Identification of acquired copy number alterations and uniparental disomies in cytogenetically normal acute myeloid leukemia using high-resolution single-nucleotide polymorphism analysis.
about
The H/ACA RNP assembly factor SHQ1 functions as an RNA mimicUse of single nucleotide polymorphism array technology to improve the identification of chromosomal lesions in leukemiaAbnormal dosage of ultraconserved elements is highly disfavored in healthy cells but not cancer cellsThe origin and evolution of mutations in acute myeloid leukemiaGenomic and epigenomic landscapes of adult de novo acute myeloid leukemia.The complexity of interpreting genomic data in patients with acute myeloid leukemia.Clinical impact of gene mutations and lesions detected by SNP-array karyotyping in acute myeloid leukemia patients in the context of gemtuzumab ozogamicin treatment: results of the ALFA-0701 trial.A novel insertion ins(18;5)(q21.1;q31.2q35.1) in acute myeloid leukemia associated with microdeletions at 5q31.2, 5q35.1q35.2 and 18q12.3q21.1 detected by oligobased array comparative genomic hybridizationAcquired genomic copy number aberrations and survival in adult acute myelogenous leukemiaEvaluation of gene expression signatures predictive of cytogenetic and molecular subtypes of pediatric acute myeloid leukemiaPrognostic significance of DNA methyltransferase 3A mutations in cytogenetically normal acute myeloid leukemia: a study by the Acute Leukemia French Association.Clinical relevance of high-resolution single nucleotide polymorphism array in patients with relapsed acute lymphoblastic leukemia with normal karyotype: a report of three cases.Integrative analysis of prostate cancer aggressiveness.CDKN1B, encoding the cyclin-dependent kinase inhibitor 1B (p27), is located in the minimally deleted region of 12p abnormalities in myeloid malignancies and its low expression is a favorable prognostic marker in acute myeloid leukemia.Heritable GATA2 mutations associated with familial myelodysplastic syndrome and acute myeloid leukemia.Integrated genomic profiling, therapy response, and survival in adult acute myelogenous leukemiaQuantitative model of R-loop forming structures reveals a novel level of RNA-DNA interactome complexity.Older patients with normal karyotype acute myeloid leukemia have a higher rate of genomic changes compared to young patients as determined by SNP array analysismiR-10a is aberrantly overexpressed in Nucleophosmin1 mutated acute myeloid leukaemia and its suppression induces cell death.High-resolution genomic profiling of adult and pediatric core-binding factor acute myeloid leukemia reveals new recurrent genomic alterations.Double inv(3)(q21q26.2) in acute myeloid leukemia is resulted from an acquired copy neutral loss of heterozygosity of chromosome 3q and associated with disease progression.Integrating multiple molecular sources into a clinical risk prediction signature by extracting complementary information.Erroneous HLA typing as a result of acquired uniparental disomy in a patient with acute lymphoblastic leukaemia in peripheral blood complete remissionAre changes in HLA Ags responsible for leukemia relapse after HLA-matched allogeneic hematopoietic SCT?Clonal evolution and devolution after chemotherapy in adult acute myelogenous leukemia.Helicase-like transcription factor is a RUNX1 target whose downregulation promotes genomic instability and correlates with complex cytogenetic features in acute myeloid leukemia.Clinical implications of genome-wide DNA methylation studies in acute myeloid leukemia.DNA methylation profiling in acute myeloid leukemia: from recent technological advances to biological and clinical insights.Older patients with acute myeloid leukemia.An integrated genomic approach to the assessment and treatment of acute myeloid leukemia.Gene expression profiling in MDS and AML: potential and future avenues.The consequences of uniparental disomy and copy number neutral loss-of-heterozygosity during human development and cancer.Detection of copy number alterations in acute myeloid leukemia and myelodysplastic syndromes.Molecular-based classification of acute myeloid leukemia and its role in directing rational therapy: personalized medicine for profoundly promiscuous proliferations.Regulation of snoRNAs in cancer: close encounters with interferon.Recent discoveries in molecular characterization of acute myeloid leukemia.Different characteristics identified by single nucleotide polymorphism array analysis in leukemia suggest the need for different application strategies depending on disease category.Acquired uniparental disomy of chromosome 9p in hematologic malignancies.Molecular and genetic alterations associated with therapy resistance and relapse of acute myeloid leukemia.Aberrant hnRNP K expression: All roads lead to cancer
P2860
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P2860
Identification of acquired copy number alterations and uniparental disomies in cytogenetically normal acute myeloid leukemia using high-resolution single-nucleotide polymorphism analysis.
description
2009 nî lūn-bûn
@nan
2009年の論文
@ja
2009年学术文章
@wuu
2009年学术文章
@zh-cn
2009年学术文章
@zh-hans
2009年学术文章
@zh-my
2009年学术文章
@zh-sg
2009年學術文章
@yue
2009年學術文章
@zh
2009年學術文章
@zh-hant
name
Identification of acquired cop ...... leotide polymorphism analysis.
@en
Identification of acquired cop ...... leotide polymorphism analysis.
@nl
type
label
Identification of acquired cop ...... leotide polymorphism analysis.
@en
Identification of acquired cop ...... leotide polymorphism analysis.
@nl
prefLabel
Identification of acquired cop ...... leotide polymorphism analysis.
@en
Identification of acquired cop ...... leotide polymorphism analysis.
@nl
P2093
P2860
P356
P1433
P1476
Identification of acquired cop ...... leotide polymorphism analysis.
@en
P2093
J R Downing
K Holzmann
K Urlbauer
P2860
P2888
P304
P356
10.1038/LEU.2009.263
P577
2009-12-17T00:00:00Z