Dramatic remodeling of advanced atherosclerotic plaques of the apolipoprotein E-deficient mouse in a novel transplantation model.
about
High density lipoprotein and metabolic disease: Potential benefits of restoring its functional propertiesRegression of atherosclerosis: insights from animal and clinical studiesInflammatory cell recruitment in cardiovascular disease: murine models and potential clinical applicationsStatins promote the regression of atherosclerosis via activation of the CCR7-dependent emigration pathway in macrophagesDeletion of ABCA1 and ABCG1 impairs macrophage migration because of increased Rac1 signaling.ACAT inhibition reduces the progression of preexisting, advanced atherosclerotic mouse lesions without plaque or systemic toxicity.Laser capture microdissection analysis of gene expression in macrophages from atherosclerotic lesions of apolipoprotein E-deficient mice.HDL induces the expression of the M2 macrophage markers arginase 1 and Fizz-1 in a STAT6-dependent process.Gene expression changes in foam cells and the role of chemokine receptor CCR7 during atherosclerosis regression in ApoE-deficient mice.Mechanisms that regulate macrophage burden in atherosclerosis.CD36, a scavenger receptor implicated in atherosclerosis.HDL promotes rapid atherosclerosis regression in mice and alters inflammatory properties of plaque monocyte-derived cells.Early decrease in carotid plaque lipid content as assessed by magnetic resonance imaging during treatment of rosuvastatin.Inhibition and regression of atherosclerotic lesions.LXR promotes the maximal egress of monocyte-derived cells from mouse aortic plaques during atherosclerosis regression.Macrophages in atherosclerosis: a dynamic balance.Aggressive very low-density lipoprotein (VLDL) and LDL lowering by gene transfer of the VLDL receptor combined with a low-fat diet regimen induces regression and reduces macrophage content in advanced atherosclerotic lesions in LDL receptor-deficienPlasma cholesterol-induced lesion networks activated before regression of early, mature, and advanced atherosclerosis.Carotid plaque regression following 6-month statin therapy assessed by 3T cardiovascular magnetic resonance: comparison with ultrasound intima media thicknessHigh density lipoprotein stimulated migration of macrophages depends on the scavenger receptor class B, type I, PDZK1 and Akt1 and is blocked by sphingosine 1 phosphate receptor antagonists.Effects of High Fat Feeding and Diabetes on Regression of Atherosclerosis Induced by Low-Density Lipoprotein Receptor Gene Therapy in LDL Receptor-Deficient MiceThe in and out of monocytes in atherosclerotic plaques: Balancing inflammation through migration.Molecular, cellular and functional imaging of atherothrombosis.Mouse model of alloimmune-induced vascular rejection and transplant arteriosclerosis.Macrophages, dendritic cells, and regression of atherosclerosis.Mechanisms of disease: macrophage-derived foam cells emerging as therapeutic targets in atherosclerosis.Interleukin-13 protects from atherosclerosis and modulates plaque composition by skewing the macrophage phenotype.Prospects for atherosclerosis regression through increase in high-density lipoprotein and other emerging therapeutic targetsEarly changes in arterial structure and function following statin initiation: quantification by magnetic resonance imagingAdeno-associated virus serotype 8 gene therapy leads to significant lowering of plasma cholesterol levels in humanized mouse models of homozygous and heterozygous familial hypercholesterolemia.Preclinical mouse models and methods for the discovery of the causes and treatments of atherosclerosisLymphatic vasculature mediates macrophage reverse