Withdrawal from intermittent ethanol exposure increases probability of burst firing in VTA neurons in vitro.
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Novel therapeutic strategies for alcohol and drug addiction: focus on GABA, ion channels and transcranial magnetic stimulationEthanol action on dopaminergic neurons in the ventral tegmental area: interaction with intrinsic ion channels and neurotransmitter inputs.Diversity of Dopaminergic Neural Circuits in Response to Drug ExposureSustained N-methyl-d-aspartate receptor hypofunction remodels the dopamine system and impairs phasic signaling.Disruption of dopamine neuron activity pattern regulation through selective expression of a human KCNN3 mutation.Cocaine sensitization increases I h current channel subunit 2 (HCN₂) protein expression in structures of the mesocorticolimbic system.Reduced nucleus accumbens SK channel activity enhances alcohol seeking during abstinence.The small-conductance calcium-activated potassium channel is a key modulator of firing and long-term depression in the dorsal striatum.Decreased sensitivity of NMDA receptors on dopaminergic neurons from the posterior ventral tegmental area following chronic nondependent alcohol consumptionAlpha-2 noradrenergic receptor activation inhibits the hyperpolarization-activated cation current (Ih) in neurons of the ventral tegmental area.Phasic dopamine release in appetitive behaviors and drug addiction.Intrinsic neuronal plasticity in the juxtacapsular nucleus of the bed nuclei of the stria terminalis (jcBNST)A review of the interactions between alcohol and the endocannabinoid system: implications for alcohol dependence and future directions for research.Excitability of jcBNST neurons is reduced in alcohol-dependent animals during protracted alcohol withdrawal.HCN Channel Targets for Novel Antidepressant Treatment.Voluntary ethanol intake enhances excitatory synaptic strength in the ventral tegmental area.Strain specific synaptic modifications on ventral tegmental area dopamine neurons after ethanol exposure.In vivo ethanol experience increases D(2) autoinhibition in the ventral tegmental area.Previous ethanol experience enhances synaptic plasticity of NMDA receptors in the ventral tegmental area.Small conductance calcium-activated potassium type 2 channels regulate alcohol-associated plasticity of glutamatergic synapses.Targeting the Small- and Intermediate-Conductance Ca-Activated Potassium Channels: The Drug-Binding Pocket at the Channel/Calmodulin InterfaceMicroRNA-30a-5p in the prefrontal cortex controls the transition from moderate to excessive alcohol consumption.Postsynaptic adenosine A2A receptors modulate intrinsic excitability of pyramidal cells in the rat basolateral amygdala.Glutathione and redox signaling in substance abuse.Alcohol consumption induces global gene expression changes in VTA dopaminergic neuronsCocaine sensitization inhibits the hyperpolarization-activated cation current Ih and reduces cell size in dopamine neurons of the ventral tegmental areaMorphine disinhibits glutamatergic input to VTA dopamine neurons and promotes dopamine neuron excitation.Ethanol increases glutamate neurotransmission in the posterior ventral tegmental area of female wistar rats.Glutamatergic plasticity and alcohol dependence-induced alterations in reward, affect and cognition.Chronic Intermittent Ethanol Exposure Enhances the Excitability and Synaptic Plasticity of Lateral Orbitofrontal Cortex Neurons and Induces a Tolerance to the Acute Inhibitory Actions of Ethanol.Effects of alcohol on the membrane excitability and synaptic transmission of medium spiny neurons in the nucleus accumbens.KCNN Genes that Encode Small-Conductance Ca2+-Activated K+ Channels Influence Alcohol and Drug Addiction.Emerging role for corticotropin releasing factor signaling in the bed nucleus of the stria terminalis at the intersection of stress and reward.Ethanol effects on dopaminergic ventral tegmental area neurons during block of Ih: involvement of barium-sensitive potassium currents.Repeated cocaine exposure increases fast-spiking interneuron excitability in the rat medial prefrontal cortex.Developmental sensitivity of hippocampal interneurons to ethanol: involvement of the hyperpolarization-activated current, IhSizing up ethanol-induced plasticity: the role of small and large conductance calcium-activated potassium channels.Ethanol and acetaldehyde action on central dopamine systems: mechanisms, modulation, and relationship to stressIncreased small conductance calcium-activated potassium type 2 channel-mediated negative feedback on N-methyl-D-aspartate receptors impairs synaptic plasticity following context-dependent sensitization to morphine.Glutamate plasticity in the drunken amygdala: the making of an anxious synapse.
P2860
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P2860
Withdrawal from intermittent ethanol exposure increases probability of burst firing in VTA neurons in vitro.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年学术文章
@wuu
2007年学术文章
@zh-cn
2007年学术文章
@zh-hans
2007年学术文章
@zh-my
2007年学术文章
@zh-sg
2007年學術文章
@yue
2007年學術文章
@zh
2007年學術文章
@zh-hant
name
Withdrawal from intermittent e ...... iring in VTA neurons in vitro.
@en
Withdrawal from intermittent e ...... iring in VTA neurons in vitro.
@nl
type
label
Withdrawal from intermittent e ...... iring in VTA neurons in vitro.
@en
Withdrawal from intermittent e ...... iring in VTA neurons in vitro.
@nl
prefLabel
Withdrawal from intermittent e ...... iring in VTA neurons in vitro.
@en
Withdrawal from intermittent e ...... iring in VTA neurons in vitro.
@nl
P2093
P2860
P356
P1476
Withdrawal from intermittent e ...... iring in VTA neurons in vitro.
@en
P2093
Antonello Bonci
Billy T Chen
F Woodward Hopf
M Scott Bowers
Maysha M Mohamedi
Miquel Martin
P2860
P304
P356
10.1152/JN.00824.2007
P407
P577
2007-08-15T00:00:00Z