Testosterone is responsible for enhanced susceptibility of males to ischemic renal injury. - Wikidata - wikimedia - TriplyDB

Testosterone is responsible for enhanced susceptibility of males to ischemic renal injury.

Testosterone is responsible for enhanced susceptibility of males to ischemic renal injury.

http://www.wikidata.org/entity/Q45052855

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Protective role of testosterone in ischemia-reperfusion-induced acute kidney injury Inhibition of MLK3-MKK4/7-JNK1/2 pathway by Akt1 in exogenous estrogen-induced neuroprotection against transient global cerebral ischemia by a non-genomic mechanism in male rats Use of ultrasound to assess renal reperfusion and P-selectin expression following unilateral renal ischemia A mitotic transcriptional switch in polycystic kidney disease Estrogen is renoprotective via a nonreceptor-dependent mechanism after cardiac arrest in vivo.Exocyst Sec10 protects epithelial barrier integrity and enhances recovery following oxidative stress, by activation of the MAPK pathway.Effect of gender differences on the regulation of renal ischemia-reperfusion-induced inflammation in mice.Estrogen-mediated renoprotection following cardiac arrest and cardiopulmonary resuscitation is robust to GPR30 gene deletion Progressive renal injury from transgenic expression of human carbonic anhydrase IV folding mutants is enhanced by deficiency of p58IPK.Preoperative fasting protects against renal ischemia-reperfusion injury in aged and overweight mice.Hormonal modulation of endothelial NO production.Restoration of tubular epithelial cells during repair of the postischemic kidney occurs independently of bone marrow-derived stem cells.Elevated testosterone levels during rat pregnancy cause hypersensitivity to angiotensin II and attenuation of endothelium-dependent vasodilation in uterine arteries Effect of testosterone on Cisplatin-induced nephrotoxicity in surgically castrated rats Gender difference and sex hormone production in rodent renal ischemia reperfusion injury and repair.Preventive role of estradiol on kidney injury induced by renal ischemia-reperfusion in male and female rats.Differential regulation of aortic growth in male and female rodents is associated with AAA development Improvement of tissue survival of skin flaps by 5α-reductase inhibitors: possible involvement of nitric oxide and inducible nitric oxide synthase.Estrogen administered after cardiac arrest and cardiopulmonary resuscitation ameliorates acute kidney injury in a sex- and age-specific manner.Increased fat deposition in injured skeletal muscle is regulated by sex-specific hormones.Aberrant macrophages mediate defective kidney repair that triggers nephritis in lupus-susceptible mice Unilateral Renal Ischemia-Reperfusion as a Robust Model for Acute to Chronic Kidney Injury in Mice Effect of Γ-aminobutyric acid on kidney injury induced by renal ischemia-reperfusion in male and female rats: Gender-related difference Meclizine Preconditioning Protects the Kidney Against Ischemia-Reperfusion Injury.A morphological investigation of sexual and lateral dimorphism in the developing metanephric kidney.Gender differences in murine airway responsiveness and lipopolysaccharide-induced inflammation.Mouse model of ischemic acute kidney injury: technical notes and tricks Preventive Role of Endothelin Antagonist on Kidney Ischemia: Reperfusion Injury in Male and Female Rats The Protective Effect of γ-aminobutyric Acid on Kidney Injury Induced by Renal Ischemia-reperfusion in Ovariectomized Estradiol-treated Rats.ET-1 actions in the kidney: evidence for sex differences.Testosterone alters maternal vascular adaptations: role of the endothelial NO system.Gender differences in the renin-angiotensin and nitric oxide systems: relevance in the normal and diseased kidney.Gender Difference in Renal Blood Flow Response to Angiotensin II Administration after Ischemia/Reperfusion in Rats: The Role of AT2 Receptor.Gender differences control the susceptibility to ER stress-induced acute kidney injury.Protective role of methionine sulfoxide reductase A against ischemia/reperfusion injury in mouse kidney and its involvement in the regulation of trans-sulfuration pathway The role of long-term label-retaining cells in the regeneration of adult mouse kidney after ischemia/reperfusion injury.Improved renal ischemia tolerance in females influences kidney transplantation outcomes.Sex steroids and cutaneous wound healing: the contrasting influences of estrogens and androgens.Myocardial AKT: the omnipresent nexus.C-reactive protein exacerbates renal ischemia-reperfusion injury

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P2860

Testosterone is responsible for enhanced susceptibility of males to ischemic renal injury.

http://www.wikidata.org/entity/Q45052855

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2004 nî lūn-bûn

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2004年の論文

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2004年学术文章

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2004年学术文章

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2004年學術文章

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Testosterone is responsible fo ...... ales to ischemic renal injury.

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Testosterone is responsible fo ...... ales to ischemic renal injury.

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Testosterone is responsible fo ...... ales to ischemic renal injury.

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Testosterone is responsible fo ...... ales to ischemic renal injury.

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Testosterone is responsible fo ...... ales to ischemic renal injury.

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Testosterone is responsible fo ...... ales to ischemic renal injury.

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Testosterone is responsible fo ...... ales to ischemic renal injury.

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P2093

Andrew J Bonventre

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Jee In Kim

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Joseph V Bonventre

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Kwon Moo Park

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Youngkeun Ahn

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P2860

Akt phosphorylation of BAD couples survival signals to the cell-intrinsic death machinery

Yama/CPP32 beta, a mammalian homolog of CED-3, is a CrmA-inhibitable protease that cleaves the death substrate poly(ADP-ribose) polymerase

Interaction of oestrogen receptor with the regulatory subunit of phosphatidylinositol-3-OH kinase

Androgen exposure increases human monocyte adhesion to vascular endothelium and endothelial cell expression of vascular cell adhesion molecule-1.

Ovariectomy exacerbates and estrogen replacement attenuates photothrombotic focal ischemic brain injury in rats.

Nitric oxide in acute renal failure: NOS versus NOS.

Testosterone signaling in T cells and macrophages.

Induction of calcium-dependent nitric oxide synthases by sex hormones.

Intercellular adhesion molecule-1-deficient mice are protected against ischemic renal injury.

Regulation of Fas expression by STAT3 and c-Jun is mediated by phosphatidylinositol 3-kinase-AKT signaling.

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279

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2004-09-08T00:00:00Z

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