Evidence for radiosensitizing by gliotoxin in HL-60 cells: implications for a role of NF-kappaB independent mechanisms.

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Deletion of the gliP gene of Aspergillus fumigatus results in loss of gliotoxin production but has no effect on virulence of the fungus in a low-dose mouse infection model.BPIFB1 (LPLUNC1) inhibits radioresistance in nasopharyngeal carcinoma by inhibiting VTN expression.

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Evidence for radiosensitizing by gliotoxin in HL-60 cells: implications for a role of NF-kappaB independent mechanisms.

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2003 nî lūn-bûn

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2003年の論文

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2003年学术文章

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2003年学术文章

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2003年学术文章

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2003年学术文章

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2003年学术文章

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2003年學術文章

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2003年學術文章

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Evidence for radiosensitizing ...... kappaB independent mechanisms.

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Evidence for radiosensitizing ...... kappaB independent mechanisms.

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Evidence for radiosensitizing ...... kappaB independent mechanisms.

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Evidence for radiosensitizing ...... kappaB independent mechanisms.

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Evidence for radiosensitizing ...... kappaB independent mechanisms.

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Evidence for radiosensitizing ...... kappaB independent mechanisms.

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P1476

Evidence for radiosensitizing ...... kappaB independent mechanisms.

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Baust H

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Brey A

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Gern U

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Los M

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Röttinger EM

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Schmid RM

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Schoke A

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Seufferlein T

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Inhibition of JNK by cellular stress- and tumor necrosis factor alpha-induced AKT2 through activation of the NF kappa B pathway in human epithelial Cells

The immunosuppressive fungal metabolite gliotoxin specifically inhibits transcription factor NF-kappaB.

p53 mutations increase resistance to ionizing radiation.

Inhibition of nuclear translocation of transcription factor NF-kappa B by a synthetic peptide containing a cell membrane-permeable motif and nuclear localization sequence.

Major deletions in the gene encoding the p53 tumor antigen cause lack of p53 expression in HL-60 cells.

Bcl-2 undergoes phosphorylation by c-Jun N-terminal kinase/stress-activated protein kinases in the presence of the constitutively active GTP-binding protein Rac1.

Polyamine regulation of plasma membrane phospholipid flip-flop during apoptosis.

Activation of the c-Jun N-terminal kinase/stress-activated protein kinase pathway by overexpression of caspase-8 and its homologs.

Inhibition of nuclear factor-kappaB activity is involved in E1A-mediated sensitization of radiation-induced apoptosis.

Persistent activation of c-Jun N-terminal kinase 1 (JNK1) in gamma radiation-induced apoptosis.

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sj.onc.1206969

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8786-8796

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10.1038/SJ.ONC.1206969

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English

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P921

NF-κB

Evidence for radiosensitizing by gliotoxin in HL-60 cells: implications for a role of NF-kappaB independent mechanisms.

about

P2860

P2860

Evidence for radiosensitizing by gliotoxin in HL-60 cells: implications for a role of NF-kappaB independent mechanisms.

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