Anti-inflammatory therapy ameliorates leukocyte adhesion and microvascular flow abnormalities in transgenic sickle mice. - Wikidata - wikimedia - TriplyDB

Anti-inflammatory therapy ameliorates leukocyte adhesion and microvascular flow abnormalities in transgenic sickle mice.

Anti-inflammatory therapy ameliorates leukocyte adhesion and microvascular flow abnormalities in transgenic sickle mice.

http://www.wikidata.org/entity/Q44786816

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Oxidative stress in β-thalassaemia and sickle cell disease Hydroxyurea therapy of a murine model of sickle cell anemia inhibits the progression of pneumococcal disease by down-modulating E-selectin Local oxidative and nitrosative stress increases in the microcirculation during leukocytes-endothelial cell interactions.Heme degradation and vascular injury.Nuclear factor-kappa B (NFkappaB) component p50 in blood mononuclear cells regulates endothelial tissue factor expression in sickle transgenic mice: implications for the coagulopathy of sickle cell disease.Global gene expression profiling of endothelium exposed to heme reveals an organ-specific induction of cytoprotective enzymes in sickle cell disease.Hydroxyurea-induced expression of glutathione peroxidase 1 in red blood cells of individuals with sickle cell anemia GMI-1070, a novel pan-selectin antagonist, reverses acute vascular occlusions in sickle cell mice.Heme oxygenase-1 is a modulator of inflammation and vaso-occlusion in transgenic sickle mice.Mechanisms of enhanced thrombus formation in cerebral microvessels of mice expressing hemoglobin-S.Newer aspects of the pathophysiology of sickle cell disease vaso-occlusion.Vasculopathy in sickle cell disease: Biology, pathophysiology, genetics, translational medicine, and new research directions Pharmacotherapy in sickle cell disease--state of the art and future prospects Pleiotropic effects of intravascular haemolysis on vascular homeostasis.Sickle cell disease: old discoveries, new concepts, and future promise.Hemolysis in sickle cell mice causes pulmonary hypertension due to global impairment in nitric oxide bioavailability Genetic diminution of circulating prothrombin ameliorates multiorgan pathologies in sickle cell disease mice Redox-dependent impairment of vascular function in sickle cell disease.Granulocytic adhesive interactions and their role in sickle cell vaso-occlusion.Inflammatory targets of therapy in sickle cell disease.Placenta growth factor augments airway hyperresponsiveness via leukotrienes and IL-13.The influence of hydroxyurea on oxidative stress in sickle cell anemia Investigational agents for sickle cell disease.The genomics of new drugs in sickle cell disease.Erythrocyte NADPH oxidase activity modulated by Rac GTPases, PKC, and plasma cytokines contributes to oxidative stress in sickle cell disease.Sickle cell disease: role of reactive oxygen and nitrogen metabolites.Placenta growth factor induces 5-lipoxygenase-activating protein to increase leukotriene formation in sickle cell disease Original Research: Diametric effects of hypoxia on pathophysiology of sickle cell disease in a murine model.C-reactive protein and interleukin-6 are decreased in transgenic sickle cell mice fed a high protein diet.Electroacupuncture in conscious free-moving mice reduces pain by ameliorating peripheral and central nociceptive mechanisms.Hepatic Overexpression of Hemopexin Inhibits Inflammation and Vascular Stasis in Murine Models of Sickle Cell Disease.Inhaled carbon monoxide reduces leukocytosis in a murine model of sickle cell disease Oxidative stress in sickle cell disease; pathophysiology and potential implications for disease management.Novel therapies targeting the endothelium in sickle cell disease.Role of oxidative stress in the pathogenesis of sickle cell disease.Simvastatin reduces vaso-occlusive pain in sickle cell anaemia: a pilot efficacy trial.Proteasome inhibition induces both antioxidant and hb f responses in sickle cell disease via the nrf2 pathway.Astrogliosis is a possible player in preventing delayed neuronal death.H-ferritin ferroxidase induces cytoprotective pathways and inhibits microvascular stasis in transgenic sickle mice.Effect of sodium butyrate on lung vascular TNFSF15 (TL1A) expression: differential expression patterns in pulmonary artery and microvascular endothelial cells.

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P2860

Anti-inflammatory therapy ameliorates leukocyte adhesion and microvascular flow abnormalities in transgenic sickle mice.

http://www.wikidata.org/entity/Q44786816

description

2004 nî lūn-bûn

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2004年の論文

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Anti-inflammatory therapy amel ...... ies in transgenic sickle mice.

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Anti-inflammatory therapy amel ...... ies in transgenic sickle mice.

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Anti-inflammatory therapy amel ...... ies in transgenic sickle mice.

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Anti-inflammatory therapy amel ...... ies in transgenic sickle mice.

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Anti-inflammatory therapy amel ...... ies in transgenic sickle mice.

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Dhananjay K Kaul

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Gregory M Vercellotti

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John D Belcher

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Robert P Hebbel

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Stephana Choong

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Xiao-Du Liu

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P2860

Sulfasalazine: a potent and specific inhibitor of nuclear factor kappa B

Primary role for adherent leukocytes in sickle cell vascular occlusion: a new paradigm

Oxygen radical inhibition of nitric oxide-dependent vascular function in sickle cell disease.

Sickle cell anemia as an inflammatory disease

In vivo demonstration of red cell-endothelial interaction, sickling and altered microvascular response to oxygen in the sickle transgenic mouse

High expression of human beta S- and alpha-globins in transgenic mice: hemoglobin composition and hematological consequences

Tissue factor expression by endothelial cells in sickle cell anemia.

Two-step model of leukocyte-endothelial cell interaction in inflammation: distinct roles for LECAM-1 and the leukocyte beta 2 integrins in vivo.

In vivo visualization of reactive oxidants and leukocyte-endothelial adherence following hemorrhagic shock.

Hypoxia/reoxygenation causes inflammatory response in transgenic sickle mice but not in normal mice.

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