about
Multiple regions within the cytoplasmic domains of the leukemia inhibitory factor receptor and gp130 cooperate in signal transduction in hepatic and neuronal cellsEnhancing leptin response by preventing SH2-containing phosphatase 2 interaction with Ob receptorThe protein tyrosine phosphatase SHP-2 negatively regulates ciliary neurotrophic factor induction of gene expressionChondroitin-4-sulfation negatively regulates axonal guidance and growthCandesartan, an angiotensin II AT₁-receptor blocker and PPAR-γ agonist, reduces lesion volume and improves motor and memory function after traumatic brain injury in mice.Temporal patterns of cortical proliferation of glial cell populations after traumatic brain injury in miceTGF-β superfamily gene expression and induction of the Runx1 transcription factor in adult neurogenic regions after brain injury.Commercially available angiotensin II At₂ receptor antibodies are nonspecific.Transforming growth factor β controls CCN3 expression in nucleus pulposus cells of the intervertebral disc.Smad proteins differentially regulate transforming growth factor-β-mediated induction of chondroitin sulfate proteoglycansCiliary neurotrophic factor coordinately activates transcription of neuropeptide genes in a neuroblastoma cell lineHepatic expression of serum amyloid A1 is induced by traumatic brain injury and modulated by telmisartanBone morphogenetic protein-2-mediated pain and inflammation in a rat model of posterolateral arthrodesis.Neurorestoration after traumatic brain injury through angiotensin II receptor blockageReceptor protein tyrosine phosphatase σ binds to neurons in the adult mouse brain.Bone morphogenetic protein-2 and spinal arthrodesis: the basic science perspective on protein interaction with the nervous system.The cAMP response element binding protein modulates expression of the transient outward current: implications for cardiac memory.Alterations in recovery from spinal cord injury in rats treated with recombinant human bone morphogenetic protein-2 for posterolateral arthrodesis.Temporal dynamics of cerebral blood flow, cortical damage, apoptosis, astrocyte-vasculature interaction and astrogliosis in the pericontusional region after traumatic brain injury.Runx1 promotes proliferation and neuronal differentiation in adult mouse neurosphere cultures.Structure and methylation of the human calcitonin/alpha-CGRP gene.Identification of a novel gp130-responsive site in the vasoactive intestinal peptide cytokine response element.Coordinate regulation of STAT signaling and c-fos expression by the tyrosine phosphatase SHP-2.Integration of Jak-Stat and AP-1 signaling pathways at the vasoactive intestinal peptide cytokine response element regulates ciliary neurotrophic factor-dependent transcription.STAT proteins are activated by ciliary neurotrophic factor in cells of central nervous system origin.Differences in nuclear signaling by leukemia inhibitory factor and interferon-gamma: the role of STAT proteins in regulating vasoactive intestinal peptide gene expression.A minimal CGRP gene promoter is inducible by nerve growth factor in adult rat dorsal root ganglion neurons but not in PC12 phaeochromocytoma cells.STAT proteins participate in the regulation of the vasoactive intestinal peptide gene by the ciliary neurotrophic factor family of cytokines.Oncostatin M regulates VIP expression in a human neuroblastoma cell line.Loss of transcriptional repression contributes to the ectopic expression of the calcitonin/alpha-CGRP gene in a human lung carcinoma cell line.LPS antagonism of TGF-β signaling results in prolonged survival and activation of rat primary microglia.Smad3 deficiency increases cortical and hippocampal neuronal loss following traumatic brain injury.NFAT4 is expressed in primary astrocytes and activated by glutamate.Activation of protein kinase C epsilon inhibits the two-pore domain K+ channel, TASK-1, inducing repolarization abnormalities in cardiac ventricular myocytes.Bone morphogenetic protein-2 used in spinal fusion with spinal cord injury penetrates intrathecally and elicits a functional signaling cascade.Histone Deacetylase Inhibition Rescues Maternal Deprivation-Induced GABAergic Metaplasticity through Restoration of AKAP Signaling.Regulation of nociceptin/orphanin FQ gene expression by neuropoietic cytokines and neurotrophic factors in neurons and astrocytes.Smad3 deficiency reduces neurogenesis in adult mice.Smad3 null mice display more rapid wound closure and reduced scar formation after a stab wound to the cerebral cortex.Introduction to special issue on traumatic brain injury.
P50
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P50
description
hulumtuese
@sq
onderzoeker
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researcher
@en
հետազոտող
@hy
name
Aviva J Symes
@ast
Aviva J Symes
@en
Aviva J Symes
@es
Aviva J Symes
@sl
type
label
Aviva J Symes
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Aviva J Symes
@en
Aviva J Symes
@es
Aviva J Symes
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Aviva J Symes
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Aviva J Symes
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Aviva J Symes
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Aviva J Symes
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P1053
S-7471-2016
P106
P21
P2798
P31
P3829
P496
0000-0003-2557-9939