A defect in hematopoietic stem cell migration explains the nonrandom X-chromosome inactivation in carriers of Wiskott-Aldrich syndrome.
about
Rho GTPases in hematopoiesis and hemopathiesActin cytoskeletal defects in immunodeficiencyWiskott-Aldrich syndrome protein and the cytoskeletal dynamics of dendritic cellsWASP deficiency leads to global defects of directed leukocyte migration in vitro and in vivo.Combined de-novo mutation and non-random X-chromosome inactivation causing Wiskott-Aldrich syndrome in a female with thrombocytopenia.Absence of WASp Enhances Hematopoietic and Megakaryocytic Differentiation in a Human Embryonic Stem Cell Model.Wiskott-Aldrich syndrome protein is required for homeostasis and function of invariant NKT cells.Surfing the big WAVE: Insights into the role of WAVE3 as a driving force in cancer progression and metastasisWiskott-Aldrich syndrome protein is required for regulatory T cell homeostasis.Genetic deletion of Cdc42GAP reveals a role of Cdc42 in erythropoiesis and hematopoietic stem/progenitor cell survival, adhesion, and engraftment.Wiskott Aldrich syndrome protein (WASP) and N-WASP are critical for T cell development.Survival of the fittest: in vivo selection and stem cell gene therapyWASP confers selective advantage for specific hematopoietic cell populations and serves a unique role in marginal zone B-cell homeostasis and function.Wiskott-Aldrich syndrome protein deficiency in B cells results in impaired peripheral homeostasis.Wiskott-Aldrich syndrome protein-deficient hematopoietic cells can be efficiently mobilized by granulocyte colony-stimulating factor.Leukocyte trafficking in primary immunodeficiencies.Wiskott-Aldrich syndrome protein is an effector of Kit signaling.Rho GTPases in hematopoietic stem cell functionsDefects of leukocyte migration in primary immunodeficiencies.Safe and Effective Gene Therapy for Murine Wiskott-Aldrich Syndrome Using an Insulated Lentiviral VectorGene Therapy with Hematopoietic Stem Cells: The Diseased Bone Marrow's Point of View.Quantitative label-free single cell tracking in 3D biomimetic matrices.Current understanding of the Wiskott-Aldrich syndrome and prospects for gene therapy.T cell responses in mammalian diaphanous-related formin mDia1 knock-out mice.The Wave2 scaffold Hem-1 is required for transition of fetal liver hematopoiesis to bone marrow.Development of lentiviral gene therapy for Wiskott Aldrich syndrome
P2860
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P2860
A defect in hematopoietic stem cell migration explains the nonrandom X-chromosome inactivation in carriers of Wiskott-Aldrich syndrome.
description
2003 nî lūn-bûn
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2003年の論文
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2003年学术文章
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name
A defect in hematopoietic stem ...... s of Wiskott-Aldrich syndrome.
@en
A defect in hematopoietic stem ...... s of Wiskott-Aldrich syndrome.
@nl
type
label
A defect in hematopoietic stem ...... s of Wiskott-Aldrich syndrome.
@en
A defect in hematopoietic stem ...... s of Wiskott-Aldrich syndrome.
@nl
prefLabel
A defect in hematopoietic stem ...... s of Wiskott-Aldrich syndrome.
@en
A defect in hematopoietic stem ...... s of Wiskott-Aldrich syndrome.
@nl
P2093
P50
P1433
P1476
A defect in hematopoietic stem ...... s of Wiskott-Aldrich syndrome.
@en
P2093
Catherine Lacout
Claude Capron
Dominique Duménil
Elie Haddad
Fedor Svinarchouk
Loic Garçon
Scott B Snapper
William Vainchenker
P304
P356
10.1182/BLOOD-2002-07-2099
P407
P577
2003-05-01T00:00:00Z