Hypoxic postconditioning reduces cardiomyocyte loss by inhibiting ROS generation and intracellular Ca2+ overload. - Wikidata - wikimedia - TriplyDB

Hypoxic postconditioning reduces cardiomyocyte loss by inhibiting ROS generation and intracellular Ca2+ overload.

Hypoxic postconditioning reduces cardiomyocyte loss by inhibiting ROS generation and intracellular Ca2+ overload.

http://www.wikidata.org/entity/Q45162551

about

Role of the MPTP in conditioning the heart - translatability and mechanism Ischaemic postconditioning: cardiac protection after the event Hypoxemic reperfusion of ischemic states: an alternative approach for the attenuation of oxidative stress mediated reperfusion injury The compound Chinese medicine "Kang Fu Ling" protects against high power microwave-induced myocardial injury Nonanticoagulant heparin reduces myocyte Na+ and Ca2+ loading during simulated ischemia and decreases reperfusion injury.Calcium-sensing receptors regulate cardiomyocyte Ca2+ signaling via the sarcoplasmic reticulum-mitochondrion interface during hypoxia/reoxygenation.PLCδ1 protein rescues ischemia-reperfused heart by the regulation of calcium homeostasis Endogenous adenosine selectively modulates oxidant stress via the A1 receptor in ischemic hearts.Isoflurane protects cardiomyocytes and mitochondria by immediate and cytosol-independent action at reperfusion.Protective effects of early hypoxic post-conditioning in cultured cortical neurons.gp-91 mediates histone deacetylase inhibition-induced cardioprotection.Diazoxide, a K(ATP) channel opener, prevents ischemia-reperfusion injury in rodent pancreatic islets.Evaluation of cardiomyocyte hypoxia injury models for the pharmacological study in vitro.Potential therapeutic benefits of strategies directed to mitochondria.Mitochondrial K+ channels are involved in ischemic postconditioning in rat hearts.Protection of ghrelin postconditioning on hypoxia/reoxygenation in gastric epithelial cells Ischemic preconditioning attenuates mitochondrial localization of PTEN induced by ischemia-reperfusion Ischemic postconditioning protects cardiomyocytes against ischemia/reperfusion injury by inducing MIP2 Endogenous cardioprotection by ischaemic postconditioning and remote conditioning.Decreased autophagy: a major factor for cardiomyocyte death induced by β1-adrenoceptor autoantibodies.Protective effect of reduced glutathione C60 derivative against hydrogen peroxide-induced apoptosis in HEK 293T cells.Reactive oxygen species trigger ischemic and pharmacological postconditioning: in vivo and in vitro characterization Role of vitamin D3 combined to alginates in preventing acid and oxidative injury in cultured gastric epithelial cells.Cardioprotection and mitochondrial S-nitrosation: effects of S-nitroso-2-mercaptopropionyl glycine (SNO-MPG) in cardiac ischemia-reperfusion injury Realizing the clinical potential of ischemic preconditioning and postconditioning.Endogenous hydrogen sulfide mediates the cardioprotection induced by ischemic postconditioning in the early reperfusion phase.Curcumin promotes cardiac repair and ameliorates cardiac dysfunction following myocardial infarction CYP1A in TCDD toxicity and in physiology-with particular reference to CYP dependent arachidonic acid metabolism and other endogenous substrates.Modulation of electron transport protects cardiac mitochondria and decreases myocardial injury during ischemia and reperfusion.Tanshinone IIA and Cryptotanshinone Prevent Mitochondrial Dysfunction in Hypoxia-Induced H9c2 Cells: Association to Mitochondrial ROS, Intracellular Nitric Oxide, and Calcium Levels Protective effect of bioactive compounds from Lonicera japonica Thunb. against H2O2-induced cytotoxicity using neonatal rat cardiomyocytes Preconditioning and postconditioning: new strategies for cardioprotection.Protective effects of vitamin D3 on fimbrial cells exposed to catalytic iron damage Ischemic postconditioning protects the heart against ischemia-reperfusion injury via neuronal nitric oxide synthase in the sarcoplasmic reticulum and mitochondria The paradigm of postconditioning to protect the heart.Stanniocalcin-1 Protects a Mouse Model from Renal Ischemia-Reperfusion Injury by Affecting ROS-Mediated Multiple Signaling Pathways Cardioprotection by ischemic postconditioning during surgical procedures.Protective ischaemia in patients: preconditioning and postconditioning.Critical role of mitochondrial ROS is dependent on their site of production on the electron transport chain in ischemic heart Acute hyperglycaemia enhances oxidative stress and aggravates myocardial ischaemia/reperfusion injury: role of thioredoxin-interacting protein.

