Activation of protein kinase C epsilon inhibits the two-pore domain K+ channel, TASK-1, inducing repolarization abnormalities in cardiac ventricular myocytes.
about
Regulation of two-pore-domain (K2P) potassium leak channels by the tyrosine kinase inhibitor genisteinThe acid-sensitive, anesthetic-activated potassium leak channel, KCNK3, is regulated by 14-3-3β-dependent, protein kinase C (PKC)-mediated endocytic trafficking.Doxapram hydrochloride aggravates adrenaline-induced arrhythmias accompanied by bidirectional ventricular tachycardiaTASK-1 current is inhibited by phosphorylation during human and canine chronic atrial fibrillation.TASK1 (K(2P)3.1) K(+) channel inhibition by endothelin-1 is mediated through Rho kinase-dependent phosphorylationPKC-dependent activation of human K(2P) 18.1 K(+) channels.Ability to induce atrial fibrillation in the peri-operative period is associated with phosphorylation-dependent inhibition of TWIK protein-related acid-sensitive potassium channel 1 (TASK-1)Neuronal two-pore-domain potassium channels and their regulation by G protein-coupled receptors.PPARalpha-mediated remodeling of repolarizing voltage-gated K+ (Kv) channels in a mouse model of metabolic cardiomyopathy.Molecular Basis of Functional Myocardial Potassium Channel Diversity.Protein kinase A and C regulate leak potassium currents in freshly isolated vascular myocytes from the aorta.Developmental expression of a functional TASK-1 2P domain K+ channel in embryonic chick heartPlatelet activating factor: the good and the bad in the ischemic/reperfused heart.Cardiac potassium channel subtypes: new roles in repolarization and arrhythmia.The role of acid-sensitive two-pore domain potassium channels in cardiac electrophysiology: focus on arrhythmias.Endocytosis as a mode to regulate functional expression of two-pore domain potassium (K₂p) channels.Effects of exercise training on excitation-contraction coupling and related mRNA expression in hearts of Goto-Kakizaki type 2 diabetic rats.Differential effects of volatile and intravenous anesthetics on the activity of human TASK-1.The inhibition of the potassium channel TASK-1 in rat cardiac muscle by endothelin-1 is mediated by phospholipase C.Carotid Body Type-I Cells Under Chronic Sustained Hypoxia: Focus on Metabolism and Membrane Excitability
P2860
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P2860
Activation of protein kinase C epsilon inhibits the two-pore domain K+ channel, TASK-1, inducing repolarization abnormalities in cardiac ventricular myocytes.
description
2004 nî lūn-bûn
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2004年の論文
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2004年学术文章
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2004年学术文章
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2004年学术文章
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2004年学术文章
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name
Activation of protein kinase C ...... cardiac ventricular myocytes.
@en
Activation of protein kinase C ...... cardiac ventricular myocytes.
@nl
type
label
Activation of protein kinase C ...... cardiac ventricular myocytes.
@en
Activation of protein kinase C ...... cardiac ventricular myocytes.
@nl
prefLabel
Activation of protein kinase C ...... cardiac ventricular myocytes.
@en
Activation of protein kinase C ...... cardiac ventricular myocytes.
@nl
P2093
P2860
P356
P1476
Activation of protein kinase C ...... cardiac ventricular myocytes.
@en
P2093
Alessandra Besana
Andrea Barbuti
Miyuki A Tateyama
Richard B Robinson
Steven J Feinmark
P2860
P304
33154-33160
P356
10.1074/JBC.M403525200
P407
P577
2004-06-07T00:00:00Z