Hypercapnia via reduced rate and tidal volume contributes to lipopolysaccharide-induced lung injury. - Wikidata - wikimedia - TriplyDB

Hypercapnia via reduced rate and tidal volume contributes to lipopolysaccharide-induced lung injury.

Hypercapnia via reduced rate and tidal volume contributes to lipopolysaccharide-induced lung injury.

http://www.wikidata.org/entity/Q45101481

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Acute respiratory distress syndrome and lung injury: Pathogenetic mechanism and therapeutic implication Acute respiratory distress syndrome induced by a swine 2009 H1N1 variant in mice Activation of GPR4 by acidosis increases endothelial cell adhesion through the cAMP/Epac pathway Effect of low-level CO2 on innate inflammatory protein response to organic dust from swine confinement barns AMP-activated protein kinase regulates CO2-induced alveolar epithelial dysfunction in rats and human cells by promoting Na,K-ATPase endocytosis Effects of age on the synergistic interactions between lipopolysaccharide and mechanical ventilation in mice.Suspended animation inducer hydrogen sulfide is protective in an in vivo model of ventilator-induced lung injury High frequency oscillatory ventilation and prone positioning in a porcine model of lavage-induced acute lung injury High CO2 levels impair alveolar epithelial function independently of pH.Functional consequences of actin nitration: in vitro and in disease states.Very low tidal volume ventilation with associated hypercapnia--effects on lung injury in a model for acute respiratory distress syndrome Extracellular signal-regulated kinase (ERK) participates in the hypercapnia-induced Na,K-ATPase downregulation.Sensing, physiological effects and molecular response to elevated CO2 levels in eukaryotes Acute lung injury and acute respiratory distress syndrome: experimental and clinical investigations.Carnosine markedly ameliorates H9N2 swine influenza virus-induced acute lung injury.Update in critical care 2005.Lung protective ventilatory strategies in acute lung injury and acute respiratory distress syndrome: from experimental findings to clinical application.Identification of miRNomes reveals ssc-miR-30d-R_1 as a potential therapeutic target for PRRS viral infection.Modulatory effects of hypercapnia on in vitro and in vivo pulmonary endothelial-neutrophil adhesive responses during inflammation.Impact of buffering hypercapnic acidosis on cell wounding in ventilator-injured rat lungs.Bactericidal function of alveolar macrophages in mechanically ventilated rabbits Two Genetically Similar H9N2 Influenza A Viruses Show Different Pathogenicity in Mice.Bench-to-bedside review: hypercapnic acidosis in lung injury--from 'permissive' to 'therapeutic'.A nonpathogenic duck-origin H9N2 influenza A virus adapts to high pathogenicity in mice.Effect of Mild Hypercapnia on Lung Oxygenation in Sitting Position During Shoulder Arthroscopy Under General Anesthesia.Inducible nitric oxide synthase contributes to ventilator-induced lung injury.Hypercapnic acidosis impairs plasma membrane wound resealing in ventilator-injured lungs.Effects of hypercapnia and NO synthase inhibition in sustained hypoxic pulmonary vasoconstriction.[Permissive and non-permissive hypercapnia: mechanisms of action and consequences of high carbon dioxide levels].Acute respiratory distress syndrome induced by avian influenza A (H5N1) virus in mice.The effect of hemolysis on plasma oxidation and nitration in patients with sickle cell disease.Outcome of extremely preterm infants randomized at birth to different PaCO2 targets during the first seven days of life.Adverse influence of mixed acidemia on the biocompatibility of continuous veno-venous hemofiltration with respect to the lungs.Inflammatory Mediators in Tracheal Aspirates of Preterm Infants Participating in a Randomized Trial of Permissive Hypercapnia.Therapeutic hypercapnia: careful science, better trials.Endotoxin-induced acute lung injury and organ dysfunction are attenuated by pentobarbital anaesthesia.The contribution of arterio-venous extracorporeal lung assist to gas exchange in a porcine model of lavage-induced acute lung injury.Therapeutic hypercapnia prevents chronic hypoxia-induced pulmonary hypertension in the newborn rat

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Hypercapnia via reduced rate and tidal volume contributes to lipopolysaccharide-induced lung injury.

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Hypercapnia via reduced rate a ...... accharide-induced lung injury.

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Hypercapnia via reduced rate a ...... accharide-induced lung injury.

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Bruce A Freeman

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John D Lang

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K David Sanders

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Mario Figueroa

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Mutay Aslan

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Phillip Chumley

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Yuliang Liu

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P2860

Ventilation with lower tidal volumes as compared with traditional tidal volumes for acute lung injury and the acute respiratory distress syndrome. The Acute Respiratory Distress Syndrome Network

Oxidative chemistry of nitric oxide: the roles of superoxide, peroxynitrite, and carbon dioxide

Nitration of proteins in bronchoalveolar lavage fluid from patients with acute respiratory distress syndrome receiving inhaled nitric oxide

Apparent hydroxyl radical production by peroxynitrite: implications for endothelial injury from nitric oxide and superoxide

Microtubule dysfunction by posttranslational nitrotyrosination of alpha-tubulin: a nitric oxide-dependent mechanism of cellular injury

Nitric oxide-dependent generation of reactive species in sickle cell disease. Actin tyrosine induces defective cytoskeletal polymerization.

Nitric oxide and nitrotyrosine in the lungs of patients with acute respiratory distress syndrome.

Controlled airway pressure therapy, nitric oxide inhalation, prone position, and extracorporeal membrane oxygenation (ECMO) as components of an integrated approach to ARDS.

Effect of a protective-ventilation strategy on mortality in the acute respiratory distress syndrome.

Repair of deaminated bases in DNA.

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