Chronic renal hypoxia after acute ischemic injury: effects of L-arginine on hypoxia and secondary damage. - Wikidata - wikimedia - TriplyDB

Chronic renal hypoxia after acute ischemic injury: effects of L-arginine on hypoxia and secondary damage.

Chronic renal hypoxia after acute ischemic injury: effects of L-arginine on hypoxia and secondary damage.

http://www.wikidata.org/entity/Q44185748

about

Hypoxia: The Force that Drives Chronic Kidney Disease Renal endothelial dysfunction in acute kidney ischemia reperfusion injury Matrix metalloproteinases in kidney homeostasis and diseases Severe renal mass reduction impairs recovery and promotes fibrosis after AKI Evaluation of Short-Term Changes in Serum Creatinine Level as a Meaningful End Point in Randomized Clinical Trials.Follow-up of Acute kidney injury in Neonates during Childhood Years (FANCY): a prospective cohort study.Progression after AKI: Understanding Maladaptive Repair Processes to Predict and Identify Therapeutic Treatments.Recognition and reporting of AKI in very low birth weight infants Pathogenesis and prognosis of thrombotic microangiopathy.Recovery from renal ischemia-reperfusion injury is associated with altered renal hemodynamics, blunted pressure natriuresis, and sodium-sensitive hypertension.Human adipose stromal cell therapy improves survival and reduces renal inflammation and capillary rarefaction in acute kidney injury.Long-term risk of chronic kidney disease and mortality in children after acute kidney injury: a systematic review.Impaired endothelial proliferation and mesenchymal transition contribute to vascular rarefaction following acute kidney injury.MMP-9 gene deletion mitigates microvascular loss in a model of ischemic acute kidney injury.Renal oxygenation in acute renal ischemia-reperfusion injury.Peritubular capillary loss after mouse acute nephrotoxicity correlates with down-regulation of vascular endothelial growth factor-A and hypoxia-inducible factor-1 alpha.The p53 inhibitor pifithrin-α can stimulate fibrosis in a rat model of ischemic acute kidney injury.Distinct effects on long-term function of injured and contralateral kidneys following unilateral renal ischemia-reperfusion Persistent oxidative stress following renal ischemia-reperfusion injury increases ANG II hemodynamic and fibrotic activity Th-17 cell activation in response to high salt following acute kidney injury is associated with progressive fibrosis and attenuated by AT-1R antagonism.Preservation of peritubular capillary endothelial integrity and increasing pericytes may be critical to recovery from postischemic acute kidney injury.Inhibition of Toll-Like Receptor 4 Signaling Mitigates Microvascular Loss but Not Fibrosis in a Model of Ischemic Acute Kidney Injury VEGF-121 preserves renal microvessel structure and ameliorates secondary renal disease following acute kidney injury Renal hypoxia and dysoxia after reperfusion of the ischemic kidney.Intrarenal oxygenation: unique challenges and the biophysical basis of homeostasis.Renal cells from spermatogonial germline stem cells protect against kidney injury Pathophysiology of acute kidney injury The suffocating kidney: tubulointerstitial hypoxia in end-stage renal disease.Perspective in chronic kidney disease: targeting hypoxia-inducible factor (HIF) as potential therapeutic approach.Invasive treatment of resistant hypertension: present and future.Hypoxia as a key player in the AKI-to-CKD transition.Nox and renal disease.Failed Tubule Recovery, AKI-CKD Transition, and Kidney Disease Progression.Transgenic sickle mice are markedly sensitive to renal ischemia-reperfusion injury Urinary C-type natriuretic peptide excretion: a promising biomarker to detect underlying renal injury and remodeling both acutely and chronically.Stress Signal Network between Hypoxia and ER Stress in Chronic Kidney Disease.Progression of Chronic Kidney Disease After Acute Kidney Injury: Role of Self-Perpetuating Versus Hemodynamic-Induced Fibrosis Effect of continuous positive airway pressure on proteinuria in obstructive sleep apnea.Mitochondrial Pathology and Glycolytic Shift during Proximal Tubule Atrophy after Ischemic AKI.Renal ischemia-reperfusion leads to long term infiltration of activated and effector-memory T lymphocytes.

