Three classes of ubiquinone analogs regulate the mitochondrial permeability transition pore through a common site.
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Quinones and aromatic chemical compounds in particulate matter induce mitochondrial dysfunction: implications for ultrafine particle toxicityCoenzyme Q and Its Role in the Dietary Therapy against AgingA defect in coenzyme Q biosynthesis is responsible for the respiratory deficiency in Saccharomyces cerevisiae abc1 mutants.Phosphate is essential for inhibition of the mitochondrial permeability transition pore by cyclosporin A and by cyclophilin D ablationElectron spin resonance investigation of semiquinone radicals formed from the reaction of ubiquinone 0 with human oxyhemoglobin.Coenzyme q10 prevents apoptosis by inhibiting mitochondrial depolarization independently of its free radical scavenging property.Ligand-selective modulation of the permeability transition pore by arginine modification. Opposing effects of p-hydroxyphenylglyoxal and phenylglyoxal.Immunochemical identification of coenzyme Q0-dihydrolipoamide adducts in the E2 components of the alpha-ketoglutarate and pyruvate dehydrogenase complexes partially explains the cellular toxicity of coenzyme Q0.Modification of permeability transition pore arginine(s) by phenylglyoxal derivatives in isolated mitochondria and mammalian cells. Structure-function relationship of arginine ligands.Ubiquinone analogs: a mitochondrial permeability transition pore-dependent pathway to selective cell deathCoenzyme Q10 reduces ethanol-induced apoptosis in corneal fibroblasts.The effects of idebenone on mitochondrial bioenergeticsUncoupling proteins 2 and 3 are highly active H(+) transporters and highly nucleotide sensitive when activated by coenzyme Q (ubiquinone)Therapeutic effects of coenzyme Q10 (CoQ10) and reduced CoQ10 in the MPTP model of Parkinsonism.Calorie restriction modifies ubiquinone and COQ transcript levels in mouse tissues.Involvement of the mitochondrial permeability transition pore in chronic ethanol-mediated liver injury in mice.Therapeutic use of coenzyme Q10 and coenzyme Q10-related compounds and formulations.New developments on the functions of coenzyme Q in mitochondria.Composition of air pollution particles and oxidative stress in cells, tissues, and living systems.Prerequisites for ubiquinone analogs to prevent mitochondrial permeability transition-induced cell death.The mitochondrial permeability transition pore: a mystery solved?Mitochondrial channels: ion fluxes and more.The regulation of coenzyme q biosynthesis in eukaryotic cells: all that yeast can tell us.The mitochondrial phosphate carrier interacts with cyclophilin D and may play a key role in the permeability transition.The voltage-dependent anion channel is the target for a new class of inhibitors of the mitochondrial permeability transition pore.Long-chain ceramide is a potent inhibitor of the mitochondrial permeability transition pore.Mitochondrial protection and anti-aging activity of Astragalus polysaccharides and their potential mechanism.Human immunodeficiency virus type 1 Vpr inhibits axonal outgrowth through induction of mitochondrial dysfunction.Rotenone inhibits the mitochondrial permeability transition-induced cell death in U937 and KB cells.Mitochondrial respiration is uniquely associated with the prooxidant and apoptotic effects of N-(4-hydroxyphenyl)retinamide.Uptake of exogenous coenzyme Q and transport to mitochondria is required for bc1 complex stability in yeast coq mutants.The Coenzyme Q10 analog decylubiquinone inhibits the redox-activated mitochondrial permeability transition: role of mitcohondrial [correction mitochondrial] complex III.Opening of the mitochondrial permeability transition pore induces reactive oxygen species production at the level of the respiratory chain complex I.Cytochrome bc(1) regulates the mitochondrial permeability transition by two distinct pathways.The Combination of Physical Exercise with Muscle-Directed Antioxidants to Counteract Sarcopenia: A Biomedical Rationale for Pleiotropic Treatment with Creatine and Coenzyme Q10.Effects of ubiquinone derivatives on the mitochondrial unselective channel of Saccharomyces cerevisiae.Mansouramycin C kills cancer cells through reactive oxygen species production mediated by opening of mitochondrial permeability transition pore.The still uncertain identity of the channel-forming unit(s) of the mitochondrial permeability transition pore
P2860
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P2860
Three classes of ubiquinone analogs regulate the mitochondrial permeability transition pore through a common site.
description
2000 nî lūn-bûn
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2000年学术文章
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2000年学术文章
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name
Three classes of ubiquinone an ...... on pore through a common site.
@en
Three classes of ubiquinone an ...... on pore through a common site.
@nl
type
label
Three classes of ubiquinone an ...... on pore through a common site.
@en
Three classes of ubiquinone an ...... on pore through a common site.
@nl
prefLabel
Three classes of ubiquinone an ...... on pore through a common site.
@en
Three classes of ubiquinone an ...... on pore through a common site.
@nl
P2093
P2860
P356
P1476
Three classes of ubiquinone an ...... on pore through a common site.
@en
P2093
P2860
P304
29521-29527
P356
10.1074/JBC.M004128200
P407
P577
2000-09-01T00:00:00Z