The E693Δ (Osaka) mutation in amyloid precursor protein potentiates cholesterol-mediated intracellular amyloid β toxicity via its impaired cholesterol efflux.
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Amyloid β Protein and Alzheimer's Disease: When Computer Simulations Complement Experimental Studies.Protein accumulation in the endoplasmic reticulum as a non-equilibrium phase transition.Oligomeric amyloid-beta induces MAPK-mediated activation of brain cytosolic and calcium-independent phospholipase A2 in a spatial-specific manner.Cholesterol impairment contributes to neuroserpin aggregation.Alteration of high-density lipoprotein functionality in Alzheimer's disease patients.Rifampicin is a candidate preventive medicine against amyloid-β and tau oligomers.
P2860
The E693Δ (Osaka) mutation in amyloid precursor protein potentiates cholesterol-mediated intracellular amyloid β toxicity via its impaired cholesterol efflux.
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name
The E693Δ (Osaka) mutation in ...... s impaired cholesterol efflux.
@en
The E693Δ
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type
label
The E693Δ (Osaka) mutation in ...... s impaired cholesterol efflux.
@en
The E693Δ
@nl
prefLabel
The E693Δ (Osaka) mutation in ...... s impaired cholesterol efflux.
@en
The E693Δ
@nl
P2093
P2860
P356
P1476
The E693Δ (Osaka) mutation in ...... s impaired cholesterol efflux.
@en
P2093
Hiroshi Mori
Sachiko Nomura
Takami Tomiyama
Tomohiro Umeda
P2860
P304
P356
10.1002/JNR.23278
P577
2013-09-16T00:00:00Z