Insulin-like growth factor-1 prevents age-related decrease in specific force and intracellular Ca2+ in single intact muscle fibres from transgenic mice.
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The effects of testosterone and insulin-like growth factor 1 on motor system form and functionCalsequestrin (CASQ1) rescues function and structure of calcium release units in skeletal muscles of CASQ1-null miceIn situ detection and measurement of intracellular reactive oxygen species in single isolated mature skeletal muscle fibers by real time fluorescence microscopy.Skeletal muscle abnormalities and exercise intolerance in older patients with heart failure and preserved ejection fraction.Myosin binding protein-C slow: an intricate subfamily of proteins.Mechanical and electrophysiological properties of the sarcolemma of muscle fibers in two murine models of muscle dystrophy: col6a1-/- and mdxA polymorphism near IGF1 is associated with body composition and muscle function in women from the Health, Aging, and Body Composition Study.Quantitative measurement of Ca²(+) in the sarcoplasmic reticulum lumen of mammalian skeletal muscle.The I4895T mutation in the type 1 ryanodine receptor induces fiber-type specific alterations in skeletal muscle that mimic premature aging.Massive alterations of sarcoplasmic reticulum free calcium in skeletal muscle fibers lacking calsequestrin revealed by a genetically encoded probe.Increased extraocular muscle strength with direct injection of insulin-like growth factor-I.Paracrine effects of IGF-1 overexpression on the functional decline due to skeletal muscle disuse: molecular and functional evaluation in hindlimb unloaded MLC/mIgf-1 transgenic miceRole of Ca2+, membrane excitability, and Ca2+ stores in failing muscle contraction with aging.Sphingomyelinase promotes oxidant production and skeletal muscle contractile dysfunction through activation of NADPH oxidase.Effect of temperature on crossbridge force changes during fatigue and recovery in intact mouse muscle fibers.Age affects the contraction-induced mitochondrial redox response in skeletal muscle.D4cpv-calsequestrin: a sensitive ratiometric biosensor accurately targeted to the calcium store of skeletal muscle.Myofibrillar myosin ATPase activity in hindlimb muscles from young and aged ratsOxidative stress, mitochondrial damage, and cores in muscle from calsequestrin-1 knockout miceEndogenously determined restriction of food intake overcomes excitation-contraction uncoupling in JP45KO mice with aging.Diverse roles of growth hormone and insulin-like growth factor-1 in mammalian aging: progress and controversies.Motor neuron targeting of IGF-1 attenuates age-related external Ca2+-dependent skeletal muscle contraction in senescent mice.Parvalbumin gene transfer impairs skeletal muscle contractility in old miceCUG-BP1 regulates RyR1 ASI alternative splicing in skeletal muscle atrophy.Sarcoplasmic reticulum Ca2+ release declines in muscle fibers from aging mice.Sod1 gene ablation in adult mice leads to physiological changes at the neuromuscular junction similar to changes that occur in old wild-type mice.Therapeutic exercise attenuates neutrophilic lung injury and skeletal muscle wasting.Calpain inhibition rescues troponin T3 fragmentation, increases Cav1.1, and enhances skeletal muscle force in aging sedentary miceRelationship of physical function to vastus lateralis capillary density and metabolic enzyme activity in elderly men and women.Ryanodine receptor oxidation causes intracellular calcium leak and muscle weakness in aging.Altered Ca2+ sparks in aging skeletal and cardiac muscle.Low-affinity Ca2+ indicators compared in measurements of skeletal muscle Ca2+ transients.Minor sarcoplasmic reticulum membrane components that modulate excitation-contraction coupling in striated muscles.Development of a high-throughput method for real-time assessment of cellular metabolism in intact long skeletal muscle fibre bundles.TLR4 as receptor for HMGB1 induced muscle dysfunction in myositis.Mitochondrial redox state and Ca2+ sparks in permeabilized mammalian skeletal muscle.High temperature does not alter fatigability in intact mouse skeletal muscle fibres.Is functional hypertrophy and specific force coupled with the addition of myonuclei at the single muscle fiber level?Length-dependent Ca2+ activation in skeletal muscle fibers from mammaliansTop-Down Targeted Proteomics Reveals Decrease in Myosin Regulatory Light-Chain Phosphorylation That Contributes to Sarcopenic Muscle Dysfunction.
P2860
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P2860
Insulin-like growth factor-1 prevents age-related decrease in specific force and intracellular Ca2+ in single intact muscle fibres from transgenic mice.
description
2003 nî lūn-bûn
@nan
2003年の論文
@ja
2003年学术文章
@wuu
2003年学术文章
@zh
2003年学术文章
@zh-cn
2003年学术文章
@zh-hans
2003年学术文章
@zh-my
2003年学术文章
@zh-sg
2003年學術文章
@yue
2003年學術文章
@zh-hant
name
Insulin-like growth factor-1 p ...... e fibres from transgenic mice.
@en
Insulin-like growth factor-1 p ...... e fibres from transgenic mice.
@nl
type
label
Insulin-like growth factor-1 p ...... e fibres from transgenic mice.
@en
Insulin-like growth factor-1 p ...... e fibres from transgenic mice.
@nl
prefLabel
Insulin-like growth factor-1 p ...... e fibres from transgenic mice.
@en
Insulin-like growth factor-1 p ...... e fibres from transgenic mice.
@nl
P2093
P2860
P1476
Insulin-like growth factor-1 p ...... e fibres from transgenic mice.
@en
P2093
Estela Gonzalez
María Laura Messi
Osvaldo Delbono
Zhenlin Zheng
P2860
P304
P356
10.1113/JPHYSIOL.2003.048165
P407
P577
2003-08-22T00:00:00Z