IGF-1 inhibits the mitochondrial apoptosis program in mesangial cells exposed to high glucose. - Wikidata - wikimedia - TriplyDB

IGF-1 inhibits the mitochondrial apoptosis program in mesangial cells exposed to high glucose.

IGF-1 inhibits the mitochondrial apoptosis program in mesangial cells exposed to high glucose.

http://www.wikidata.org/entity/Q44522383

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A novel pro-apoptotic function of RACK1: suppression of Src activity in the intrinsic and Akt pathways A novel mitochondrial matrix serine/threonine protein phosphatase regulates the mitochondria permeability transition pore and is essential for cellular survival and development Role of Receptor Tyrosine Kinase Signaling in Renal Fibrosis Human conditions of insulin-like growth factor-I (IGF-I) deficiency Insulin-like growth factor binding protein-3 mediates cytokine-induced mesangial cell apoptosis Enhanced expression of naofen in kidney of streptozotocin-induced diabetic rats: possible correlation to apoptosis of tubular epithelial cells.Crosstalk between mitogen-activated protein kinases and mitochondria in cardiac diseases: therapeutic perspectives Human ghrelin protects animals from renal ischemia-reperfusion injury through the vagus nerve.Bifidobacterial recombinant thymidine kinase-ganciclovir gene therapy system induces FasL and TNFR2 mediated antitumor apoptosis in solid tumors miR-29a promotes myocardial cell apoptosis induced by high glucose through down-regulating IGF-1 Null mutations at the p66 and bradykinin 2 receptor loci induce divergent phenotypes in the diabetic kidney.A proteomic screen identified stress-induced chaperone proteins as targets of Akt phosphorylation in mesangial cells Hepatitis B virus X protein modulates apoptosis in human renal proximal tubular epithelial cells by activating the JAK2/STAT3 signaling pathway.Functional characterization of a mitochondrial Ser/Thr protein phosphatase in cell death regulation Inhibition of p66ShcA redox activity in cardiac muscle cells attenuates hyperglycemia-induced oxidative stress and apoptosis.IGF-IR in neuroprotection and brain tumors.The role of reactive oxygen species in apoptosis of the diabetic kidney.MnSOD in oxidative stress response-potential regulation via mitochondrial protein influx.IGF-1 potentiates sensory innervation signalling by modulating the mitochondrial fission/fusion balance.Diabetic nephropathy: lessons from the mouse.Angiotensin II- and glucose-stimulated extracellular matrix production: mediation by the insulin-like growth factor (IGF) axis in a murine mesangial cell line.IGF-1 increases laminin, cyclin D1, and p21Cip1 expression in glomerular mesangial cells: an investigation of the intracellular signaling pathway and cell-cycle progression.Lithium activates the Wnt and phosphatidylinositol 3-kinase Akt signaling pathways to promote cell survival in the absence of soluble survival factors.Effect of recombinant Hepatitis B virus on human glomerular mesangial cell apoptosis Insulin-like growth factor-I-forkhead box O transcription factor 3a counteracts high glucose/tumor necrosis factor-α-mediated neuronal damage: implications for human immunodeficiency virus encephalitis.A distinctive physiological role for IkappaBbeta in the propagation of mitochondrial respiratory stress signaling.After Myocardial Ischemia-Reperfusion, miR-29a, and Let7 Could Affect Apoptosis through Regulating IGF-1.Regulation of high glucose-induced apoptosis of brain pericytes by mitochondrial CA VA: A specific target for prevention of diabetic cerebrovascular pathology.PKC-{epsilon}-dependent survival signals in diabetic hearts.Activated IGF-1R inhibits hyperglycemia-induced DNA damage and promotes DNA repair by homologous recombination.Neurotrophic and Neuroregenerative Effects of GH/IGF1.Unexpected Neuroprotective Effects of Loganin on 1-Methyl-4-Phenyl-1,2,3,6-Tetrahydropyridine-Induced Neurotoxicity and Cell Death in Zebrafish.Expression of immunoglobulin A in human mesangial cells and its effects on cell apoptosis and adhesion.IGF-1 protects SH-SY5Y cells against MPP+-induced apoptosis via PI3K/PDK-1/Akt pathway.The effect of recombinant human growth hormone and insulin-like growth factor-1 on the mitochondrial function and viability of peripheral blood mononuclear cells in vitro.

P2860

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P2860

IGF-1 inhibits the mitochondrial apoptosis program in mesangial cells exposed to high glucose.

http://www.wikidata.org/entity/Q44522383

description

2003 nî lūn-bûn

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2003年の論文

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2003年学术文章

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2003年学术文章

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2003年学术文章

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2003年学术文章

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2003年学术文章

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2003年学术文章

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2003年學術文章

@yue

2003年學術文章

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name

IGF-1 inhibits the mitochondri ...... cells exposed to high glucose.

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IGF-1 inhibits the mitochondri ...... cells exposed to high glucose.

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type

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IGF-1 inhibits the mitochondri ...... cells exposed to high glucose.

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IGF-1 inhibits the mitochondri ...... cells exposed to high glucose.

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IGF-1 inhibits the mitochondri ...... cells exposed to high glucose.

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AJPRENAL.00209.2003

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American Journal of Physiology - Renal Physiology

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IGF-1 inhibits the mitochondri ...... cells exposed to high glucose.

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Andrew Baddoo

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Arunas Urbonas

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Ashwani Malhotra

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Barinder P S Kang

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Leonard G Meggs

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Stuart Baskin

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P2860

Mitochondrial ATP-sensitive potassium channels inhibit apoptosis induced by oxidative stress in cardiac cells

Mitochondrial PKCepsilon and MAPK form signaling modules in the murine heart: enhanced mitochondrial PKCepsilon-MAPK interactions and differential MAPK activation in PKCepsilon-induced cardioprotection

Bax suppresses tumorigenesis and stimulates apoptosis in vivo

Overexpression of insulin-like growth factor-1 attenuates the myocyte renin-angiotensin system in transgenic mice

Mitochondria and apoptosis

Regulation of neuronal survival by the serine-threonine protein kinase Akt

Normalizing mitochondrial superoxide production blocks three pathways of hyperglycaemic damage

Growth factor-mediated phosphorylation of proapoptotic BAD reduces tubule cell death in vitro and in vivo.

Antiapoptotic signalling by the insulin-like growth factor I receptor, phosphatidylinositol 3-kinase, and Akt.

Akt-dependent and -independent survival signaling pathways utilized by insulin-like growth factor I.

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10.1152/AJPRENAL.00209.2003

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