P2860

Q26768865-A7FD0C9A-6986-4774-957C-6AE52999F5EC Q26859947-852A9F8D-D2AE-4237-9BDF-8492F9D96939 Q27004621-1FF12963-94CD-458C-897F-FAFAE6BFA868 Q28388800-21AF9B6B-3097-4DB5-9CC1-EBF5311A84DD Q30251988-1AA6A3D7-4B58-4274-B8C9-292A7D241BBE Q30253047-DD249DCE-B5B9-4948-8B7F-3DEF83C98808 Q30278441-E2F1D99E-0F37-42A8-9E2B-1758E5BAE272 Q30303164-6A9A02F7-306D-4BA8-B8F4-7E7A89D31793 Q33375692-86F63FF4-1A03-40D4-9E7A-5DA62D9DACAC Q33497112-C4325D5D-EEE0-4987-8A48-361EA06CC791 Q33844666-7CE832A4-4BE7-4197-9D39-FC8717DA40EB Q34611452-B2E9BEAD-53DF-428C-AC0E-381F8F0130F8 Q34719308-6B2D3348-A20E-4C03-BF7F-DE42D735DCB9 Q35086941-59C6174C-8C85-4A2F-8C96-B5C77B7CE522 Q35112037-94532B57-0777-4079-97C0-E75095E01F3D Q35843371-72A96B24-59BD-4BB1-B917-365BB962E2C2 Q35924490-9D71D96D-9D26-4308-9F83-689DF5021C14 Q35988997-18F64B42-473C-4DB3-BC53-80A681A7289E Q36042174-E33DF5B1-C399-4E16-BEA9-9F41F1E02B57 Q36108896-AAE4ADB5-4DB6-48E5-BCE6-50919E9349AE Q36844525-57CD1B85-7062-4484-90D2-10A619AFCA7B Q36939146-513CF90C-8BFD-4199-B168-9D862C801ECB Q37018432-D0B36563-0D5D-4938-920F-54F2E9C88A9B Q37167225-60A8B6C3-3B14-42F8-AD9A-DC20944285CC Q37190279-74AFB264-0ED0-4A72-8F64-DEAEBC276F07 Q37524091-FEAD786A-55D6-4ACB-8294-402C7D9A38ED Q37574787-9D7A55F0-354C-4F19-AE9B-9B11A6A5EA6B Q37793828-60206558-47E5-452C-AB5E-AD41EF0634FA Q37977513-08B8F3F8-5418-462E-B464-CD71E52BE0EB Q38245560-DEB9DF63-5FE8-4806-AE5A-571BD3880728 Q38263324-0F6EEB8A-F23F-4975-84B7-B289C0D3075E Q38335626-50544196-B0D5-41B7-A745-A292B6981970 Q38391570-4B000F2B-4B30-41E1-83FE-F136ECF67C52 Q38585776-CEB5F015-AD34-4476-BA62-AFF0BEC6AD1C Q38825225-10C2915F-41CC-44BC-891B-3DE61000191C Q39147185-3A531135-9C82-438F-8E57-99D650A32E34 Q39464387-687E9FE4-541F-4CE4-B778-F71CB5C3F74D Q39720560-529BAF82-D368-4180-B330-039EA4A1A720 Q39901297-234AFA1E-DD8E-4438-A7AC-B8EFC559A053 Q41922589-DE10D3CF-2646-4A5C-BF74-80D6659EDDFB

P2860

Chronic renal hypoxia after acute ischemic injury: effects of L-arginine on hypoxia and secondary damage.

http://www.wikidata.org/entity/Q44185748

description

2002 nî lūn-bûn

@nan

2002年の論文

@ja

2002年学术文章

@wuu

2002年学术文章

@zh

2002年学术文章

@zh-cn

2002年学术文章

@zh-hans

2002年学术文章

@zh-my

2002年学术文章

@zh-sg

2002年學術文章

@yue

2002年學術文章

@zh-hant

name

Chronic renal hypoxia after ac ...... hypoxia and secondary damage.

@en

Chronic renal hypoxia after ac ...... hypoxia and secondary damage.

@nl

type

label

Chronic renal hypoxia after ac ...... hypoxia and secondary damage.

