Increased contractility of diabetic rabbit corpora smooth muscle in response to endothelin is mediated via Rho-kinase beta.
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Activation of dopamine D4 receptors by ABT-724 induces penile erection in ratsThe role of phosphodiesterase-5 inhibitors in prostatic inflammation: a reviewInhibition of Rho-kinase improves erectile function, increases nitric oxide signaling and decreases penile apoptosis in a rat model of cavernous nerve injuryUp-regulation of the RhoA/Rho-kinase signaling pathway in corpus cavernosum from endothelial nitric-oxide synthase (NOS), but not neuronal NOS, null mice.Inactivation of phosphorylated endothelial nitric oxide synthase (Ser-1177) by O-GlcNAc in diabetes-associated erectile dysfunctionRhoA/Rho-kinase and vascular diseases: what is the link?RhoA/Rho-kinase suppresses endothelial nitric oxide synthase in the penis: a mechanism for diabetes-associated erectile dysfunctionArginase II deletion increases corpora cavernosa relaxation in diabetic mice.The future is today: emerging drugs for the treatment of erectile dysfunctionErectile dysfunction - an update of current practice and future strategies.Endothelial nitric oxide synthase keeps erection regulatory function balance in the penis.Rho kinase as potential therapeutic target for cardiovascular diseases: opportunities and challenges.Gene and stem cell therapy for erectile dysfunction.Lower urinary tract symptoms and sexual dysfunction: epidemiology and pathophysiology.Endothelial dysfunction in diabetic erectile dysfunction.The future of the oral pharmacotherapy of male erectile dysfunction: things to come.Molecular Yin and Yang of erectile function and dysfunction.Deletion of SM-B, the high ATPase isoform of myosin, upregulates the PKC-mediated signal transduction pathway in murine urinary bladder smooth muscle.Attenuated RhoA/Rho-kinase signaling in penis of transgenic sickle cell miceMarkers of erectile dysfunction.Activated Rho kinase mediates diabetes-induced elevation of vascular arginase activation and contributes to impaired corpora cavernosa relaxation: possible involvement of p38 MAPK activationLong-term diabetic complications may be ameliorated by targeting Rho kinase.The promise of inhibition of smooth muscle tone as a treatment for erectile dysfunction: where are we now?The use of PDE-5 inhibitors in the treatment of lower urinary tract symptoms due to benign prostatic hyperplasia.Models for erectile dysfunction and their importance to novel drug discovery.Dysregulation of cGMP-dependent protein kinase 1 (PKG-1) impairs erectile function in diabetic rats: influence of in vivo gene therapy of PKG1alpha.Elevated plasma aldosterone is an independent risk factor for erectile dysfunction in men.Sialorphin (the mature peptide product of Vcsa1) relaxes corporal smooth muscle tissue and increases erectile function in the ageing ratErectile properties of the Rho-kinase inhibitor SAR407899 in diabetic animals and human isolated corpora cavernosa.Endothelin-1 induces alveolar epithelial-mesenchymal transition through endothelin type A receptor-mediated production of TGF-beta1Pravastatin improves the impaired nitric oxide-mediated neurogenic and endothelium-dependent relaxation of corpus cavernosum in aged rats.Role of some vasoactive mediators in patients with erectile dysfunction: their relationship with angiotensin-converting enzyme and growth hormone.Downregulation of cGMP-dependent protein kinase-1 activity in the corpus cavernosum smooth muscle of diabetic rabbits.Gene transfer with a vector expressing Maxi-K from a smooth muscle-specific promoter restores erectile function in the aging rat.Chronic treatment with taurine ameliorates diabetes-induced dysfunction of nitric oxide-mediated neurogenic and endothelium-dependent corpus cavernosum relaxation in rats.Increased basal phosphorylation of detrusor smooth muscle myosin in alloxan-induced diabetic rabbit is mediated by upregulation of Rho-kinase beta and CPI-17.Comparison of sildenafil with strontium fructose diphosphate in improving erectile dysfunction against upregulated cavernosal NADPH oxidase, protein kinase Cε, and endothelin system in diabetic rats.The effect of mirabegron, used for overactive bladder treatment, on female sexual function: a prospective controlled study.
P2860
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P2860
Increased contractility of diabetic rabbit corpora smooth muscle in response to endothelin is mediated via Rho-kinase beta.
description
2003 nî lūn-bûn
@nan
2003年の論文
@ja
2003年学术文章
@wuu
2003年学术文章
@zh
2003年学术文章
@zh-cn
2003年学术文章
@zh-hans
2003年学术文章
@zh-my
2003年学术文章
@zh-sg
2003年學術文章
@yue
2003年學術文章
@zh-hant
name
Increased contractility of dia ...... mediated via Rho-kinase beta.
@en
Increased contractility of dia ...... mediated via Rho-kinase beta.
@nl
type
label
Increased contractility of dia ...... mediated via Rho-kinase beta.
@en
Increased contractility of dia ...... mediated via Rho-kinase beta.
@nl
prefLabel
Increased contractility of dia ...... mediated via Rho-kinase beta.
@en
Increased contractility of dia ...... mediated via Rho-kinase beta.
@nl
P2093
P2860
P356
P1476
Increased contractility of dia ...... mediated via Rho-kinase beta.
@en
P2093
P2860
P2888
P356
10.1038/SJ.IJIR.3900947
P577
2003-02-01T00:00:00Z