Protection against loss of innate defenses in adulthood by low advanced glycation end products (AGE) intake: role of the antiinflammatory AGE receptor-1. - Wikidata - wikimedia - TriplyDB

Protection against loss of innate defenses in adulthood by low advanced glycation end products (AGE) intake: role of the antiinflammatory AGE receptor-1.

Protection against loss of innate defenses in adulthood by low advanced glycation end products (AGE) intake: role of the antiinflammatory AGE receptor-1.

http://www.wikidata.org/entity/Q37417494

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Do advanced glycation end-products play a role in malaria susceptibility?Advanced glycation end products in foods and a practical guide to their reduction in the diet Dietary Advanced Glycation End Products and Risk Factors for Chronic Disease: A Systematic Review of Randomised Controlled Trials Current perspectives on the health risks associated with the consumption of advanced glycation end products: recommendations for dietary management Advanced glycation end-products: modifiable environmental factors profoundly mediate insulin resistance Dietary advanced glycation end products and their role in health and disease Role of advanced glycation end products in cellular signaling Oral glycotoxins are a modifiable cause of dementia and the metabolic syndrome in mice and humans Dietary interventions to contrast the onset and progression of diabetic nephropathy: A critical survey of new data.Effects of Two Different Dietary Patterns on Inflammatory Markers, Advanced Glycation End Products and Lipids in Subjects without Type 2 Diabetes: A Randomised Crossover Study.Dietary Metabolites and Chronic Kidney Disease.Lifestyle and Advanced Glycation End Products (AGEs) Burden: Its Relevance to Healthy Aging Consumption of diets with low advanced glycation end products improves cardiometabolic parameters: meta-analysis of randomised controlled trials.Effect of dietary advanced glycation end products on inflammation and cardiovascular risks in healthy overweight adults: a randomised crossover trial Diet and Diabetic Kidney Disease: Plant Versus Animal Protein.Maternally transmitted and food-derived glycotoxins: a factor preconditioning the young to diabetes?Dietary advanced glycation end products are associated with decline in memory in young elderly.Pre-pregnancy fried food consumption and the risk of gestational diabetes mellitus: a prospective cohort study Dietary fat quantity and quality modifies advanced glycation end products metabolism in patients with metabolic syndrome.Diabetic patients could do as well as non-diabetic patients without inflammation on peritoneal dialysis Restriction of advanced glycation end products improves insulin resistance in human type 2 diabetes: potential role of AGER1 and SIRT1 Advanced glycation end products and oxidative stress in type 2 diabetes mellitus.Effects of sevelamer carbonate on advanced glycation end products and antioxidant/pro-oxidant status in patients with diabetic kidney disease.Elevated serum advanced glycation endproducts in obese indicate risk for the metabolic syndrome: a link between healthy and unhealthy obesity?Dietary advanced glycation end products and aging.Natural history of age-related retinal lesions that precede AMD in mice fed high or low glycemic index diets.Effects of sevelamer on HbA1c, inflammation, and advanced glycation end products in diabetic kidney disease Relationship of Advanced Glycation End Products With Cardiovascular Disease in Menopausal Women Inhibition of Advanced Glycation End Products (AGEs) Accumulation by Pyridoxamine Modulates Glomerular and Mesangial Cell Estrogen Receptor α Expression in Aged Female Mice.The AGE-RAGE Axis and Its Relationship to Markers of Cardiovascular Disease in Newly Diagnosed Diabetic Patients.Oral advanced glycation endproducts (AGEs) promote insulin resistance and diabetes by depleting the antioxidant defenses AGE receptor-1 and sirtuin 1.Contribution of dietary advanced glycation end products (AGE) to circulating AGE: role of dietary fat Uremic Toxicity of Advanced Glycation End Products in CKD.Managing chronic inflammation in the aging diabetic patient with CKD by diet or sevelamer carbonate: a modern paradigm shift.Dietary Advanced Glycation End Products and Their Potential Role in Cardiometabolic Disease in Children.AGE restriction in diabetes mellitus: a paradigm shift.Consumption of deep-fried foods and risk of prostate cancer Identifying advanced glycation end products as a major source of oxidants in aging: implications for the management and/or prevention of reduced renal function in elderly persons Increased circulating advanced glycation endproducts (AGEs) in acute trauma patients.Suppression of native defense mechanisms, SIRT1 and PPARγ, by dietary glycoxidants precedes disease in adult humans; relevance to lifestyle-engendered chronic diseases.

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Protection against loss of innate defenses in adulthood by low advanced glycation end products (AGE) intake: role of the antiinflammatory AGE receptor-1.

http://www.wikidata.org/entity/Q37417494

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article científic

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article scientifique

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articolo scientifico

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bilimsel makale

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scientific article published on 09 October 2009

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vedecký článok

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vetenskaplig artikel

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vědecký článek

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Protection against loss of inn ...... tiinflammatory AGE receptor-1.

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Protection against loss of inn ...... dvanced glycation end products

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Protection against loss of inn ...... tiinflammatory AGE receptor-1.

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Protection against loss of inn ...... dvanced glycation end products

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Protection against loss of inn ...... tiinflammatory AGE receptor-1.

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Protection against loss of inn ...... dvanced glycation end products

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The Journal of Clinical Endocrinology and Metabolism

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Protection against loss of inn ...... tiinflammatory AGE receptor-1.

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Angie Yong

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Gary E Striker

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Helen Vlassara

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Jaime Uribarri

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Jeremy M Silverman

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Laurie Tansman

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Li Zhu

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Luigi Ferrucci

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Michal Beeri

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Renata Pyzik

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P2860

Advanced glycation endproduct (AGE) receptor 1 is a negative regulator of the inflammatory response to AGE in mesangial cells

Circulating glycotoxins and dietary advanced glycation endproducts: two links to inflammatory response, oxidative stress, and aging

Orally absorbed reactive glycation products (glycotoxins): an environmental risk factor in diabetic nephropathy

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Biochemistry and molecular cell biology of diabetic complications

Deficiency of cyclooxygenases transcripts in cultured primary bronchial epithelial cells of aspirin-sensitive asthmatics

Presence of diabetic complications in type 1 diabetic patients correlates with low expression of mononuclear cell AGE-receptor-1 and elevated serum AGE

The SH2 and SH3 domains of mammalian Grb2 couple the EGF receptor to the Ras activator mSos1

Inflammation in atherosclerosis

K/DOQI clinical practice guidelines for chronic kidney disease: evaluation, classification, and stratification

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4483-4491

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scholarly article

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10.1210/JC.2009-0089

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11

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94

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2009-10-09T00:00:00Z

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19820033

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