Lysophosphatidylcholines prime the NADPH oxidase and stimulate multiple neutrophil functions through changes in cytosolic calcium.
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Stop the flow: a paradigm for cell signaling mediated by reactive oxygen species in the pulmonary endotheliumNeisseria gonorrhoeae phagosomes delay fusion with primary granules to enhance bacterial survival inside human neutrophils.Reduced use of allogeneic platelets through high-yield perioperative autologous plateletpheresis and reinfusion.Tumor necrosis factor-alpha causes release of cytosolic interleukin-18 from human neutrophils.Peroxiredoxin 6 phosphorylation and subsequent phospholipase A2 activity are required for agonist-mediated activation of NADPH oxidase in mouse pulmonary microvascular endothelium and alveolar macrophages.Platelet-activating factor-mediated endosome formation causes membrane translocation of p67phox and p40phox that requires recruitment and activation of p38 MAPK, Rab5a, and phosphatidylinositol 3-kinase in human neutrophils.A standardized blood test for the routine clinical diagnosis of impaired GM-CSF signaling using flow cytometry.Lysophosphatidylcholines activate G2A inducing G(αi)₋₁-/G(αq/)₁₁- Ca²(+) flux, G(βγ)-Hck activation and clathrin/β-arrestin-1/GRK6 recruitment in PMNs.Transfusion-related acute lung injury (TRALI): current concepts and misconceptions.Identification of lipids that accumulate during the routine storage of prestorage leukoreduced red blood cells and cause acute lung injury.Cationic additives in nanosystems activate cytotoxicity and inflammatory response of human neutrophils: lipid nanoparticles versus polymeric nanoparticles.Hypertonic saline inhibits arachidonic acid priming of the human neutrophil oxidase.Soluble CD40 ligand accumulates in stored blood components, primes neutrophils through CD40, and is a potential cofactor in the development of transfusion-related acute lung injuryFresh red blood cell transfusion and short-term pulmonary, immunologic, and coagulation status: a randomized clinical trial.Regulation of superoxide production in neutrophils: role of calcium influx.TRALI--definition, mechanisms, incidence and clinical relevance.Amantadine inhibits platelet-activating factor induced clathrin-mediated endocytosis in human neutrophils.Therapeutic options for transfusion related acute lung injury; the potential of the G2A receptor.Phox activity of differentiated PLB-985 cells is enhanced, in an agonist specific manner, by the PLA2 activity of Prdx6-PLA2.Oral administration of 2-docosahexaenoyl lysophosphatidylcholine displayed anti-inflammatory effects on zymosan A-induced peritonitis.Platelet-derived growth factor BB stimulates vasculogenesis of embryonic stem cell-derived endothelial cells by calcium-mediated generation of reactive oxygen species.LysoPCs induce Hck- and PKCδ-mediated activation of PKCγ causing p47phox phosphorylation and membrane translocation in neutrophils.Properties and functions of lactosylceramide from mouse neutrophils.Bioactive lysophosphatidylcholine 16:0 and 18:0 are elevated in lungs of asthmatic subjects.Stimulated bronchial epithelial cells release bioactive lysophosphatidylcholine 16:0, 18:0, and 18:1.The phospholipase A2 activity of peroxiredoxin 6 modulates NADPH oxidase 2 activation via lysophosphatidic acid receptor signaling in the pulmonary endothelium and alveolar macrophages.Regulation of eosinophil adhesion by lysophosphatidylcholine via a non-store-operated Ca2+ channelMirasol Pathogen Reduction Technology treatment does not affect acute lung injury in a two-event in vivo model caused by stored blood components.Red blood cells, transfusion-related acute lung injury, and lipids: a role for liporeduction?Pentoxifylline attenuates leukoreduced stored blood-induced neutrophil activation through inhibition of mitogen-activated protein kinases.Integration of TRPC6 and NADPH oxidase activation in lysophosphatidylcholine-induced TRPC5 externalization.Blood lysophosphatidylcholine (LPC) levels and characteristic molecular species in neonates: prolonged low blood LPC levels in very low birth weight infants.Antibodies to major histocompatibility complex class II antigens directly prime neutrophils and cause acute lung injury in a two-event in vivo rat model.Neutrophil function in children following allogeneic hematopoietic stem cell transplant.Generation of neutrophil priming activity by cell-containing blood components treated with pathogen reduction technology and stored in platelet additive solutions.Comparative analysis of the pathological events involved in immune and non-immune TRALI models.
P2860
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P2860
Lysophosphatidylcholines prime the NADPH oxidase and stimulate multiple neutrophil functions through changes in cytosolic calcium.
description
2003 nî lūn-bûn
@nan
2003年の論文
@ja
2003年学术文章
@wuu
2003年学术文章
@zh
2003年学术文章
@zh-cn
2003年学术文章
@zh-hans
2003年学术文章
@zh-my
2003年学术文章
@zh-sg
2003年學術文章
@yue
2003年學術文章
@zh-hant
name
Lysophosphatidylcholines prime ...... changes in cytosolic calcium.
@en
Lysophosphatidylcholines prime ...... changes in cytosolic calcium.
@nl
type
label
Lysophosphatidylcholines prime ...... changes in cytosolic calcium.
@en
Lysophosphatidylcholines prime ...... changes in cytosolic calcium.
@nl
prefLabel
Lysophosphatidylcholines prime ...... changes in cytosolic calcium.
@en
Lysophosphatidylcholines prime ...... changes in cytosolic calcium.
@nl
P2093
P2860
P356
P1476
Lysophosphatidylcholines prime ...... changes in cytosolic calcium.
@en
P2093
A Jason Bjornsen
Andrew J Paterson
Anirban Bannerjee
Carlton C Barnett
Christine Hamiel
Christopher C Silliman
Daniel R Ambruso
Junichi Aiboshi
Kelly M England
Marguerite Kelher
P2860
P304
P356
10.1189/JLB.0402179
P50
P577
2003-04-01T00:00:00Z