Rapid suppression of mitochondrial permeability transition by methylglyoxal. Role of reversible arginine modification.
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GLO1 overexpression in human malignant melanomaModification of permeability transition pore arginine(s) by phenylglyoxal derivatives in isolated mitochondria and mammalian cells. Structure-function relationship of arginine ligands.Yeast protein glycation in vivo by methylglyoxal. Molecular modification of glycolytic enzymes and heat shock proteins.Destabilization of the outer and inner mitochondrial membranes by core and linker histones.Glyoxalase I polymorphism rs2736654 causing the Ala111Glu substitution modulates enzyme activity--implications for autism.Arabidopsis thaliana Contains Both Ni2+ and Zn2+ Dependent Glyoxalase I Enzymes and Ectopic Expression of the Latter Contributes More towards Abiotic Stress Tolerance in E. coli.Comparative proteomic analysis provides novel insight into the interaction between resistant vs susceptible tomato cultivars and TYLCV infection.A brief critical overview of the biological effects of methylglyoxal and further evaluation of a methylglyoxal-based anticancer formulation in treating cancer patients.Neurotoxic lipid peroxidation species formed by ischemic stroke increase injury.A short review on creatine-creatine kinase system in relation to cancer and some experimental results on creatine as adjuvant in cancer therapy.The role of methylglyoxal and the glyoxalase system in diabetes and other age-related diseases.Preserving Brain Function in Aging: The Anti-glycative Potential of Berry Fruit.Transgenic expression and activation of PGC-1α protect dopaminergic neurons in the MPTP mouse model of Parkinson's disease.Dicarbonyls linked to damage in the powerhouse: glycation of mitochondrial proteins and oxidative stress.Peroxisome proliferator-activated receptor-γ coactivator-1α mediates neuroprotection against excitotoxic brain injury in transgenic mice: role of mitochondria and X-linked inhibitor of apoptosis protein.Sugar beet M14 glyoxalase I gene can enhance plant tolerance to abiotic stresses.Glycation damage targets glutamate dehydrogenase in the rat liver mitochondrial matrix during aging.Proteomic studies identified a single nucleotide polymorphism in glyoxalase I as autism susceptibility factor.GLYI and D-LDH play key role in methylglyoxal detoxification and abiotic stress tolerance.Arginine 107 of yeast ATP synthase subunit g mediates sensitivity of the mitochondrial permeability transition to phenylglyoxal
P2860
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P2860
Rapid suppression of mitochondrial permeability transition by methylglyoxal. Role of reversible arginine modification.
description
2003 nî lūn-bûn
@nan
2003年の論文
@ja
2003年学术文章
@wuu
2003年学术文章
@zh-cn
2003年学术文章
@zh-hans
2003年学术文章
@zh-my
2003年学术文章
@zh-sg
2003年學術文章
@yue
2003年學術文章
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2003年學術文章
@zh-hant
name
Rapid suppression of mitochond ...... ersible arginine modification.
@en
Rapid suppression of mitochond ...... ersible arginine modification.
@nl
type
label
Rapid suppression of mitochond ...... ersible arginine modification.
@en
Rapid suppression of mitochond ...... ersible arginine modification.
@nl
prefLabel
Rapid suppression of mitochond ...... ersible arginine modification.
@en
Rapid suppression of mitochond ...... ersible arginine modification.
@nl
P2093
P2860
P356
P1476
Rapid suppression of mitochond ...... versible arginine modification
@en
P2093
Linn Hensbo
Marina Franck
Matts D Linder
Oliver Speer
Ove Eriksson
Paavo K J Kinnunen
Sarune Morkunaite-Haimi
P2860
P304
34757-34763
P356
10.1074/JBC.M301990200
P407
P577
2003-06-18T00:00:00Z