Dicarbonyl stress in the absence of hyperglycemia increases endothelial inflammation and atherogenesis similar to that observed in diabetes.
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Molecular and Cellular Mechanisms of Cardiovascular Disorders in DiabetesEffects of Metformin on Tissue Oxidative and Dicarbonyl Stress in Transgenic Spontaneously Hypertensive Rats Expressing Human C-Reactive ProteinIncreased Dicarbonyl Stress as a Novel Mechanism of Multi-Organ Failure in Critical IllnessDicarbonyl stress and atherosclerosis: is it all RAGE?Low-molecular-weight chitosan scavenges methylglyoxal and N (ε)-(carboxyethyl)lysine, the major factors contributing to the pathogenesis of nephropathy.Dicarbonyls and glyoxalase in disease mechanisms and clinical therapeuticsMethylglyoxal, a glycolysis side-product, induces Hsp90 glycation and YAP-mediated tumor growth and metastasis.Methylglyoxal-Glyoxalase 1 Balance: The Root of Vascular Damage.The role of methylglyoxal and the glyoxalase system in diabetes and other age-related diseases.Effect of diet-derived advanced glycation end products on inflammation.Damaging effects of hyperglycemia on cardiovascular function: spotlight on glucose metabolic pathways.Inhibition of Methylglyoxal-Induced AGEs/RAGE Expression Contributes to Dermal Protection by N-Acetyl-L-Cysteine.Cellular mechanisms and consequences of glycation in atherosclerosis and obesity.RAGE and glyoxalase in kidney disease.Methylglyoxal in Metabolic Disorders: Facts, Myths, and Promises.Carbonyl stress phenomena during chronic infection with Opisthorchis felineus.Chitosan Prevents Gentamicin-Induced Nephrotoxicity via a Carbonyl Stress-Dependent Pathway.A Glyoxalase-1 Knockdown Does Not Have Major Short Term Effects on Energy Expenditure and Atherosclerosis in Mice.Glyoxalase 1-knockdown in human aortic endothelial cells - effect on the proteome and endothelial function estimates.ADAM10 modulates calcitriol-regulated RAGE in cardiomyocytes.Diabetic retinopathy: hyperglycaemia, oxidative stress and beyond.Impaired glyoxalase activity is associated with reduced expression of neurotrophic factors and pro-inflammatory processes in diabetic skin cells.Deleterious Effect of Advanced CKD on Glyoxalase System Activity not Limited to Diabetes Aetiology.
P2860
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P2860
Dicarbonyl stress in the absence of hyperglycemia increases endothelial inflammation and atherogenesis similar to that observed in diabetes.
description
2014 nî lūn-bûn
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2014年の論文
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2014年学术文章
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2014年学术文章
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2014年学术文章
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2014年学术文章
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name
Dicarbonyl stress in the absen ...... to that observed in diabetes.
@en
Dicarbonyl stress in the absen ...... to that observed in diabetes.
@nl
type
label
Dicarbonyl stress in the absen ...... to that observed in diabetes.
@en
Dicarbonyl stress in the absen ...... to that observed in diabetes.
@nl
prefLabel
Dicarbonyl stress in the absen ...... to that observed in diabetes.
@en
Dicarbonyl stress in the absen ...... to that observed in diabetes.
@nl
P2093
P356
P1433
P1476
Dicarbonyl stress in the absen ...... to that observed in diabetes.
@en
P2093
Chris Tikellis
Despina Tsorotes
Karin Jandeleit-Dahm
Mark E Cooper
Raelene J Pickering
P304
P356
10.2337/DB13-0932
P407
P577
2014-05-08T00:00:00Z