Role of AT1 receptors and NAD(P)H oxidase in diabetes-aggravated ischemic brain injury. - Wikidata - wikimedia - TriplyDB

Role of AT1 receptors and NAD(P)H oxidase in diabetes-aggravated ischemic brain injury.

Role of AT1 receptors and NAD(P)H oxidase in diabetes-aggravated ischemic brain injury.

http://www.wikidata.org/entity/Q44899587

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NADPH Oxidase: A Potential Target for Treatment of Stroke Natural compounds as modulators of NADPH oxidases Type-1 angiotensin receptor signaling in central nervous system myeloid cells is pathogenic during fatal alphavirus encephalitis in mice Atorvastatin protects against cerebral infarction via inhibition of NADPH oxidase-derived superoxide in ischemic stroke Protective effect of melatonin upon neuropathology, striatal function, and memory ability after intracerebral hemorrhage in rats.Dexamethasone inhibits the Nox-dependent ROS production via suppression of MKP-1-dependent MAPK pathways in activated microglia.Novel approach to reactive oxygen species in nontransfusion-dependent thalassemia Mechanisms contributing to cerebral infarct size after stroke: gender, reperfusion, T lymphocytes, and Nox2-derived superoxide Hyperglycemia as a Risk Factor of Ischemic Stroke NADPH oxidase mediates striatal neuronal injury after transient global cerebral ischemia.Microvascular responses to cardiovascular risk factors.Exacerbated brain damage, edema and inflammation in type-2 diabetic mice subjected to focal ischemia.NADPH oxidase inhibition improves neurological outcomes in surgically-induced brain injury.Reperfusion injury and reactive oxygen species: The evolution of a concept.Effects of Etanercept against Transient Cerebral Ischemia in Diabetic Rats.Hyperglycemia in acute ischemic stroke: a vascular perspective.Which NADPH oxidase isoform is relevant for ischemic stroke? The case for nox 2.Splenic immune cells in experimental neonatal hypoxia-ischemia Influence of exercise training on ischemic brain injury in type 1 diabetic rats Losartan improves impaired nitric oxide synthase-dependent dilatation of cerebral arterioles in type 1 diabetic rats.NADPH oxidase mediates hypersomnolence and brain oxidative injury in a murine model of sleep apnea.Inhibition of NADPH oxidase is neuroprotective after ischemia-reperfusion.Cerebral ischemic damage in diabetes: an inflammatory perspective.Renin-angiotensin system blockers and modulation of radiation-induced brain injury.The role of the blood transcriptome in innate inflammation and stroke.The neuroprotective effects of apocynin.NADPH oxidases as therapeutic targets in ischemic stroke.The immune system in stroke: clinical challenges and their translation to experimental research.Oxidative stress after subarachnoid hemorrhage in gp91phox knockout mice.Increased NADPH oxidase-derived superoxide is involved in the neuronal cell death induced by hypoxia-ischemia in neonatal hippocampal slice cultures.Fingolimod reduces cerebral lymphocyte infiltration in experimental models of rodent intracerebral hemorrhage.Suppression of NADPH oxidase- and mitochondrion-derived superoxide by Notoginsenoside R1 protects against cerebral ischemia-reperfusion injury through estrogen receptor-dependent activation of Akt/Nrf2 pathways.Hyperglycaemia promotes cerebral barrier dysfunction through activation of protein kinase C-β.Effects of apocynin and ethanol on intracerebral haemorrhage-induced brain injury in rats.Inducible nitric oxide synthase contributes to gender differences in ischemic brain injury.Melatonin decreases mortality following severe subarachnoid hemorrhage.15-Deoxy-∆12,14-PGJ 2, by activating peroxisome proliferator-activated receptor-gamma, suppresses p22phox transcription to protect brain endothelial cells against hypoxia-induced apoptosis.Oral supplementation with glycine reduces oxidative stress in patients with metabolic syndrome, improving their systolic blood pressure.Cardioprotective effects of melatonin against myocardial injuries induced by chronic intermittent hypoxia in rats.Is shorter transient middle cerebral artery occlusion (t-MCAO) duration better in stroke experiments on diabetic female Sprague Dawely rats?

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P2860

Role of AT1 receptors and NAD(P)H oxidase in diabetes-aggravated ischemic brain injury.

http://www.wikidata.org/entity/Q44899587

description

2004 nî lūn-bûn

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2004年の論文

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2004年学术文章

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2004年学术文章

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2004年学术文章

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2004年学术文章

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2004年学术文章

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2004年學術文章

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2004年學術文章

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2004年學術文章

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name

Role of AT1 receptors and NAD

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Role of AT1 receptors and NAD(P)H oxidase in diabetes-aggravated ischemic brain injury.

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type

label

Role of AT1 receptors and NAD

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Role of AT1 receptors and NAD(P)H oxidase in diabetes-aggravated ischemic brain injury.

@en

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Role of AT1 receptors and NAD

@nl

Role of AT1 receptors and NAD(P)H oxidase in diabetes-aggravated ischemic brain injury.

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American Journal of Physiology - Heart and Circulatory Physiology

P1476

Role of AT1 receptors and NAD(P)H oxidase in diabetes-aggravated ischemic brain injury.

@en

P2093

Anil Nanda

http://www.w3.org/2001/XMLSchema#string

Changman Zhou

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D Neil Granger

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Gen Kusaka

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Ikuyo Kusaka

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Jiping Tang

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Mami Ishikawa

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P2860

Distinction between surmountable and insurmountable selective AT1 receptor antagonists by use of CHO-K1 cells expressing human angiotensin II AT1 receptors

Angiotensin II AT(1) blockade normalizes cerebrovascular autoregulation and reduces cerebral ischemia in spontaneously hypertensive rats.

Altered expression of P(2) receptor mRNAs in the basilar artery in a rat double hemorrhage model.

Reactive oxygen radicals in signaling and damage in the ischemic brain.

Reactive oxygen species as mediators of angiotensin II signaling.

Free radical production and angiotensin.

Modulation of protein kinase activity and gene expression by reactive oxygen species and their role in vascular physiology and pathophysiology.

The role of hyperglycemia in acute stroke.

Role of augmented expression of intermediate-conductance Ca2+-activated K+ channels in postischaemic heart.

Effects of admission hyperglycemia on mortality and costs in acute ischemic stroke.

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