acinar activation of p38 and MK2-dependent mechanisms.Hsp90 regulation affects the treatment of glucocorticoid for pancreatitis-induced lung injury.Resolvin D1 protects against inflammation in experimental acute pancreatitis and associated lung injury.Inhibition of p38/Mk2 signaling pathway improves the anti-inflammatory effect of WIN55 on mouse experimental colitis.

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P2860

Regulation of HSP60 and the role of MK2 in a new model of severe experimental pancreatitis.

http://www.wikidata.org/entity/Q43054672

description

2009 nî lūn-bûn

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2009年の論文

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2009年学术文章

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2009年学术文章

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2009年學術文章

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2009年學術文章

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Regulation of HSP60 and the ro ...... ere experimental pancreatitis.

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Regulation of HSP60 and the ro ...... ere experimental pancreatitis.

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Regulation of HSP60 and the ro ...... ere experimental pancreatitis.

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Regulation of HSP60 and the ro ...... ere experimental pancreatitis.

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Regulation of HSP60 and the ro ...... ere experimental pancreatitis.

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American Journal of Physiology - Gastrointestinal and Liver Physiology

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Regulation of HSP60 and the ro ...... ere experimental pancreatitis.

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Antje Malo

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Burkhard Göke

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Chang-Jie Chen

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Claus Schäfer

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Eike Gallmeier

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Shuai Lv

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Stephanie Ochs

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Yong-Yu Li

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Zhi-Rong Gao

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P2860

Arginine induced acute pancreatitis alters the actin cytoskeleton and increases heat shock protein expression in rat pancreatic acinar cells

Possible association of chaperonin 60 with secretory proteins in pancreatic acinar cells

MK2 targets AU-rich elements and regulates biosynthesis of tumor necrosis factor and interleukin-6 independently at different post-transcriptional levels

MAPKAP kinase 2 is essential for LPS-induced TNF-alpha biosynthesis

A mouse model of severe acute pancreatitis induced with caerulein and lipopolysaccharide.

Pathophysiology of pancreatitis. Role of cytokines and other mediators of inflammation.

Early trypsinogen activation in acute pancreatitis.

Acute experimental pancreatitis and NF-kappaB/Rel activation.

Role of cytokines and oxidative stress in the pathophysiology of acute pancreatitis: therapeutical implications.

Pathophysiology of acute experimental pancreatitis: lessons from genetically engineered animal models and new molecular approaches.

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