Attenuation of cardiac dysfunction by HSPA12B in endotoxin-induced sepsis in mice through a PI3K-dependent mechanism. - Wikidata - wikimedia - TriplyDB

Attenuation of cardiac dysfunction by HSPA12B in endotoxin-induced sepsis in mice through a PI3K-dependent mechanism.

Attenuation of cardiac dysfunction by HSPA12B in endotoxin-induced sepsis in mice through a PI3K-dependent mechanism.

http://www.wikidata.org/entity/Q43617921

about

Transcriptome-wide RNA sequencing analysis of rat skeletal muscle feed arteries. II. Impact of exercise training in obesity.Plasma HSPA12B is a potential predictor for poor outcome in severe sepsis The acute transcriptional response of the coral Acropora millepora to immune challenge: expression of GiMAP/IAN genes links the innate immune responses of corals with those of mammals and plants.HSPA12B inhibits lipopolysaccharide-induced inflammatory response in human umbilical vein endothelial cells.Tissue inflammation and nitric oxide-mediated alterations in cardiovascular function are major determinants of endotoxin-induced insulin resistance HSPA12B: a novel facilitator of lung tumor growth Class III PI3K-mediated prolonged activation of autophagy plays a critical role in the transition of cardiac hypertrophy to heart failure.Notoginsenoside R1 attenuates cardiac dysfunction in endotoxemic mice: an insight into oestrogen receptor activation and PI3K/Akt signalling.HSPA12B Attenuated Acute Myocardial Ischemia/reperfusion Injury via Maintaining Endothelial Integrity in a PI3K/Akt/mTOR-dependent Mechanism.Inhibition of miR-155 Protects Against LPS-induced Cardiac Dysfunction and Apoptosis in Mice.Heat shock protein 72 expressing stress in sepsis: unbridgeable gap between animal and human studies--a hypothetical "comparative" study Regulation of A-Kinase-Anchoring Protein 12 by Heat Shock Protein A12B to Prevent Ventricular Dysfunction Following Acute Myocardial Infarction in Diabetic Rats.Melatonin attenuates sepsis-induced cardiac dysfunction via a PI3K/Akt-dependent mechanism.Phosphoinositide-3-kinase/akt - dependent signaling is required for maintenance of [Ca(2+)](i), I(Ca), and Ca(2+) transients in HL-1 cardiomyocytes.Sepsis-Induced Cardiomyopathy: Mechanisms and Treatments HSPA12B and repairing the heart: beauty in simplicity.Over-expression of HSPA12B protects mice against myocardium ischemic/reperfusion injury through a PPARγ-dependent PI3K/Akt/eNOS pathway.The role of HSPA12B in regulating neuronal apoptosis.HSPA12B promotes functional recovery after ischaemic stroke through an eNOS-dependent mechanism.Silencing Poly (ADP-Ribose) glycohydrolase (PARG) expression inhibits growth of human colon cancer cells in vitro via PI3K/Akt/NFκ-B pathway.

P2860

Q33673283-6EFCD6C5-0CAB-4F4B-8B2D-3A529AE375FA Q33825845-909F559E-9587-46D5-AC9A-3E1C331F4622 Q34773967-30CBD164-1DA7-4594-9191-91439A4EE927 Q35206504-7A3678C5-734C-4125-9DE2-B9E2C927311C Q35794402-3C035120-E08B-4838-93A4-029D7E6BD3C3 Q35833194-3563B32B-63EB-46DC-86AA-A298F0DCE602 Q35879553-A0B029B2-7C4C-40C4-B43D-317424A12820 Q36709261-7F7DED59-5BC0-4D8F-BE07-FC889FD50EED Q37267487-54186625-25AB-40BF-800B-4F294E88660C Q37393972-FB05BE81-B55B-4F50-BA32-034FDC5E9AAC Q37552140-AF15E43F-5DBB-4793-9B73-39904B5EEED3 Q38915485-218E83D1-DCE5-449C-9759-12E8231673A0 Q40869902-14753604-3EFD-4455-B6FB-C4F054D11569 Q42202030-624CCF3C-22C9-437D-AB40-072EA36DD80D Q42362538-AF8EA64C-F0EA-4336-B934-D9E404B05650 Q42559907-5B97EE7B-5022-4AA9-95AF-4ABAD3DDC3AD Q43062839-BA6E1FB8-F467-4E73-BCC5-A889DDD9D684 Q45853650-E7C57812-51DD-4462-A293-D54B3E470FF5 Q48091647-B8FECA3D-6E9D-43D4-9AE4-76BD055B387F Q53237156-FA5C68D0-3CA1-4BDC-8C35-592141964544

P2860

Attenuation of cardiac dysfunction by HSPA12B in endotoxin-induced sepsis in mice through a PI3K-dependent mechanism.

http://www.wikidata.org/entity/Q43617921

description

2010 nî lūn-bûn

@nan

2010年の論文

@ja

2010年学术文章

@wuu

2010年学术文章

@zh

2010年学术文章

@zh-cn

2010年学术文章

@zh-hans

2010年学术文章

@zh-my

2010年学术文章

@zh-sg

2010年學術文章

@yue

2010年學術文章

@zh-hant

name

Attenuation of cardiac dysfunc ...... gh a PI3K-dependent mechanism.

@en

Attenuation of cardiac dysfunc ...... gh a PI3K-dependent mechanism.

