Hypercholesterolemia promotes a CD36-dependent and endothelial nitric-oxide synthase-mediated vascular dysfunction. - Wikidata - wikimedia - TriplyDB

Hypercholesterolemia promotes a CD36-dependent and endothelial nitric-oxide synthase-mediated vascular dysfunction.

Hypercholesterolemia promotes a CD36-dependent and endothelial nitric-oxide synthase-mediated vascular dysfunction.

http://www.wikidata.org/entity/Q43971228

about

CD36: implications in cardiovascular disease Molecular basis of human CD36 gene mutations CD36 deficiency leads to choroidal involution via COX2 down-regulation in rodents CD36, a scavenger receptor involved in immunity, metabolism, angiogenesis, and behavior.Type 2 scavenger receptor CD36 in platelet activation: the role of hyperlipemia and oxidative stress.Oxidized LDL: diversity, patterns of recognition, and pathophysiology.Mitral and tufted cells are potential cellular targets of nitration in the olfactory bulb of aged mice.Impact of oxLDL on Cholesterol-Rich Membrane Rafts Identification of renal Cd36 as a determinant of blood pressure and risk for hypertension.HDL-associated estradiol stimulates endothelial NO synthase and vasodilation in an SR-BI-dependent manner.Endothelial nitric oxide synthase, caveolae and the development of atherosclerosis.Lipids: potential regulators of nitric oxide generation.CD36 as a therapeutic target for endothelial dysfunction in stroke.Novel biological functions of high-density lipoprotein cholesterol CD36 and Na/K-ATPase-α1 form a proinflammatory signaling loop in kidney.oxLDL facilitates flow-induced realignment of aortic endothelial cells.Enhancing cardiovascular dynamics by inhibition of thrombospondin-1/CD47 signaling.Microarray analysis reveals novel gene expression changes associated with erectile dysfunction in diabetic rats.Cholesterol and Kir channels.Caveolae, caveolin, and cavins: potential targets for the treatment of cardiac disease.Role of phospholipid oxidation products in atherosclerosis.MicroRNAs in the onset and development of cardiovascular disease.Role of free radical in atherosclerosis, diabetes and dyslipidaemia: larger-than-life.Sarcolemmal dependence of cardiac protection and stress-resistance: roles in aged or diseased hearts.Alteration of actin dependent signaling pathways associated with membrane microdomains in hyperlipidemia.Effect of endothelial cell-based iNOS gene transfer on cavernosal eNOS expression and mouse erectile responses.Reduction of blood pressure, plasma cholesterol, and atherosclerosis by elevated endothelial nitric oxide.OxLDL increases endothelial stiffness, force generation, and network formation.

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P2860

Hypercholesterolemia promotes a CD36-dependent and endothelial nitric-oxide synthase-mediated vascular dysfunction.

http://www.wikidata.org/entity/Q43971228

description

2002 nî lūn-bûn

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2002年の論文

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2002年学术文章

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Hypercholesterolemia promotes ...... mediated vascular dysfunction.

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Hypercholesterolemia promotes ...... mediated vascular dysfunction.

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Hypercholesterolemia promotes ...... mediated vascular dysfunction.

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Hypercholesterolemia promotes ...... mediated vascular dysfunction.

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Hypercholesterolemia promotes ...... mediated vascular dysfunction.

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P1433

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P1476

Hypercholesterolemia promotes ...... mediated vascular dysfunction.

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Annette Uittenbogaard

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Eric J Smart

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James Dressman

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Jeanie F Kincer

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Ling Guo

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Maria Febbraio

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Theresa M Guerin

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P2860

High density lipoprotein binding to scavenger receptor, Class B, type I activates endothelial nitric-oxide synthase in a ceramide-dependent manner

Targeted disruption of the class B scavenger receptor CD36 protects against atherosclerotic lesion development in mice

Cleavage of structural proteins during the assembly of the head of bacteriophage T4

Signal transduction of eNOS activation

Endothelial nitric oxide synthase is regulated by tyrosine phosphorylation and interacts with caveolin-1

Transgenic rescue of defective Cd36 ameliorates insulin resistance in spontaneously hypertensive rats

Endothelial nitric oxide synthase targeting to caveolae. Specific interactions with caveolin isoforms in cardiac myocytes and endothelial cells

Cholesterol depletion disrupts caveolae and insulin receptor signaling for metabolic control via insulin receptor substrate-1, but not for mitogen-activated protein kinase control.

Localization of phospholipase C-gamma1 signaling in caveolae: importance in EGF-induced phosphoinositide hydrolysis but not in tyrosine phosphorylation.

Endothelium-derived nitric oxide and vascular physiology and pathology.

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23525-23533

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10.1074/JBC.M202465200

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26

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277

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11393329

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11976335

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P921

hypercholesterolemia