Granulocyte macrophage colony-stimulating factor signaling and proteasome inhibition delay neutrophil apoptosis by increasing the stability of Mcl-1.
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Proliferating cell nuclear antigen acts as a cytoplasmic platform controlling human neutrophil survivalBaicalein, a component of Scutellaria baicalensis, induces apoptosis by Mcl-1 down-regulation in human pancreatic cancer cellsApoptotic killing of HIV-1-infected macrophages is subverted by the viral envelope glycoproteinThr 163 phosphorylation causes Mcl-1 stabilization when degradation is independent of the adjacent GSK3-targeted phosphodegron, promoting drug resistance in cancerCyclin-dependent kinase 9 activity regulates neutrophil spontaneous apoptosisIron overload induces apoptosis of murine preosteoblast cells via ROS and inhibition of AKT pathway.Expression profile and specific network features of the apoptotic machinery explain relapse of acute myeloid leukemia after chemotherapy.Farnesoid X receptor protects liver cells from apoptosis induced by serum deprivation in vitro and fasting in vivo.Dynamic changes in Mcl-1 expression regulate macrophage viability or commitment to apoptosis during bacterial clearanceEffective caspase inhibition blocks neutrophil apoptosis and reveals Mcl-1 as both a regulator and a target of neutrophil caspase activation.Mcl-1 ubiquitination: unique regulation of an essential survival protein.IL-17A expression is localised to both mononuclear and polymorphonuclear synovial cell infiltrates.Small interfering RNA targeting mcl-1 enhances proteasome inhibitor-induced apoptosis in various solid malignant tumorsCommon genetic variations in the NALP3 inflammasome are associated with delayed apoptosis of human neutrophils.Anaplasma phagocytophilum delay of neutrophil apoptosis through the p38 mitogen-activated protein kinase signal pathway.Signal transducer and activators of transcription 3 regulates cryptotanshinone-induced apoptosis in human mucoepidermoid carcinoma cells.Serine 162, an essential residue for the mitochondrial localization, stability and anti-apoptotic function of Mcl-1.RNA-seq reveals activation of both common and cytokine-specific pathways following neutrophil priming.Granulocyte-macrophage colony-stimulating factor-transfected bone marrow stromal cells for the treatment of ischemic strokeParaquat-induced reactive oxygen species inhibit neutrophil apoptosis via a p38 MAPK/NF-κB-IL-6/TNF-α positive-feedback circuit.Inhibition of IRAK1/4 sensitizes T cell acute lymphoblastic leukemia to chemotherapies.Peculiarities of cell death mechanisms in neutrophils.IL-32γ delays spontaneous apoptosis of human neutrophils through MCL-1, regulated primarily by the p38 MAPK pathway.Increased serum soluble Fas after major trauma is associated with delayed neutrophil apoptosis and development of sepsisThe antiapoptotic protein Mcl-1 is essential for the survival of neutrophils but not macrophagesInfection with Anaplasma phagocytophilum activates the phosphatidylinositol 3-Kinase/Akt and NF-κB survival pathways in neutrophil granulocytes.IQGAP1 promotes CXCR4 chemokine receptor function and trafficking via EEA-1+ endosomesBay 61-3606 Sensitizes TRAIL-Induced Apoptosis by Downregulating Mcl-1 in Breast Cancer Cells.Apoptosis in lung injury and remodeling.Regulation of ubiquitination-mediated protein degradation by survival kinases in cancer.Molecular mechanisms underlying delayed apoptosis in neutrophils from multiple trauma patients with and without sepsisThe protein kinase C delta catalytic fragment targets Mcl-1 for degradation to trigger apoptosis.The differential effect of dexamethasone on granulocyte apoptosis involves stabilization of Mcl-1L in neutrophils but not in eosinophils.Bax/Mcl-1 balance affects neutrophil survival in intermittent hypoxia and obstructive sleep apnea: effects of p38MAPK and ERK1/2 signaling.Flavones induce neutrophil apoptosis by down-regulation of Mcl-1 via a proteasomal-dependent pathwayProteasome inhibitors: antitumor effects and beyond.Serum amyloid P inhibits granulocyte adhesionG-CSF activation of AKT is not sufficient to prolong neutrophil survivalBortezomib for the treatment of non-Hodgkin's lymphomaGastrin increases mcl-1 expression in type I gastric carcinoid tumors and a gastric epithelial cell line that expresses the CCK-2 receptor.
P2860
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P2860
Granulocyte macrophage colony-stimulating factor signaling and proteasome inhibition delay neutrophil apoptosis by increasing the stability of Mcl-1.
description
2004 nî lūn-bûn
@nan
2004年の論文
@ja
2004年学术文章
@wuu
2004年学术文章
@zh
2004年学术文章
@zh-cn
2004年学术文章
@zh-hans
2004年学术文章
@zh-my
2004年学术文章
@zh-sg
2004年學術文章
@yue
2004年學術文章
@zh-hant
name
Granulocyte macrophage colony- ...... easing the stability of Mcl-1.
@en
Granulocyte macrophage colony- ...... easing the stability of Mcl-1.
@nl
type
label
Granulocyte macrophage colony- ...... easing the stability of Mcl-1.
@en
Granulocyte macrophage colony- ...... easing the stability of Mcl-1.
@nl
prefLabel
Granulocyte macrophage colony- ...... easing the stability of Mcl-1.
@en
Granulocyte macrophage colony- ...... easing the stability of Mcl-1.
@nl
P2093
P2860
P356
P1476
Granulocyte macrophage colony- ...... easing the stability of Mcl-1.
@en
P2093
Andrew Cross
Luke Thomas
Mathieu Derouet
Robert J Moots
Steven W Edwards
P2860
P304
26915-26921
P356
10.1074/JBC.M313875200
P407
P577
2004-04-12T00:00:00Z