Aromatic hydrocarbon receptor-driven Bax gene expression is required for premature ovarian failure caused by biohazardous environmental chemicals. - Wikidata - wikimedia - TriplyDB

Aromatic hydrocarbon receptor-driven Bax gene expression is required for premature ovarian failure caused by biohazardous environmental chemicals.

Aromatic hydrocarbon receptor-driven Bax gene expression is required for premature ovarian failure caused by biohazardous environmental chemicals.

http://www.wikidata.org/entity/Q43675933

about

Overview of AHR functional domains and the classical AHR signaling pathway: induction of drug-metabolizing enzymes Cables1 protects p63 from proteasomal degradation to ensure deletion of cells after genotoxic stress Oocyte formation by mitotically active germ cells purified from ovaries of reproductive-age women In vivo delivery of FTY720 prevents radiation-induced ovarian failure and infertility in adult female nonhuman primates Xenobiotic effects on ovarian preantral follicles Ovarian follicle counts--not as simple as 1, 2, 3 Understanding the human health effects of chemical mixtures.Menstrual function among women exposed to polybrominated biphenyls: a follow-up prevalence study Serum dioxin concentrations and age at menopause.The role of autoimmunity in premature ovarian failure Genetics of the ovarian reserve Aryl hydrocarbon receptor control of adaptive immunity Synergistic Anticancer Effect of Tocotrienol Combined with Chemotherapeutic Agents or Dietary Components: A Review Effects of cigarette smoking on reproduction Protective role for ovarian glutathione S-transferase isoform pi during 7,12-dimethylbenz[a]anthracene-induced ovotoxicity Ah Receptor Pathway Intricacies; Signaling Through Diverse Protein Partners and DNA-Motifs Effect of CYP2E1 gene deletion in mice on expression of microsomal epoxide hydrolase in response to VCD exposure Metabolism of the environmental toxicant benzo(a)pyrene by subcellular fractions of human ovary Exactly the same but different: promiscuity and diversity in the molecular mechanisms of action of the aryl hydrocarbon (dioxin) receptor The impact of smoking on antimüllerian hormone levels in women aged 38 to 50 years Effects of deletion of the transcription factor Nrf2 and benzo [a]pyrene treatment on ovarian follicles and ovarian surface epithelial cells in mice Enhancing survival of mouse oocytes following chemotherapy or aging by targeting Bax and Rad51 Chemotherapy-induced late transgenerational effects in mice Black tattoos entail substantial uptake of genotoxicpolycyclic aromatic hydrocarbons (PAH) in human skin and regional lymph nodes.Maternal exposure to polycyclic aromatic hydrocarbons diminishes murine ovarian reserve via induction of Harakiri Cosmetics use and age at menopause: is there a connection?Discrimination between thermodynamic models of cis-regulation using transcription factor occupancy data.Environment-specific combinatorial cis-regulation in synthetic promoters.Aged mouse ovaries possess rare premeiotic germ cells that can generate oocytes following transplantation into a young host environment.TCDD and a putative endogenous AhR ligand, ITE, elicit the same immediate changes in gene expression in mouse lung fibroblasts.Primary peripheral T cells become susceptible to 2,3,7,8-tetrachlorodibenzo-p-dioxin-mediated apoptosis in vitro upon activation and in the presence of dendritic cells Active and passive smoking and fecundability in Danish pregnancy planners.Human primordial germ cell formation is diminished by exposure to environmental toxicants acting through the AHR signaling pathway Constitutive expression of aryl hydrocarbon receptor in keratinocytes causes inflammatory skin lesions Effects of maternal smoking on the placental expression of genes related to angiogenesis and apoptosis during the first trimester CH223191 is a ligand-selective antagonist of the Ah (Dioxin) receptor An evolutionarily conserved role for the aryl hydrocarbon receptor in the regulation of movement.Dietary polyphenols increase paraoxonase 1 gene expression by an aryl hydrocarbon receptor-dependent mechanism.The aryl hydrocarbon receptor directs hematopoietic progenitor cell expansion and differentiation.Tamoxifen decreases ovarian follicular loss from experimental toxicant DMBA and chemotherapy agents cyclophosphamide and doxorubicin in the rat.

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Aromatic hydrocarbon receptor-driven Bax gene expression is required for premature ovarian failure caused by biohazardous environmental chemicals.

http://www.wikidata.org/entity/Q43675933

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Casper RF

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355-360

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