Increased iNOS activity is essential for hepatic epithelial tight junction dysfunction in endotoxemic mice.
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Changes in hepatic cell junctions structure during experimental necrotizing enterocolitis: effect of EGF treatment.Regulation of intestinal barrier function by signal transducer and activator of transcription 5b.Protective effect of resin adsorption on septic plasma-induced tubular injury.Liver alkaline phosphatase: a missing link between choleresis and biliary inflammation.Liver-specific β-catenin knockout mice have bile canalicular abnormalities, bile secretory defect, and intrahepatic cholestasis.Mechanisms of disease: mechanisms and clinical implications of cholestasis in sepsis.Inflammatory levels of nitric oxide inhibit airway epithelial cell migration by inhibition of the kinase ERK1/2 and activation of hypoxia-inducible factor-1 alpha.Cyclic stretch-induced oxidative stress increases pulmonary alveolar epithelial permeability.Advances in sepsis-associated liver dysfunctionNitric oxide and cyclic nucleotides: their roles in junction dynamics and spermatogenesis.Metabolomics in pneumonia and sepsis: an analysis of the GenIMS cohort study.The molecular pathogenesis of cholestasis in sepsisLiver - guardian, modifier and target of sepsis.Protective effect and mechanisms of radix astragali injection on the intestinal mucosa of rats with obstructive jaundice.Vps33b is crucial for structural and functional hepatocyte polarity.Traumatic brain injury and intestinal dysfunction: uncovering the neuro-enteric axisNitric oxide and airway epithelial barrier function: regulation of tight junction proteins and epithelial permeability.Role of TNF-alpha in ileum tight junction alteration in mouse model of restraint stress.Role of peroxisome proliferator-activated receptor-alpha in ileum tight junction alteration in mouse model of restraint stress.Bile high-mobility group box 1 contributes to gut barrier dysfunction in experimental endotoxemia.Adenovirus-mediated human aquaporin-1 expression in hepatocytes improves lipopolysaccharide-induced cholestasis.Tight junction proteins in gallbladder epithelium: different expression in acute acalculous and calculous cholecystitis.Mild endotoxemia, NF-kappaB translocation, and cytokine increase during exertional heat stress in trained and untrained individuals.Prostasin attenuates inducible nitric oxide synthase expression in lipopolysaccharide-induced urinary bladder inflammation.
P2860
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P2860
Increased iNOS activity is essential for hepatic epithelial tight junction dysfunction in endotoxemic mice.
description
2003 nî lūn-bûn
@nan
2003年の論文
@ja
2003年学术文章
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2003年学术文章
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2003年学术文章
@zh-cn
2003年学术文章
@zh-hans
2003年学术文章
@zh-my
2003年学术文章
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2003年學術文章
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2003年學術文章
@zh-hant
name
Increased iNOS activity is ess ...... sfunction in endotoxemic mice.
@en
Increased iNOS activity is ess ...... sfunction in endotoxemic mice.
@nl
type
label
Increased iNOS activity is ess ...... sfunction in endotoxemic mice.
@en
Increased iNOS activity is ess ...... sfunction in endotoxemic mice.
@nl
prefLabel
Increased iNOS activity is ess ...... sfunction in endotoxemic mice.
@en
Increased iNOS activity is ess ...... sfunction in endotoxemic mice.
@nl
P2093
P2860
P356
P1476
Increased iNOS activity is ess ...... sfunction in endotoxemic mice.
@en
P2093
Mitchell P Fink
Runkuan Yang
Russell L Delude
Takashi Uchiyama
Xiaonan Han
P2860
P304
P356
10.1152/AJPGI.00231.2003
P577
2003-08-28T00:00:00Z