SOD2 overexpression: enhanced mitochondrial tolerance but absence of effect on UCP activity. - Wikidata - wikimedia - TriplyDB

SOD2 overexpression: enhanced mitochondrial tolerance but absence of effect on UCP activity.

SOD2 overexpression: enhanced mitochondrial tolerance but absence of effect on UCP activity.

http://www.wikidata.org/entity/Q43192513

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Loss of UCP2 attenuates mitochondrial dysfunction without altering ROS production and uncoupling activity Mitochondrial ROS regulate thermogenic energy expenditure and sulfenylation of UCP1 Synergy of fatty acid and reactive alkenal activation of proton conductance through uncoupling protein 1 in mitochondria Mitochondrial dysfunction accounts for the stochastic heterogeneity in telomere-dependent senescence.Role of the transmembrane potential in the membrane proton leak.High fat diet-induced changes in mouse muscle mitochondrial phospholipids do not impair mitochondrial respiration despite insulin resistance.Mechanism for the protective effect of resveratrol against oxidative stress-induced neuronal death.Mouse Models of Oxidative Stress Indicate a Role for Modulating Healthy Aging.Enhanced mitochondrial superoxide scavenging does not improve muscle insulin action in the high fat-fed mouse MnSOD overexpression reduces fibrosis and pro-apoptotic signaling in the aging mouse heart.Mitochondrial antioxidative capacity regulates muscle glucose uptake in the conscious mouse: effect of exercise and diet.Mitochondrial uncoupling: a key contributor to reduced cardiac efficiency in diabetes.Metabolically inert perfluorinated fatty acids directly activate uncoupling protein 1 in brown-fat mitochondria Manganese superoxide dismutase deficiency triggers mitochondrial uncoupling and the Warburg effect.Superoxide dismutase 2 overexpression alleviates maternal diabetes-induced neural tube defects, restores mitochondrial function and suppresses cellular stress in diabetic embryopathy.Uncoupling protein-2 regulates lifespan in mice.Reduced mitochondrial SOD displays mortality characteristics reminiscent of natural aging Mechanisms of stress resistance in Snell dwarf mouse fibroblasts: enhanced antioxidant and DNA base excision repair capacity, but no differences in mitochondrial metabolism Overexpression of Mn superoxide dismutase does not increase life span in mice.Neuroprotective role of mitochondrial uncoupling protein 2 in cerebral stroke.Curcumin and liver disease.Carboxyatractyloside effects on brown-fat mitochondria imply that the adenine nucleotide translocator isoforms ANT1 and ANT2 may be responsible for basal and fatty-acid-induced uncoupling respectively.The molecular features of uncoupling protein 1 support a conventional mitochondrial carrier-like mechanism Multiple phytoestrogens inhibit cell growth and confer cytoprotection by inducing manganese superoxide dismutase expression.Glutathionylation acts as a control switch for uncoupling proteins UCP2 and UCP3.Rescue of neurons from ischemic injury by peroxisome proliferator-activated receptor-gamma requires a novel essential cofactor LMO4 Caveolin-1-ablated mice survive in cold by nonshivering thermogenesis despite desensitized adrenergic responsiveness.UCP1 and defense against oxidative stress. 4-Hydroxy-2-nonenal effects on brown fat mitochondria are uncoupling protein 1-independent.Uncoupling protein 1 decreases superoxide production in brown adipose tissue mitochondria.(-)-Hydroxycitric acid attenuates endoplasmic reticulum stress-mediated alterations in 3T3-L1 adipocytes by protecting mitochondria and downregulating inflammatory markers.Mitochondrial reactive oxygen species and adipose tissue thermogenesis: Bridging physiology and mechanisms.Uncoupling protein 1 controls reactive oxygen species in brown adipose tissue.Stress turns on the heat: Regulation of mitochondrial biogenesis and UCP1 by ROS in adipocytes.Reactive oxygen species and fatigue-induced prolonged low-frequency force depression in skeletal muscle fibres of rats, mice and SOD2 overexpressing mice.Implications of PI3K/AKT/PTEN Signaling on Superoxide Dismutases Expression and in the Pathogenesis of Alzheimer's Disease.Reactive Oxygen Species, Superoxide Dimutases, and PTEN-p53-AKT-MDM2 Signaling Loop Network in Mesenchymal Stem/Stromal Cells Regulation.Oxidative status of cardinal ligament in pelvic organ prolapse

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SOD2 overexpression: enhanced mitochondrial tolerance but absence of effect on UCP activity.

http://www.wikidata.org/entity/Q43192513

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SOD2 overexpression: enhanced ...... nce of effect on UCP activity.

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SOD2 overexpression: enhanced ...... nce of effect on UCP activity.

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SOD2 overexpression: enhanced ...... nce of effect on UCP activity.

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SOD2 overexpression: enhanced ...... nce of effect on UCP activity.

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Barbara Cannon

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Emma C Backlund

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José P Silva

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Natasa Petrovic

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P2860

The uncoupling protein homologues: UCP1, UCP2, UCP3, StUCP and AtUCP

Disruption of the uncoupling protein-2 gene in mice reveals a role in immunity and reactive oxygen species production

Superoxide activates mitochondrial uncoupling proteins

Dilated cardiomyopathy and neonatal lethality in mutant mice lacking manganese superoxide dismutase

A novel neurological phenotype in mice lacking mitochondrial manganese superoxide dismutase

The basal proton conductance of skeletal muscle mitochondria from transgenic mice overexpressing or lacking uncoupling protein-3

Mitochondrial disease in superoxide dismutase 2 mutant mice

Increased mitochondrial oxidative stress in the Sod2 (+/-) mouse results in the age-related decline of mitochondrial function culminating in increased apoptosis

Neurodegeneration, myocardial injury, and perinatal death in mitochondrial superoxide dismutase-deficient mice

High protonic potential actuates a mechanism of production of reactive oxygen species in mitochondria

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Irina G. Shabalina

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