Alterations in mesolimbic dopamine function during the abstinence period following chronic ethanol consumption.
about
Glial cell line-derived neurotrophic factor mediates the desirable actions of the anti-addiction drug ibogaine against alcohol consumptionAcute withdrawal, protracted abstinence and negative affect in alcoholism: are they linked?Novel therapeutic strategies for alcohol and drug addiction: focus on GABA, ion channels and transcranial magnetic stimulationEthanol action on dopaminergic neurons in the ventral tegmental area: interaction with intrinsic ion channels and neurotransmitter inputs.Glial cell line-derived neurotrophic factor reverses alcohol-induced allostasis of the mesolimbic dopaminergic system: implications for alcohol reward and seekingChronic intermittent ethanol exposure reduces presynaptic dopamine neurotransmission in the mouse nucleus accumbens.Microinjection of glycine into the ventral tegmental area selectively decreases ethanol consumption.Effects of alcohol on the membrane excitability and synaptic transmission of medium spiny neurons in the nucleus accumbens.High Frequency Electrical Stimulation of Lateral Habenula Reduces Voluntary Ethanol Consumption in Rats.Sizing up ethanol-induced plasticity: the role of small and large conductance calcium-activated potassium channels.Adolescent Social Isolation as a Model of Heightened Vulnerability to Comorbid Alcoholism and Anxiety Disorders.Neuronal nicotinic acetylcholine receptors as pharmacotherapeutic targets for the treatment of alcohol use disorders.Glial cell line-derived neurotrophic factor (GDNF) is an endogenous protector in the mesolimbic system against excessive alcohol consumption and relapse.The dopamine hypothesis of drug addiction and its potential therapeutic value.Switch from excitatory to inhibitory actions of ethanol on dopamine levels after chronic exposure: Role of kappa opioid receptors.Anhedonia and substance dependence: clinical correlates and treatment options.Enduring effects of chronic ethanol in the CNS: basis for alcoholism.Effects of naltrexone alone and in combination with acamprosate on the alcohol deprivation effect in rats.Impulse activity of midbrain dopamine neurons modulates drug-seeking behavior.Withdrawal from intermittent ethanol exposure increases probability of burst firing in VTA neurons in vitro.Methylphenidate does not increase ethanol consumption in a rat model for attention-deficit hyperactivity disorder-the spontaneously hypertensive rat.Midbrain circuit regulation of individual alcohol drinking behaviors in mice.Remission and resurgence of anxiety-like behavior across protracted withdrawal stages in ethanol-dependent rats.Transgenerational transmission of hedonic behaviors and metabolic phenotypes induced by maternal overnutrition
P2860
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P2860
Alterations in mesolimbic dopamine function during the abstinence period following chronic ethanol consumption.
description
2001 nî lūn-bûn
@nan
2001年の論文
@ja
2001年学术文章
@wuu
2001年学术文章
@zh
2001年学术文章
@zh-cn
2001年学术文章
@zh-hans
2001年学术文章
@zh-my
2001年学术文章
@zh-sg
2001年學術文章
@yue
2001年學術文章
@zh-hant
name
Alterations in mesolimbic dopa ...... g chronic ethanol consumption.
@en
Alterations in mesolimbic dopa ...... g chronic ethanol consumption.
@nl
type
label
Alterations in mesolimbic dopa ...... g chronic ethanol consumption.
@en
Alterations in mesolimbic dopa ...... g chronic ethanol consumption.
@nl
prefLabel
Alterations in mesolimbic dopa ...... g chronic ethanol consumption.
@en
Alterations in mesolimbic dopa ...... g chronic ethanol consumption.
@nl
P2093
P1433
P1476
Alterations in mesolimbic dopa ...... ng chronic ethanol consumption
@en
P2093
C P Bailey
H J Little
M J O'Callaghan
S J Manley
P304
P356
10.1016/S0028-3908(01)00146-0
P577
2001-12-01T00:00:00Z