Roles for Arg- and Lys-gingipains in the disruption of cytokine responses and loss of viability of human endothelial cells by Porphyromonas gingivalis infection.
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Gingipain-dependent interactions with the host are important for survival of Porphyromonas gingivalisGingipains from Porphyromonas gingivalis W83 induce cell adhesion molecule cleavage and apoptosis in endothelial cells.The cysteine protease α-clostripain is not essential for the pathogenesis of Clostridium perfringens-mediated myonecrosis.Dichotomy of gingipains action as virulence factors: from cleaving substrates with the precision of a surgeon's knife to a meat chopper-like brutal degradation of proteins.Porphyromonas gingivalis induces receptor activator of NF-kappaB ligand expression in osteoblasts through the activator protein 1 pathway.Gingipains: Critical Factors in the Development of Aspiration Pneumonia Caused by Porphyromonas gingivalis.Bacterial proteases: targets for diagnostics and therapy.Potential value of a rice protein extract, containing proteinaceous inhibitors against cysteine proteinases from Porphyromonas gingivalis, for managing periodontal diseases.Periodontitis is associated with preeclampsia in pregnant women.Regulation of protease-activated receptor-2 expression in gingival fibroblasts and Jurkat T cells by Porphyromonas gingivalis.Gingipains from Porphyromonas gingivalis synergistically induce the production of proinflammatory cytokines through protease-activated receptors with Toll-like receptor and NOD1/2 ligands in human monocytic cells.Apple- and hop-polyphenols protect periodontal ligament cells stimulated with enamel matrix derivative from Porphyromonas gingivalis.Effect of enamel matrix derivative on periodontal ligament cells in vitro is diminished by Porphyromonas gingivalis.A functional virulence complex composed of gingipains, adhesins, and lipopolysaccharide shows high affinity to host cells and matrix proteins and escapes recognition by host immune systems.Expression levels of novel cytokine IL-32 in periodontitis and its role in the suppression of IL-8 production by human gingival fibroblasts stimulated with Porphyromonas gingivalisEffects of various growth conditions in a chemostat on expression of virulence factors in Porphyromonas gingivalis.Role for gingipains in Porphyromonas gingivalis traffic to phagolysosomes and survival in human aortic endothelial cells.Isolation and characterization of a novel and potent inhibitor of Arg-gingipain from Streptomyces sp. strain FA-70.Porphyromonas gingivalis-induced platelet aggregation in plasma depends on Hgp44 adhesin but not Rgp proteinase.Signal-Regulated Protein Kinases/Protein Kinase B-p53-BH3-Interacting Domain Death Agonist Pathway Regulates Gingipain-Induced Apoptosis in Osteoblasts.Inhibitors of bacterial protease enzymes for periodontal therapy.
P2860
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P2860
Roles for Arg- and Lys-gingipains in the disruption of cytokine responses and loss of viability of human endothelial cells by Porphyromonas gingivalis infection.
description
2002 nî lūn-bûn
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2002年の論文
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2002年学术文章
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2002年学术文章
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2002年学术文章
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name
Roles for Arg- and Lys-gingipa ...... yromonas gingivalis infection.
@en
Roles for Arg- and Lys-gingipa ...... yromonas gingivalis infection.
@nl
type
label
Roles for Arg- and Lys-gingipa ...... yromonas gingivalis infection.
@en
Roles for Arg- and Lys-gingipa ...... yromonas gingivalis infection.
@nl
prefLabel
Roles for Arg- and Lys-gingipa ...... yromonas gingivalis infection.
@en
Roles for Arg- and Lys-gingipa ...... yromonas gingivalis infection.
@nl
P2093
P2860
P356
P1433
P1476
Roles for Arg- and Lys-gingipa ...... yromonas gingivalis infection.
@en
P2093
Atsuyo Baba
Kenji Yamamoto
Tetsuji Asao
Tomoko Kadowaki
P2860
P304
P356
10.1515/BC.2002.135
P577
2002-07-01T00:00:00Z