@nl

type

label

Attenuation of cardiac dysfunc ...... gh a PI3K-dependent mechanism.

@en

Attenuation of cardiac dysfunc ...... gh a PI3K-dependent mechanism.

@nl

prefLabel

Attenuation of cardiac dysfunc ...... gh a PI3K-dependent mechanism.

@en

Attenuation of cardiac dysfunc ...... gh a PI3K-dependent mechanism.

@nl

P1433

Q43617921-F6EB731C-EA01-475E-BAA3-F0235D9BF6C6

P1476

Q43617921-281EBED1-633F-416A-89A7-B81FEB10B61C

P2093

Q43617921-0A0EFCD8-FA8C-4961-821E-6A275208FDA1

Q43617921-182446CE-D04F-42C9-A678-9E55C31AB1B1

Q43617921-1C0F5C17-44F7-4846-8A84-763A642F366F

Q43617921-2292E34B-CD03-435E-8C5E-5393FAA1BDE6

Q43617921-3AA1C5AE-627D-4A7B-A953-43785BD5F3B6

Q43617921-95DC75D5-1825-4B82-A131-4114499182E6

Q43617921-A813EFFA-AAA4-4C9A-9E94-79F37369871B

Q43617921-C19C607A-F937-408C-B995-C265C4833762

Q43617921-C5642A43-C31B-4D9C-B1D8-8BAED768E837

Q43617921-FAFE383A-CA21-43E6-8B30-0804CF31A5BB

P2860

Q43617921-03499E05-95AD-4828-B2CC-A05F2FD1753C

Q43617921-0CB75007-565B-4353-8FE9-6D4A43E66A06

Q43617921-0F0673C9-2991-4CFF-899E-FB9DB4646DF4

Q43617921-2240E06F-6D19-4C6B-9E1B-FA8CFE696098

Q43617921-23E3B721-44DE-4E80-A434-B506E495310E

Q43617921-2CF6C5DA-2102-44C5-9B18-94B0182957F9

Q43617921-41EA900B-6E49-4F45-B58F-A06A1F63AAD3

Q43617921-47670059-ADAC-4B9D-BB1B-1D7CE12894AF

Q43617921-4970354E-4044-4199-835E-0F6CB20022B0

Q43617921-49CAD1F1-372D-4EE5-A96A-8408C8F37233

P304

Q43617921-59A04605-CE89-4767-973D-64A823232623

P31

Q43617921-A97B6182-1AB3-4978-B9C1-400EAB3ADED4

P356

Q43617921-DD75F4BA-56EB-4C1E-AD73-0EE73C67D542

P433

Q43617921-32B21E27-2409-4691-B368-4F35FCAE5B92

P478

Q43617921-2FF1CB00-7C08-45C7-B957-6474CD83EE5B

P577

Q43617921-FE8C234F-98E1-4090-8404-7236CFED98F6

P5875

Q43617921-BAAD3E69-0217-4053-82AD-84A0D21FCA1A

P698

Q43617921-D96B19B9-0016-4747-9E4F-319B68C6DB7E

P921

Q43617921-2B74DA93-9077-49E1-8C67-0B498EFF3425

Q43617921-C27FCC48-F653-49CA-A2FD-9DD82E4AC90C

P356

P1433

Cardiovascular Research

P1476

Attenuation of cardiac dysfunc ...... gh a PI3K-dependent mechanism.

@en

P2093

Chuanfu Li

http://www.w3.org/2001/XMLSchema#string

Hongmei Zhou

http://www.w3.org/2001/XMLSchema#string

Jin Qian

http://www.w3.org/2001/XMLSchema#string

Jingjin Li

http://www.w3.org/2001/XMLSchema#string

Li Liu

http://www.w3.org/2001/XMLSchema#string

Xiang Gao

http://www.w3.org/2001/XMLSchema#string

Xiaojin Zhang

http://www.w3.org/2001/XMLSchema#string

Yunlin Cheng

http://www.w3.org/2001/XMLSchema#string

Zhengnian Ding

http://www.w3.org/2001/XMLSchema#string

Zhihua Han

http://www.w3.org/2001/XMLSchema#string

P2860

Two Hsp70 family members expressed in atherosclerotic lesions

Prevention of LPS-induced acute lung injury in mice by mesenchymal stem cells overexpressing angiopoietin 1

A highly efficient recombineering-based method for generating conditional knockout mutations

PI3K and negative regulation of TLR signaling.

Heat shock proteins as emerging therapeutic targets.

Alpha-lipoic acid attenuates LPS-induced inflammatory responses by activating the phosphoinositide 3-kinase/Akt signaling pathway

Mechanisms of sepsis-induced cardiac dysfunction.

Sepsis and the heart.

Cardiac-specific expression of heat shock protein 27 attenuated endotoxin-induced cardiac dysfunction and mortality in mice through a PI3K/Akt-dependent mechanism.

Resistance to endotoxin shock in transgenic mice overexpressing endothelial nitric oxide synthase.

P304

109-118

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1093/CVR/CVQ268

http://www.w3.org/2001/XMLSchema#string

P433

1

http://www.w3.org/2001/XMLSchema#string

P478

89

http://www.w3.org/2001/XMLSchema#string

P577

2010-08-23T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P5875

45821103

http://www.w3.org/2001/XMLSchema#string

P698

20733008

http://www.w3.org/2001/XMLSchema#string

P921