Treatment of ischemic brain damage by perturbing NMDA receptor- PSD-95 protein interactions. - Wikidata - wikimedia - TriplyDB

Treatment of ischemic brain damage by perturbing NMDA receptor- PSD-95 protein interactions.

Treatment of ischemic brain damage by perturbing NMDA receptor- PSD-95 protein interactions.

http://www.wikidata.org/entity/Q44193907

about

Dopamine transporter cell surface localization facilitated by a direct interaction with the dopamine D2 receptor A thermodynamic ligand binding study of the third PDZ domain (PDZ3) from the mammalian neuronal protein PSD-95 Human embryonic stem cell-derived neurons as a tool for studying neuroprotection and neurodegeneration Beta-arrestin1 phosphorylation by GRK5 regulates G protein-independent 5-HT4 receptor signalling PDZ domains and their binding partners: structure, specificity, and modification Traumatic brain injury: can the consequences be stopped?Applications and Challenges for Use of Cell-Penetrating Peptides as Delivery Vectors for Peptide and Protein Cargos Imaging of prehospital stroke therapeutics α7 nicotinic ACh receptors as a ligand-gated source of Ca(2+) ions: the search for a Ca(2+) optimum Neuroprotection for stroke: current status and future perspectives Current understanding of TRPM7 pharmacology and drug development for stroke The role of nitric oxide in pre-synaptic plasticity and homeostasis The NMDA receptor complex: a multifunctional machine at the glutamatergic synapse Optogenetic activation of intracellular adenosine A2A receptor signaling in the hippocampus is sufficient to trigger CREB phosphorylation and impair memory Disrupting GluA2-GAPDH Interaction Affects Axon and Dendrite Development Biomimetic divalent ligands for the acute disruption of synaptic AMPAR stabilization Mechanism underlying selective regulation of G protein-gated inwardly rectifying potassium channels by the psychostimulant-sensitive sorting nexin 27 A high-affinity, dimeric inhibitor of PSD-95 bivalently interacts with PDZ1-2 and protects against ischemic brain damage Protein grafting of p53TAD onto a leucine zipper scaffold generates a potent HDM dual inhibitor Tonic GABAA Receptors as Potential Target for the Treatment of Temporal Lobe Epilepsy.Role of NMDA Receptor-Mediated Glutamatergic Signaling in Chronic and Acute Neuropathologies Therapeutic use of PDZ protein-protein interaction antagonism Disruption of PTEN coupling with 5-HT2C receptors suppresses behavioral responses induced by drugs of abuse Inhibition of N-Methyl-D-aspartate-induced Retinal Neuronal Death by Polyarginine Peptides Is Linked to the Attenuation of Stress-induced Hyperpolarization of the Inner Mitochondrial Membrane Potential Synaptic neurotransmission depression in ventral tegmental dopamine neurons and cannabinoid-associated addictive learning DAPK1 interaction with NMDA receptor NR2B subunits mediates brain damage in stroke Key role of tissue plasminogen activator in neurovascular coupling An in silico analysis of the binding modes and binding affinities of small molecule modulators of PDZ-peptide interactions Targeting protein-protein interactions with trimeric ligands: high affinity inhibitors of the MAGUK protein family Schizophrenia, amphetamine-induced sensitized state and acute amphetamine exposure all show a common alteration: increased dopamine D2 receptor dimerization Extrasynaptic NMDA receptors couple preferentially to excitotoxicity via calpain-mediated cleavage of STEP CaMKIIalpha interacts with M4 muscarinic receptors to control receptor and psychomotor function Stress- and mitogen-induced phosphorylation of the synapse-associated protein SAP90/PSD-95 by activation of SAPK3/p38gamma and ERK1/ERK2 Physical interaction between the serotonin transporter and neuronal nitric oxide synthase underlies reciprocal modulation of their activity Glycine induces bidirectional modifications in N-methyl-D-aspartate receptor-mediated synaptic responses in hippocampal CA1 neurons Intracellular protein interaction mapping with FRET hybrids.PDZ domain proteins: plug and play!Temporal dynamics of glyoxalase 1 in secondary neuronal injury Accidental interaction between PDZ domains and diclofenac revealed by NMR-assisted virtual screening.Cell Permeable Peptides: A Promising Tool to Deliver Neuroprotective Agents in the Brain.

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P2860

Treatment of ischemic brain damage by perturbing NMDA receptor- PSD-95 protein interactions.

http://www.wikidata.org/entity/Q44193907

description

2002 nî lūn-bûn

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2002年の論文

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2002年学术文章

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2002年学术文章

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2002年学术文章

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2002年学术文章

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2002年学术文章

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2002年學術文章

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2002年學術文章

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name

Treatment of ischemic brain da ...... - PSD-95 protein interactions.

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Treatment of ischemic brain da ...... - PSD-95 protein interactions.

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type

label

Treatment of ischemic brain da ...... - PSD-95 protein interactions.

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Treatment of ischemic brain da ...... - PSD-95 protein interactions.

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prefLabel

Treatment of ischemic brain da ...... - PSD-95 protein interactions.

@en

Treatment of ischemic brain da ...... - PSD-95 protein interactions.

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James W Gurd

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Lidong Liu

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Mark Arundine

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Michael Tymianski

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Michael W Salter

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Michelle Aarts

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Yitao Liu

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