Increased serum levels of the specific AGE-compound methylglyoxal-derived hydroimidazolone in patients with type 2 diabetes.
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Resveratrol protects mouse oocytes from methylglyoxal-induced oxidative damageHydrogen sulfide releasing aspirin, ACS14, attenuates high glucose-induced increased methylglyoxal and oxidative stress in cultured vascular smooth muscle cellsAdvanced glycated end-products affect HIF-transcriptional activity in renal cellsAdvanced glycation endproduct (AGE) accumulation and AGE receptor (RAGE) up-regulation contribute to the onset of diabetic cardiomyopathy.Advanced glycation end products in children with type 1 diabetes and early reduced diastolic heart function.Serum carboxymethyllysine, an advanced glycation end product, and age-related macular degeneration: the Age, Gene/Environment Susceptibility-Reykjavik StudyMethylglyoxal mediates adipocyte proliferation by increasing phosphorylation of Akt1Differential effects of glyoxalase 1 overexpression on diabetic atherosclerosis and renal dysfunction in streptozotocin-treated, apolipoprotein E-deficient mice.Advanced glycation end products and oxidative stress in type 2 diabetes mellitus.Chemical modification of proteins by lipids in diabetes.Posttranslationally modified proteins as mediators of sustained intestinal inflammation.Association between Advanced Glycation End Products and Impaired Fasting Glucose: Results from the SALIA StudyNatural history of age-related retinal lesions that precede AMD in mice fed high or low glycemic index diets.Methylglyoxal-derived hydroimidazolone residue of plasma protein can behave as a predictor of prediabetes in Spontaneously Diabetic Torii rats.Soluble RAGE and atherosclerosis in youth with type 1 diabetes: a 5-year follow-up studyExploring post-translational arginine modification using chemically synthesized methylglyoxal hydroimidazolones.Accumulation of methylglyoxal and d-lactate in Pb-induced nephrotoxicity in rats.Glycation products as markers and predictors of the progression of diabetic complications.Oral advanced glycation endproducts (AGEs) promote insulin resistance and diabetes by depleting the antioxidant defenses AGE receptor-1 and sirtuin 1.Continuous Glucose Monitoring Systems: A Review.Aldehydes and disturbance of carbohydrate metabolism: some consequences and possible approaches to its normalization.The clinical relevance of assessing advanced glycation endproducts accumulation in diabetes.Modification of collagen IV by glucose or methylglyoxal alters distinct mesangial cell functions.Protection against loss of innate defenses in adulthood by low advanced glycation end products (AGE) intake: role of the antiinflammatory AGE receptor-1.Reduced soluble receptor for advanced glycation end-products (sRAGE) scavenger capacity precedes pre-eclampsia in Type 1 diabetes.Oxidative stress and aging: is methylglyoxal the hidden enemy?Glycation in diabetic nephropathy.Diabetes-related adduct formation and retinopathy.Reactive oxygen species in diabetic nephropathy: friend or foe?Dicarbonyl stress and glyoxalases in ovarian function.Methylglyoxal in diabetes: link to treatment, glycaemic control and biomarkers of complications.Mass spectrometric determination of early and advanced glycation in biology.AGE-receptor-1 counteracts cellular oxidant stress induced by AGEs via negative regulation of p66shc-dependent FKHRL1 phosphorylation.Ethyl pyruvate prevents methyglyoxal-induced retinal vascular injury in rats.Effect of high glucose, Porphyromonas gingivalis lipopolysaccharide and advanced glycation end-products on production of interleukin-6/-8 by gingival fibroblasts.Plasma Proteins Modified by Advanced Glycation End Products (AGEs) Reveal Site-specific Susceptibilities to Glycemic Control in Patients with Type 2 Diabetes.Below the radar: advanced glycation end products that detour "around the side". Is HbA1c not an accurate enough predictor of long term progression and glycaemic control in diabetes?Resveratrol protects against methylglyoxal-induced apoptosis and disruption of embryonic development in mouse blastocysts.Methylglyoxal content in drinking coffee as a cytotoxic factor.The methylglyoxal-derived AGE tetrahydropyrimidine is increased in plasma of individuals with type 1 diabetes mellitus and in atherosclerotic lesions and is associated with sVCAM-1.
P2860
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P2860
Increased serum levels of the specific AGE-compound methylglyoxal-derived hydroimidazolone in patients with type 2 diabetes.
description
2003 nî lūn-bûn
@nan
2003年の論文
@ja
2003年学术文章
@wuu
2003年学术文章
@zh
2003年学术文章
@zh-cn
2003年学术文章
@zh-hans
2003年学术文章
@zh-my
2003年学术文章
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2003年學術文章
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2003年學術文章
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name
Increased serum levels of the ...... patients with type 2 diabetes.
@en
Increased serum levels of the ...... patients with type 2 diabetes.
@nl
type
label
Increased serum levels of the ...... patients with type 2 diabetes.
@en
Increased serum levels of the ...... patients with type 2 diabetes.
@nl
prefLabel
Increased serum levels of the ...... patients with type 2 diabetes.
@en
Increased serum levels of the ...... patients with type 2 diabetes.
@nl
P2093
P356
P1433
P1476
Increased serum levels of the ...... patients with type 2 diabetes
@en
P2093
Birkeland KI
Brownlee M
Giardino I
Hanssen KF
Kilhovd BK
Torjesen PA
P304
P356
10.1053/META.2003.50035
P577
2003-02-01T00:00:00Z