Production of superoxide through NADH oxidase in thick ascending limb of Henle's loop in rat kidney. - Wikidata - wikimedia - TriplyDB

Production of superoxide through NADH oxidase in thick ascending limb of Henle's loop in rat kidney.

Production of superoxide through NADH oxidase in thick ascending limb of Henle's loop in rat kidney.

http://www.wikidata.org/entity/Q43984535

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NADPH oxidase as a therapeutic target for oxalate induced injury in kidneys O(2) affinity of cross-linked hemoglobins modifies O(2) metabolism in proximal tubules Activation of NAD(P)H oxidase by outward movements of H+ ions in renal medullary thick ascending limb of Henle Early degenerative effects of diabetes mellitus on pancreas, liver, and kidney in rats: an immunohistochemical study.Hydroethidine- and MitoSOX-derived red fluorescence is not a reliable indicator of intracellular superoxide formation: another inconvenient truth Multiphoton imaging of the functioning kidney.Enhanced amiloride-sensitive superoxide production in renal medullary thick ascending limb of Dahl salt-sensitive rats.Proton channels and renal hypertensive injury: a key piece of the Dahl salt-sensitive rat puzzle?Rac1 mediates NaCl-induced superoxide generation in the thick ascending limb.Regulation of macromolecular modulators of urinary stone formation by reactive oxygen species: transcriptional study in an animal model of hyperoxaluria.Molecular diversity and regulation of renal potassium channels.PKC-alpha mediates flow-stimulated superoxide production in thick ascending limbs.Angiotensin II stimulates thick ascending limb superoxide production via protein kinase C(α)-dependent NADPH oxidase activation Nitric oxide and superoxide transport in a cross section of the rat outer medulla. I. Effects of low medullary oxygen tension.Nitric oxide and superoxide transport in a cross section of the rat outer medulla. II. Reciprocal interactions and tubulovascular cross talk.Reactive oxygen species, inflammation and calcium oxalate nephrolithiasis.NAD(P)H oxidase and renal epithelial ion transport HV1 acts as a sodium sensor and promotes superoxide production in medullary thick ascending limb of Dahl salt-sensitive rats NADPH oxidases in cardiovascular disease: insights from in vivo models and clinical studies.Modulation of outer medullary NaCl transport and oxygenation by nitric oxide and superoxide Impacts of nitric oxide and superoxide on renal medullary oxygen transport and urine concentration.Redox regulation of the afferent arteriole and tubuloglomerular feedback.Reactive oxygen species and molecular regulation of renal oxygenation.Increase of sodium delivery stimulates the mitochondrial respiratory chain H2O2 production in rat renal medullary thick ascending limb Codon-Optimized NADH Oxidase Gene Expression and Gene Fusion with Glycerol Dehydrogenase for Bienzyme System with Cofactor Regeneration.Increased expression of NAD(P)H oxidase subunit p67(phox) in the renal medulla contributes to excess oxidative stress and salt-sensitive hypertension.Protein kinase C-dependent NAD(P)H oxidase activation induced by type 1 diabetes in renal medullary thick ascending limb.Angiotensin II stimulates superoxide production in the thick ascending limb by activating NOX4.NADPH oxidase 4 mediates flow-induced superoxide production in thick ascending limbs.Impact of nitric oxide-mediated vasodilation on outer medullary NaCl transport and oxygenation.Angiotensin II stimulates superoxide production by nitric oxide synthase in thick ascending limbs.Renal oxygen delivery: matching delivery to metabolic demand.Characterization and Activation of NLRP3 Inflammasomes in the Renal Medulla in Mice.Angiotensin II stimulates basolateral 10-pS Cl channels in the thick ascending limb.The reactive nitrogen species peroxynitrite is a potent inhibitor of renal Na-K-ATPase activity Redox control of renal function and hypertension.Modulation of pressure-natriuresis by renal medullary reactive oxygen species and nitric oxide.Renal medullary oxidative stress, pressure-natriuresis, and hypertension.NOX enzymes and diabetic complications.Nitric oxide reduces flow-induced superoxide production via cGMP-dependent protein kinase in thick ascending limbs.

P2860

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P2860

Production of superoxide through NADH oxidase in thick ascending limb of Henle's loop in rat kidney.

http://www.wikidata.org/entity/Q43984535

description

2002 nî lūn-bûn

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2002年の論文

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2002年学术文章

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2002年學術文章

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2002年學術文章

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Production of superoxide throu ...... of Henle's loop in rat kidney.

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Production of superoxide throu ...... of Henle's loop in rat kidney.

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Production of superoxide throu ...... of Henle's loop in rat kidney.

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Production of superoxide throu ...... of Henle's loop in rat kidney.

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Ai-Ping Zou

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Carol A Bobrowitz

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Fu-Xian Yi

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Jamie L Spurrier

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Ningjun Li

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P2860

Distribution of angiotensin AT1 and AT2 receptor subtypes in the rat kidney

p22phox is a critical component of the superoxide-generating NADH/NADPH oxidase system and regulates angiotensin II-induced hypertrophy in vascular smooth muscle cells

Sodium-potassium-adenosinetriphosphatase-dependent sodium transport in the kidney: hormonal control

Mitochondrial free radical generation, oxidative stress, and aging

NAD(P)H oxidase: role in cardiovascular biology and disease

alpha(2)-adrenergic receptor-mediated increase in NO production buffers renal medullary vasoconstriction.

Production and actions of superoxide in the renal medulla.

Role of NAD(P)H oxidase in angiotensin II-induced JAK/STAT signaling and cytokine induction.

Angiotensin II-induced superoxide anion generation in human vascular endothelial cells: role of membrane-bound NADH-/NADPH-oxidases.

A central role for the endothelial NADPH oxidase in atherosclerosis

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