Progression of hair cell ejection and molecular markers of apoptosis in the avian cochlea following gentamicin treatment.
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Gene expression analysis of forskolin treated basilar papillae identifies microRNA181a as a mediator of proliferation.RNA granulesFunctional hair cell mechanotransducer channels are required for aminoglycoside ototoxicityMechanisms of aminoglycoside ototoxicity and targets of hair cell protection.Lead roles for supporting actors: critical functions of inner ear supporting cellsExtracellular divalent cations modulate aminoglycoside-induced hair cell death in the zebrafish lateral lineGap junctional coupling is essential for epithelial repair in the avian cochlea.Molecular regulation of auditory hair cell death and approaches to protect sensory receptor cells and/or stimulate repair following acoustic trauma.Supporting cell division is not required for regeneration of auditory hair cells after ototoxic injury in vitro.Inner ear supporting cells: rethinking the silent majority.Cisplatin and aminoglycoside antibiotics: hearing loss and its prevention.Intracellular mechanisms of aminoglycoside-induced cytotoxicity.Response of mechanosensory hair cells of the zebrafish lateral line to aminoglycosides reveals distinct cell death pathways.Hair cell fate decisions in cochlear development and regeneration5-Ethynyl-2'-deoxyuridine labeling detects proliferating cells in the regenerating avian cochleaLarge P body-like RNPs form in C. elegans oocytes in response to arrested ovulation, heat shock, osmotic stress, and anoxia and are regulated by the major sperm protein pathway.Hsp70 inhibits aminoglycoside-induced hearing loss and cochlear hair cell death.Geldanamycin induces production of heat shock protein 70 and partially attenuates ototoxicity caused by gentamicin in the organ of Corti explantsThe adult mouse utricle as an in vitro preparation for studies of ototoxic-drug-induced sensory hair cell death.Hsp70 inhibits aminoglycoside-induced hair cell death and is necessary for the protective effect of heat shock.Genetic and pharmacological intervention for treatment/prevention of hearing loss.Comparison of activated caspase detection methods in the gentamicin-treated chick cochleaJNK signaling in neomycin-induced vestibular hair cell death.Differential expression of unconventional myosins in apoptotic and regenerating chick hair cells confirms two regeneration mechanisms.Uptake of fluorescent gentamicin by vertebrate sensory cells in vivo.Caprin-1 is a target of the deafness gene Pou4f3 and is recruited to stress granules in cochlear hair cells in response to ototoxic damage.Rac/Rho pathway regulates actin depolymerization induced by aminoglycoside antibiotics.Mechanisms of noise-induced hearing loss indicate multiple methods of prevention.Mitochondrial calcium uptake underlies ROS generation during aminoglycoside-induced hair cell deathc-Myb knockdown increases the neomycin-induced damage to hair-cell-like HEI-OC1 cells in vitroProfiling drug-induced cell death pathways in the zebrafish lateral line.NaHS Protects Cochlear Hair Cells from Gentamicin-Induced Ototoxicity by Inhibiting the Mitochondrial Apoptosis PathwayCytoplasmic RNA Granules and Viral Infection.Granulosa cells express three inositol 1,4,5-trisphosphate receptor isoforms: cytoplasmic and nuclear Ca2+ mobilization.Morin hydrate promotes inner ear neural stem cell survival and differentiation and protects cochlea against neuronal hearing loss.Inhibition of ARC decreases the survival of HEI-OC-1 cells after neomycin damage in vitroTwo cell populations participate in clearance of damaged hair cells from the sensory epithelia of the inner ear.Chicken RNA-binding protein T-cell internal antigen-1 contributes to stress granule formation in chicken cells and tissues.Non-autonomous Cellular Responses to Ototoxic Drug-Induced Stress and Death.The role of molecular microtubule motors and the microtubule cytoskeleton in stress granule dynamics.
P2860
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P2860
Progression of hair cell ejection and molecular markers of apoptosis in the avian cochlea following gentamicin treatment.
description
2004 nî lūn-bûn
@nan
2004年の論文
@ja
2004年学术文章
@wuu
2004年学术文章
@zh-cn
2004年学术文章
@zh-hans
2004年学术文章
@zh-my
2004年学术文章
@zh-sg
2004年學術文章
@yue
2004年學術文章
@zh
2004年學術文章
@zh-hant
name
Progression of hair cell eject ...... ollowing gentamicin treatment.
@en
Progression of hair cell eject ...... ollowing gentamicin treatment.
@nl
type
label
Progression of hair cell eject ...... ollowing gentamicin treatment.
@en
Progression of hair cell eject ...... ollowing gentamicin treatment.
@nl
prefLabel
Progression of hair cell eject ...... ollowing gentamicin treatment.
@en
Progression of hair cell eject ...... ollowing gentamicin treatment.
@nl
P2093
P356
P1476
Progression of hair cell eject ...... ollowing gentamicin treatment.
@en
P2093
David C Mountain
Dominic A Mangiardi
Elizabeth P Messana
Kara E May
Katherine McLaughlin-Williamson
P356
10.1002/CNE.20129
P407
P577
2004-07-01T00:00:00Z