Mechanism of acute ischemic injury of oligodendroglia in early myelinating white matter: the importance of astrocyte injury and glutamate release.
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Optic neuritis in neuromyelitis optica.Pathophysiology of glia in perinatal white matter injury.Perinatal white matter injury: the changing spectrum of pathology and emerging insights into pathogenetic mechanisms.alpha-Phenyl-n-tert-butyl-nitrone attenuates lipopolysaccharide-induced brain injury and improves neurological reflexes and early sensorimotor behavioral performance in juvenile rats.Bacterial cytolysin during meningitis disrupts the regulation of glutamate in the brain, leading to synaptic damage.An organotypic slice culture model of chronic white matter injury with maturation arrest of oligodendrocyte progenitors.Differential susceptibility to axonopathy in necrotic and non-necrotic perinatal white matter injury.A sublethal dose of TNFalpha potentiates kainate-induced excitotoxicity in optic nerve oligodendrocytes.Disruption of ionic and cell volume homeostasis in cerebral ischemia: The perfect stormAstrocytes and developmental white matter disorders.The pathophysiology of ischemic injury to developing white matterNovel morphological features of developing white matter pericytes and rapid scavenging of reactive oxygen species in the neighbouring endothelia.White matter injury: Ischemic and nonischemic.Ultrastructural Pathology of Oligodendroglial Cells in Traumatic and Hydrocephalic Human Brain Edema: A Review.Plasmalemmal and mitochondrial Na(+) -Ca(2+) exchange in neuroglia.Glial Na(+) -dependent ion transporters in pathophysiological conditions.Premyelinated central axons express neurotoxic NMDA receptors: relevance to early developing white-matter injury.Hypoxic injury of isolated axons is independent of ionotropic glutamate receptors.P2X7 receptors mediate ischemic damage to oligodendrocytes.Glutamate receptor-mediated ischemic injury of premyelinated central axons.Acute ischemic injury of astrocytes is mediated by Na-K-Cl cotransport and not Ca2+ influx at a key point in white matter development.Functional glutamate transport in rodent optic nerve axons and glia.Conduction block and glial injury induced in developing central white matter by glycine, GABA, noradrenalin, or nicotine, studied in isolated neonatal rat optic nerve.Astrogliosis in EAE spinal cord: derivation from radial glia, and relationships to oligodendroglia.Extracellular amino acids and lipid peroxidation products in periventricular white matter during and after cerebral ischemia in preterm fetal sheep.NMDA receptors are expressed in developing oligodendrocyte processes and mediate injury.The mechanisms of acute ischemic injury in the cell processes of developing white matter astrocytes.Hypoxia-ischemia preferentially triggers glutamate depletion from oligodendroglia and axons in perinatal cerebral white matter.
P2860
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P2860
Mechanism of acute ischemic injury of oligodendroglia in early myelinating white matter: the importance of astrocyte injury and glutamate release.
description
2004 nî lūn-bûn
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2004年の論文
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2004年学术文章
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2004年学术文章
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2004年学术文章
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2004年学术文章
@zh-my
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@zh-sg
2004年學術文章
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name
Mechanism of acute ischemic in ...... injury and glutamate release.
@en
Mechanism of acute ischemic in ...... injury and glutamate release.
@nl
type
label
Mechanism of acute ischemic in ...... injury and glutamate release.
@en
Mechanism of acute ischemic in ...... injury and glutamate release.
@nl
prefLabel
Mechanism of acute ischemic in ...... injury and glutamate release.
@en
Mechanism of acute ischemic in ...... injury and glutamate release.
@nl
P2093
P2860
P356
P1476
Mechanism of acute ischemic in ...... injury and glutamate release.
@en
P2093
Natalie Allcock
Robert Fern
Robert Thomas
Scott Wilke
P2860
P304
P356
10.1093/JNEN/63.8.872
P577
2004-08-01T00:00:00Z