Na(+)-K(+)-ATPase inhibition and depolarization induce glutamate release via reverse Na(+)-dependent transport in spinal cord white matter.
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Temporal and topographic alterations in expression of the alpha3 isoform of Na+, K(+)-ATPase in the rat freeze lesion model of microgyria and epileptogenesisThe Na,K-ATPase alpha 2 isoform is expressed in neurons, and its absence disrupts neuronal activity in newborn miceWallerian-like axonal degeneration in the optic nerve after excitotoxic retinal insult: an ultrastructural study.Ionic storm in hypoxic/ischemic stress: can opioid receptors subside it?Pathology dynamics predict spinal cord injury therapeutic successPhysical and functional interaction of NCX1 and EAAC1 transporters leading to glutamate-enhanced ATP production in brain mitochondriaChanges in gap junction expression and function following ischemic injury of spinal cord white matterTransporters for L-glutamate: an update on their molecular pharmacology and pathological involvement.The pathophysiology of ischemic injury to developing white matterNeuronal activity and glutamate uptake decrease mitochondrial mobility in astrocytes and position mitochondria near glutamate transportersIon channel blockers and spinal cord injury.Taming glutamate excitotoxicity: strategic pathway modulation for neuroprotection.Anthocyanins control neuroinflammation and consequent memory dysfunction in mice exposed to lipopolysaccharide.Riluzole blocks perioperative ischemia-reperfusion injury and enhances postdecompression outcomes in cervical spondylotic myelopathy.The loss-of-function disease-mutation G301R in the Na+/K+-ATPase α2 isoform decreases lesion volume and improves functional outcome after acute spinal cord injury in mice.In vitro studies of the influence of glutamatergic agonists on the Na+,K(+)-ATPase and K(+)-p-nitrophenylphosphatase activities in the hippocampus and frontal cortex of rats.Experimental and modeling studies of novel bursts induced by blocking na(+) pump and synaptic inhibition in the rat spinal cord.Mechanism of acute ischemic injury of oligodendroglia in early myelinating white matter: the importance of astrocyte injury and glutamate release.Acute creatine administration improves mitochondrial membrane potential and protects against pentylenetetrazol-induced seizures.Rats with different thresholds to clonic convulsions induced by DMCM differ in the binding of [3H]-MK-801 and [3H]-ouabain in the membranes of brain regions.Increased expression of GAP43 in interneurons in a rat model of experimental polymicrogyria.Exercise pre-conditioning reduces brain inflammation and protects against toxicity induced by traumatic brain injury: behavioral and neurochemical approach.
P2860
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P2860
Na(+)-K(+)-ATPase inhibition and depolarization induce glutamate release via reverse Na(+)-dependent transport in spinal cord white matter.
description
2001 nî lūn-bûn
@nan
2001年の論文
@ja
2001年学术文章
@wuu
2001年学术文章
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2001年学术文章
@zh-cn
2001年学术文章
@zh-hans
2001年学术文章
@zh-my
2001年学术文章
@zh-sg
2001年學術文章
@yue
2001年學術文章
@zh-hant
name
Na(+)-K(+)-ATPase inhibition a ...... t in spinal cord white matter.
@en
type
label
Na(+)-K(+)-ATPase inhibition a ...... t in spinal cord white matter.
@en
prefLabel
Na(+)-K(+)-ATPase inhibition a ...... t in spinal cord white matter.
@en
P1433
P1476
Na(+)-K(+)-ATPase inhibition a ...... t in spinal cord white matter.
@en
P304
P356
10.1016/S0306-4522(01)00385-2
P407
P577
2001-01-01T00:00:00Z