cholesterol transport in miceCD36 modulates migration of mouse and human macrophages in response to oxidized LDL and may contribute to macrophage trapping in the arterial intima.Emigration of monocyte-derived cells to lymph nodes during resolution of inflammation and its failure in atherosclerosisInducible ApoE gene repair in hypomorphic ApoE mice deficient in the low-density lipoprotein receptor promotes atheroma stabilization with a human-like lipoprotein profile.Emigration of monocyte-derived cells from atherosclerotic lesions characterizes regressive, but not progressive, plaques.The role of a murine transplantation model of atherosclerosis regression in drug discovery.Macrophages, atherosclerosis and the potential of netrin-1 as a novel target for future therapeutic intervention.Atherosclerosis regression and high-density lipoproteins.Dendritic cells in human atherosclerosis: from circulation to atherosclerotic plaques
P2860
Q26750603-2773D96D-12EB-4451-A948-7A593322F3ACQ27028112-769D741B-F526-4FAC-8AE0-D9C6B990C8DEQ28275259-AA1C7B65-E17B-4D3A-8DA5-D5DBEC0F36ECQ28741815-340CDDA7-4B96-4E25-9DF3-75A5AEBEB323Q30500307-ED0D63E6-D209-4B40-B5BF-BEBA9BA192B3Q30538414-17EE374D-4374-4892-9E11-F795184CC099Q31038590-313FC29E-7970-4F67-8E86-B37A44B24572Q31134072-C170D49C-F79C-44C8-B98C-EDC877AF12E3Q33236495-6DDECF1B-1289-4420-A549-04E73C64F607Q33761114-765AD58F-52F6-40BD-A9C8-5F523AB83A89Q33844864-D6808B94-C4F5-4CDA-B24B-0EF82F542DBCQ33868782-44407EF5-9B40-4911-8C97-9B6AFA2A8B0CQ33936780-3F957CE0-0026-4914-8DFA-410313D9D56AQ34335187-F14DCEE6-F7EB-41D7-8AEE-BD6D52C9F528Q34360087-6BFC4745-97FC-44CF-9EBB-BA7883396F02Q34368005-83C9411A-E754-491A-9919-3C4B70AE39FDQ35088381-9E1FC468-9BFC-448B-AF60-AF09D071B891Q35105781-AB9D7D94-0845-4599-A46C-BBF722F2D6AEQ35196130-03CE9692-1747-4559-A025-9BCD8F839D7DQ35238369-90BCCF3E-A49D-4FBB-94AB-0580C6B678FAQ35653968-94A4D81A-D33A-4998-829B-899AB7B0F56DQ35855146-A36759B1-F7E0-4D77-B28B-2FE4534EE52FQ35936423-17F2ED57-C82D-4087-A79D-E6275CBFEA14Q35975238-81952CF8-7375-4178-8AC3-705A1C42AE43Q36193752-CE7D09E2-E689-48E9-ADB6-8C9B599AE052Q36304339-2F2E363F-580E-4BA5-B208-108FD22429EAQ36379539-ACAA36E1-45BF-4F36-9377-1A343D45D858Q36383197-0C1D5962-1D98-472C-9F97-E34C236F561FQ36537985-ECC7ED3C-82D0-4569-BF12-F06C055E8D2FQ36562652-C260DD6C-649D-4518-9788-B6D765858057Q36728741-93C70E8F-BDDB-449F-8E33-C1A84815A120Q36733402-25680C0C-BDA8-4C35-8497-99584C50F905Q37036080-2147E392-789D-42E4-AA19-B8C295C0B439Q37260168-24E40815-4A49-429D-ABDA-D8D11DC7ED72Q37262945-1AFA0753-1E66-486F-B187-5AEC997BB83EQ37416056-2BE67553-7317-4533-B376-0749B759DC61Q37427608-E6435AA1-64A2-4FC1-8E50-B14C5DAFA7CDQ37576679-100CFADD-D972-4701-AC77-8C38B3482952Q37786650-A52E40BF-27C7-4654-A1E7-FE7427374E1FQ37942858-00825DDC-6D82-413B-89E8-B7F92D89BD81
P2860
Dramatic remodeling of advanced atherosclerotic plaques of the apolipoprotein E-deficient mouse in a novel transplantation model.
description
2001 nî lūn-bûn
@nan
2001年の論文
@ja
2001年学术文章
@wuu
2001年学术文章
@zh-cn
2001年学术文章
@zh-hans
2001年学术文章
@zh-my
2001年学术文章
@zh-sg
2001年學術文章
@yue
2001年學術文章
@zh
2001年學術文章
@zh-hant
name
Dramatic remodeling of advance ...... a novel transplantation model.
@en
Dramatic remodeling of advance ...... a novel transplantation model.
@nl
type
label
Dramatic remodeling of advance ...... a novel transplantation model.
@en
Dramatic remodeling of advance ...... a novel transplantation model.
@nl
prefLabel
Dramatic remodeling of advance ...... a novel transplantation model.
@en
Dramatic remodeling of advance ...... a novel transplantation model.
@nl
P2093
P356
P1476
Dramatic remodeling of advance ...... a novel transplantation model
@en
P2093
P304
P356
10.1067/MVA.2001.115963
P577
2001-09-01T00:00:00Z