@en

Chronic renal hypoxia after ac ...... hypoxia and secondary damage.

@nl

prefLabel

Chronic renal hypoxia after ac ...... hypoxia and secondary damage.

@en

Chronic renal hypoxia after ac ...... hypoxia and secondary damage.

@nl

P1433

Q44185748-81288D7E-0D04-4C4D-927B-C99EF5000B35

P1476

Q44185748-FDEB938E-0654-46BA-AF89-343FECDDC3F9

P2093

Q44185748-42938CF3-2E72-4222-90C8-CB1936705E39

Q44185748-58DA74CE-C17B-45DD-A21A-C04C8B8182C1

Q44185748-6D796CA7-327E-41C4-8C60-14900EE2A010

Q44185748-E3BBFB70-0723-441B-B977-F76B9CA137FA

P2860

Q44185748-12EE1863-FC54-40DE-81F8-F00CAF7CC5CC

Q44185748-3780B5AB-09A2-4790-B8B7-4F8C62670693

Q44185748-3BD6147C-42E6-4D43-8DF2-148254C8CF9D

Q44185748-40B0E9EA-7284-48E4-947E-37EEC70BEF2D

Q44185748-4135C662-93EB-40A5-8339-E88BEA7CAED3

Q44185748-5ABD5056-63F2-4AD8-B06D-46B2E85D68B8

Q44185748-6061892E-8619-40D8-A39E-7677BF5279E3

Q44185748-7007A2DA-4D65-4099-A867-04C07B73127C

Q44185748-74353517-C0F9-4747-BFA2-A1A4F7E5308D

Q44185748-BCE1B432-6E47-443E-A82F-2C5A46C772CD

P304

Q44185748-C714E406-DCF5-495B-9E7B-AE8107F2E886

P31

Q44185748-49400633-4B1E-4849-822B-28AF29D1124D

P356

Q44185748-B8D4636F-AF66-48D9-996F-0013C815D059

P433

Q44185748-7EEDD981-FD3E-4CB3-AF1A-9A14AA2B08EE

P478

Q44185748-87A4B4B9-581C-487B-874C-7AE047521A89

P577

Q44185748-7DB3CFF5-928C-43D2-B974-06778F4024C0

P698

Q44185748-336A1AAA-66E4-4F73-B4B9-BB004D8B5025

P921

Q44185748-422F0F27-3703-4AD6-BBEE-1589E43153A6

P356

AJPRENAL.00169.2002

P1433

American Journal of Physiology - Renal Physiology

P1476

Chronic renal hypoxia after ac ...... hypoxia and secondary damage.

@en

P2093

David L Mattson

http://www.w3.org/2001/XMLSchema#string

David P Basile

http://www.w3.org/2001/XMLSchema#string

Deborah L Donohoe

http://www.w3.org/2001/XMLSchema#string

Kelly Roethe

http://www.w3.org/2001/XMLSchema#string

P2860

Arginine feeding modifies cyclosporine nephrotoxicity in rats.

Localization of proliferating cell nuclear antigen, vimentin, c-Fos, and clusterin in the postischemic kidney. Evidence for a heterogenous genetic response among nephron segments, and a large pool of mitotically active and dedifferentiated cells.

Role of nitric oxide in renal medullary oxygenation. Studies in isolated and intact rat kidneys.

Hypoxia of the renal medulla--its implications for disease.

The transforming growth factor beta system in kidney disease and repair: recent progress and future directions.

Hypothesis: the role of acquired tubulointerstitial disease in the pathogenesis of salt-dependent hypertension.

Renal vasodilation with L-arginine. Effects of dietary salt.

Reductive metabolism of the hypoxia marker pimonidazole is regulated by oxygen tension independent of the pyridine nucleotide redox state.

Differences in vascular reactivity in models of ischemic acute renal failure.

Delayed graft function: risk factors and implications for renal allograft survival.

P304

F338-48

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1152/AJPRENAL.00169.2002

http://www.w3.org/2001/XMLSchema#string

P433

2

http://www.w3.org/2001/XMLSchema#string

P478

284

http://www.w3.org/2001/XMLSchema#string

P577

2002-10-01T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P698

12388385

http://www.w3.org/2001/XMLSchema#string

P921