Tauroursodeoxycholic acid reduces endoplasmic reticulum stress, acinar cell damage, and systemic inflammation in acute pancreatitis.
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Ca2+ toxicity and mitochondrial damage in acute pancreatitis: translational overviewThe Unexpected Uses of Urso- and Tauroursodeoxycholic Acid in the Treatment of Non-liver DiseasesEndoplasmic reticulum stress and oxidative stress in cell fate decision and human disease.Administration of Tauroursodeoxycholic Acid Attenuates Early Brain Injury via Akt Pathway ActivationhCG-induced endoplasmic reticulum stress triggers apoptosis and reduces steroidogenic enzyme expression through activating transcription factor 6 in Leydig cells of the testisSoluble Epoxide Hydrolase Pharmacological Inhibition Ameliorates Experimental Acute Pancreatitis in Mice.Decreased hepatotoxic bile acid composition and altered synthesis in progressive human nonalcoholic fatty liver disease.Effect of a chemical chaperone, tauroursodeoxycholic acid, on HDM-induced allergic airway disease.General control nonderepressible 2 deletion predisposes to asparaginase-associated pancreatitis in mice.The role of protein synthesis and digestive enzymes in acinar cell injury.The unfolded protein response in fatty liver disease.Modulation of mitochondrial dysfunction and endoplasmic reticulum stress are key mechanisms for the wide-ranging actions of epoxy fatty acids and soluble epoxide hydrolase inhibitors.Necroptosis: a potential, promising target and switch in acute pancreatitis.Application of Tauroursodeoxycholic Acid for Treatment of Neurological and Non-neurological Diseases: Is There a Potential for Treating Traumatic Brain Injury?Endoplasmic reticulum stress is activated in acute pancreatitis.Lipid signaling and lipotoxicity in metaflammation: indications for metabolic disease pathogenesis and treatment.The effect of pioglitazone on aldosterone and cortisol production in HAC15 human adrenocortical carcinoma cells.Exendin-4 protects HUVECs from tunicamycin-induced apoptosis via inhibiting the IRE1a/JNK/caspase-3 pathway.Chemical chaperone, TUDCA unlike PBA, mitigates protein aggregation efficiently and resists ER and non-ER stress induced HepG2 cell death.A Carboxyl Ester Lipase (CEL) Mutant Causes Chronic Pancreatitis by Forming Intracellular Aggregates That Activate Apoptosis.Effect of emodin on endoplasmic reticulum stress in rats with severe acute pancreatitis.Tauroursodeoxycholic acid inhibits intestinal inflammation and barrier disruption in NAFLD mice.Developmental competence of bovine early embryos depends on the coupled response between oxidative and endoplasmic reticulum stress.Induction of autophagy promotes preattachment development of bovine embryos by reducing endoplasmic reticulum stress.Tauroursodeoxycholic Acid Protects Nucleus Pulposus Cells from Compression-Induced Apoptosis and Necroptosis via Inhibiting Endoplasmic Reticulum Stress.Ptf1a inactivation in adult pancreatic acinar cells causes apoptosis through activation of the endoplasmic reticulum stress pathway
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P2860
Tauroursodeoxycholic acid reduces endoplasmic reticulum stress, acinar cell damage, and systemic inflammation in acute pancreatitis.
description
2011 nî lūn-bûn
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2011年の論文
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2011年学术文章
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2011年学术文章
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2011年学术文章
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2011年学术文章
@zh-hans
2011年学术文章
@zh-my
2011年学术文章
@zh-sg
2011年學術文章
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2011年學術文章
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name
Tauroursodeoxycholic acid redu ...... mmation in acute pancreatitis.
@en
Tauroursodeoxycholic acid redu ...... mmation in acute pancreatitis.
@nl
type
label
Tauroursodeoxycholic acid redu ...... mmation in acute pancreatitis.
@en
Tauroursodeoxycholic acid redu ...... mmation in acute pancreatitis.
@nl
prefLabel
Tauroursodeoxycholic acid redu ...... mmation in acute pancreatitis.
@en
Tauroursodeoxycholic acid redu ...... mmation in acute pancreatitis.
@nl
P2093
P2860
P356
P1476
Tauroursodeoxycholic acid redu ...... mmation in acute pancreatitis.
@en
P2093
Antje Malo
Burkhard Göke
Claus Schäfer
Constanze H Kubisch
Ersin Seyhun
Ralf-Thorsten Hoffmann
P2860
P304
P356
10.1152/AJPGI.00483.2010
P577
2011-07-21T00:00:00Z