Erbb4 deletion from fast-spiking interneurons causes schizophrenia-like phenotypes.
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Chandelier Cells in Functional and Dysfunctional Neural CircuitsHippocampal-Prefrontal Interactions in Cognition, Behavior and Psychiatric DiseaseGenetic disruption of voltage-gated calcium channels in psychiatric and neurological disordersSynchrony in schizophrenia: a window into circuit-level pathophysiologyNeuropathologic implication of peripheral neuregulin-1 and EGF signals in dopaminergic dysfunction and behavioral deficits relevant to schizophrenia: their target cells and time windowNeuronal migration abnormalities and its possible implications for schizophreniaSynaptic plasticity, neural circuits, and the emerging role of altered short-term information processing in schizophrenia.Molecular underpinnings of prefrontal cortex development in rodents provide insights into the etiology of neurodevelopmental disordersGamma rhythms link prefrontal interneuron dysfunction with cognitive inflexibility in Dlx5/6(+/-) mice.Activity-dependent adaptations in inhibitory axonsSepsis-induced selective parvalbumin interneuron phenotype loss and cognitive impairments may be mediated by NADPH oxidase 2 activation in mice.Neuregulin 3 Mediates Cortical Plate Invasion and Laminar Allocation of GABAergic InterneuronsCytosolic Accumulation of L-Proline Disrupts GABA-Ergic Transmission through GAD BlockadeBehavioral, Neurophysiological, and Synaptic Impairment in a Transgenic Neuregulin1 (NRG1-IV) Murine Schizophrenia Model.Spontaneous Gamma Activity in Schizophrenia.Defective craniofacial development and brain function in a mouse model for depletion of intracellular inositol synthesis.Distinct phenotypes of new transmembrane-domain neuregulin 1 mutant mice and the rescue effects of valproate on the observed schizophrenia-related cognitive deficitsA negative feedback loop controls NMDA receptor function in cortical interneurons via neuregulin 2/ErbB4 signalling.Tuftsin-driven experimental autoimmune encephalomyelitis recovery requires neuropilin-1.Inhibition of STEP61 ameliorates deficits in mouse and hiPSC-based schizophrenia models.Modulation of GABAergic transmission in development and neurodevelopmental disorders: investigating physiology and pathology to gain therapeutic perspectives.Cell autonomous regulation of hippocampal circuitry via Aph1b-γ-secretase/neuregulin 1 signalling.Cannabinoids and Vanilloids in Schizophrenia: Neurophysiological Evidence and Directions for Basic Research.Ablation of ErbB4 from excitatory neurons leads to reduced dendritic spine density in mouse prefrontal cortex.Genetic labeling reveals novel cellular targets of schizophrenia susceptibility gene: distribution of GABA and non-GABA ErbB4-positive cells in adult mouse brain.Glycine receptors control the generation of projection neurons in the developing cerebral cortexAlterations in cortical network oscillations and parvalbumin neurons in schizophrenia.Pathological Basis for Deficient Excitatory Drive to Cortical Parvalbumin Interneurons in Schizophrenia.Neurodevelopment, GABA system dysfunction, and schizophreniaTransient overexposure of neuregulin 3 during early postnatal development impacts selective behaviors in adulthood.Neuregulin 1 expression and electrophysiological abnormalities in the Neuregulin 1 transmembrane domain heterozygous mutant mouseDysregulated ErbB4 Splicing in Schizophrenia: Selective Effects on Parvalbumin ExpressionSpatial distribution of neurons innervated by chandelier cells.Interneuron epigenomes during the critical period of cortical plasticity: Implications for schizophrenia.Converging models of schizophrenia--Network alterations of prefrontal cortex underlying cognitive impairments.Interneuronal DISC1 regulates NRG1-ErbB4 signalling and excitatory-inhibitory synapse formation in the mature cortex.Transgenic labeling of parvalbumin-expressing neurons with tdTomato.Gamma band oscillations: a key to understanding schizophrenia symptoms and neural circuit abnormalities.Sensory encoding in Neuregulin 1 mutantsIncreased GABAB receptor signaling in a rat model for schizophrenia.
P2860
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P2860
Erbb4 deletion from fast-spiking interneurons causes schizophrenia-like phenotypes.
description
2013 nî lūn-bûn
@nan
2013年の論文
@ja
2013年学术文章
@wuu
2013年学术文章
@zh-cn
2013年学术文章
@zh-hans
2013年学术文章
@zh-my
2013年学术文章
@zh-sg
2013年學術文章
@yue
2013年學術文章
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2013年學術文章
@zh-hant
name
Erbb4 deletion from fast-spiking interneurons causes schizophrenia-like phenotypes.
@en
Erbb4 deletion from fast-spiking interneurons causes schizophrenia-like phenotypes.
@nl
type
label
Erbb4 deletion from fast-spiking interneurons causes schizophrenia-like phenotypes.
@en
Erbb4 deletion from fast-spiking interneurons causes schizophrenia-like phenotypes.
@nl
prefLabel
Erbb4 deletion from fast-spiking interneurons causes schizophrenia-like phenotypes.
@en
Erbb4 deletion from fast-spiking interneurons causes schizophrenia-like phenotypes.
@nl
P2093
P50
P1433
P1476
Erbb4 deletion from fast-spiking interneurons causes schizophrenia-like phenotypes
@en
P2093
Efrén Alvarez-Salvado
Jorge R Brotons-Mas
María Victoria Gabaldón
P304
P356
10.1016/J.NEURON.2013.07.010
P407
P50
P577
2013-09-01T00:00:00Z