Temporally regulated and tissue-specific gene manipulations in the adult and embryonic heart using a tamoxifen-inducible Cre protein.
about
Essential role for mitochondrial thioredoxin reductase in hematopoiesis, heart development, and heart functionTbx20 regulates a genetic program essential to adult mouse cardiomyocyte functionCardiomyocyte cyclooxygenase-2 influences cardiac rhythm and functionElevation of RNA-binding protein CUGBP1 is an early event in an inducible heart-specific mouse model of myotonic dystrophyIs spinal muscular atrophy a disease of the motor neurons only: pathogenesis and therapeutic implications?A Murine Myh6MerCreMer Knock-In Allele Specifically Mediates Temporal Genetic Deletion in Cardiomyocytes after Tamoxifen InductionSuppression of Induced microRNA-15b Prevents Rapid Loss of Cardiac Function in a Dicer Depleted Model of Cardiac DysfunctionDedifferentiation and proliferation of mammalian cardiomyocytesPI3Ks maintain the structural integrity of T-tubules in cardiac myocytesThe beta-catenin/T-cell factor/lymphocyte enhancer factor signaling pathway is required for normal and stress-induced cardiac hypertrophyLoss of cadherin-binding proteins β-catenin and plakoglobin in the heart leads to gap junction remodeling and arrhythmogenesisCardiac ryanodine receptors control heart rate and rhythmicity in adult miceCardiac defects contribute to the pathology of spinal muscular atrophy modelsCardiomyocyte ATP production, metabolic flexibility, and survival require calcium flux through cardiac ryanodine receptors in vivoCardiac tissue-restricted deletion of plakoglobin results in progressive cardiomyopathy and activation of {beta}-catenin signalingProtein tyrosine phosphatase-like A regulates myoblast proliferation and differentiation through MyoG and the cell cycling signaling pathwayFibroblast growth factor signals regulate a wave of Hedgehog activation that is essential for coronary vascular developmentDirect, differential effects of tamoxifen, 4-hydroxytamoxifen, and raloxifene on cardiac myocyte contractility and calcium handlingPDK1 coordinates survival pathways and beta-adrenergic response in the heartMyocardin is required for cardiomyocyte survival and maintenance of heart functionTargeted inactivation of serum response factor in the developing heart results in myocardial defects and embryonic lethalityPhospholipase Cε hydrolyzes perinuclear phosphatidylinositol 4-phosphate to regulate cardiac hypertrophyPerinatal loss of Nkx2-5 results in rapid conduction and contraction defectsIn vivo reprogramming of murine cardiac fibroblasts into induced cardiomyocytesThe mitochondrial Na+/Ca2+ exchanger is essential for Ca2+ homeostasis and viability.Hedgehog signaling is critical for maintenance of the adult coronary vasculature in miceG protein-coupled receptor kinase 2 ablation in cardiac myocytes before or after myocardial infarction prevents heart failure.Rapid Knockout and Reporter Mouse Line Generation and Breeding Colony Establishment Using EUCOMM Conditional-Ready Embryonic Stem Cells: A Case StudyPKC inhibition ameliorates the cardiac phenotype in a mouse model of myotonic dystrophy type 1.Cardiomyocyte PDGFR-beta signaling is an essential component of the mouse cardiac response to load-induced stress.Deep bradycardia and heart block caused by inducible cardiac-specific knockout of the pacemaker channel gene Hcn4Genetic deletion of the mitochondrial phosphate carrier desensitizes the mitochondrial permeability transition pore and causes cardiomyopathyCombining TGF-β signal inhibition and connexin43 silencing for iPSC induction from mouse cardiomyocytes.Characterization of parameters required for effective use of tamoxifen-regulated recombination.Validation of the cardiosphere method to culture cardiac progenitor cells from myocardial tissue.Regulation of the mitochondrial permeability transition pore by the outer membrane does not involve the peripheral benzodiazepine receptor (Translocator Protein of 18 kDa (TSPO)).Heart-specific overexpression of CUGBP1 reproduces functional and molecular abnormalities of myotonic dystrophy type 1.TRPV2 is critical for the maintenance of cardiac structure and function in mice.Tamoxifen-independent recombination in the RIP-CreER mouse.Cardiac stem cell therapy for cardiac repair.
P2860
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P2860
Temporally regulated and tissue-specific gene manipulations in the adult and embryonic heart using a tamoxifen-inducible Cre protein.
description
2001 nî lūn-bûn
@nan
2001年の論文
@ja
2001年学术文章
@wuu
2001年学术文章
@zh
2001年学术文章
@zh-cn
2001年学术文章
@zh-hans
2001年学术文章
@zh-my
2001年学术文章
@zh-sg
2001年學術文章
@yue
2001年學術文章
@zh-hant
name
Temporally regulated and tissu ...... moxifen-inducible Cre protein.
@en
Temporally regulated and tissu ...... moxifen-inducible Cre protein.
@nl
type
label
Temporally regulated and tissu ...... moxifen-inducible Cre protein.
@en
Temporally regulated and tissu ...... moxifen-inducible Cre protein.
@nl
prefLabel
Temporally regulated and tissu ...... moxifen-inducible Cre protein.
@en
Temporally regulated and tissu ...... moxifen-inducible Cre protein.
@nl
P2093
P356
P1433
P1476
Temporally regulated and tissu ...... moxifen-inducible Cre protein.
@en
P2093
Crackower MA
Kimball TR
Molkentin JD
P356
10.1161/HH1301.092687
P577
2001-07-01T00:00:00Z