P2860

Q26830560-1FC15142-636C-4E77-B66B-E71C817B5F6C Q26853129-DB0CBF9C-8C1A-4320-9A56-1A20C065A9F9 Q28066715-EFB38717-7A4C-4FD6-A126-12B92A4AEE10 Q28540340-292CE25A-8A51-4FE8-9FCD-C8CC252F0867 Q33589221-25355FDE-C152-4EF0-8D10-A02490CB9D2F Q33610970-A2B4D124-8738-4F59-BF03-C53E82B9EE7F Q33724248-58A4D7A5-5505-4717-A387-56521C665E9C Q33818121-7C8AA3C1-D2FC-4C21-B38D-62735A30BC88 Q33870380-8059B366-021E-4877-AFA3-C0F19732E21E Q33887273-CECD1185-183E-4140-AE98-F094E47ED7EF Q33892798-0340B2C6-7D9F-4A0A-A59D-D4BC773AA800 Q33954456-D0075C52-E445-407A-BD9A-8F4265AF0F4C Q34074793-950FE968-CD00-41F2-A80E-70066238DBD5 Q34117168-4344046D-86B6-48C5-9AA2-AF586269E6CC Q34243115-2375CB60-0D5E-4E1B-B17D-AB54153C5EFE Q34451810-87F34E68-AFA3-4907-9511-19BEDADE3BC2 Q35056941-50A17889-1EA9-4C54-83F1-A1D0ED0ED2B5 Q35216382-85D9A030-193D-4BF1-AEF6-41727DF5BB9B Q35902467-F5F7ED5D-E92E-4532-9B1C-FB46A598597F Q36022333-4B5C084C-3FE3-4EAE-BCAB-5CBF2E276A17 Q36067528-68F2C88E-265F-48DA-AE54-313EC74D0096 Q36149032-17645011-2352-4DE6-8293-9E234096E868 Q36157989-7A38C4DE-B296-4D8F-A44B-3A8EE1353514 Q36259597-DAEE96C1-9639-4E37-B6B6-B03C7709797A Q36301135-7ED52591-3376-425E-8693-6879A185A09E Q36386591-35F263B8-0941-4B03-85A0-6A8E32D66ABD Q36446530-B77DE36C-85D5-4AE8-8CDA-22233ED17460 Q36473507-451B45E5-80CE-4048-AC17-B4867C6F6D2D Q36593116-B9098FE0-DE9A-4ABC-A733-8F3BAA2C437C Q36702981-764837F8-E232-4130-9FAB-719754B1241C Q36773830-92E7CE59-44C8-448B-BDB1-31DE7DA5EA86 Q36933844-1EBF79E4-FA05-4015-81B4-F82DC63F47D5 Q37017956-DD101D4C-E0C3-4A36-89D8-CDE07927A1FC Q37030270-B130B111-296A-4E9C-AB53-22337CC7D4CE Q37053179-BC4D2058-06C0-49B7-A785-58D11BFF16E8 Q37134433-C40BFCEB-8D33-4306-BA8F-67EB7AFCB0A3 Q37230400-52D4F757-4B87-4858-A3EA-FDAB4A944C9D Q37240955-207DB8DE-5AD7-43FE-AF16-7ED9B6440968 Q37271027-F9DB699A-2C9C-49A8-8551-B66B54AD789A Q37295345-F7B97F0F-F9F5-41D0-B973-DF892EC89C46

P2860

Hypoxic postconditioning reduces cardiomyocyte loss by inhibiting ROS generation and intracellular Ca2+ overload.

http://www.wikidata.org/entity/Q45162551

description

2004 nî lūn-bûn

@nan

2004年の論文

@ja

2004年学术文章

@wuu

2004年学术文章

@zh

2004年学术文章

@zh-cn

2004年学术文章

@zh-hans

2004年学术文章

@zh-my

2004年学术文章

@zh-sg

2004年學術文章

@yue

2004年學術文章

@zh-hant

name

Hypoxic postconditioning reduc ...... d intracellular Ca2+ overload.

@en

Hypoxic postconditioning reduc ...... d intracellular Ca2+ overload.

@nl

type

label

Hypoxic postconditioning reduc ...... d intracellular Ca2+ overload.

@en

Hypoxic postconditioning reduc ...... d intracellular Ca2+ overload.

@nl

prefLabel

Hypoxic postconditioning reduc ...... d intracellular Ca2+ overload.

@en

Hypoxic postconditioning reduc ...... d intracellular Ca2+ overload.

@nl

P1433

Q45162551-99408AB6-7BF3-4646-A8E6-737DB535EEF8

P1476

Q45162551-C7A84A5C-D0D5-4CBD-A5AB-58C0949FAE8E

P2093

Q45162551-2AA0F7C2-0EEC-49E7-A0C6-BB1469821F3F

Q45162551-433F0BA5-5BA7-4024-9F37-0BCC514321C0

Q45162551-4788B62B-9268-48B4-B39C-9CA373B399B6

Q45162551-B9C7BE31-9B93-4677-AF29-B4EF73B4DBD6

Q45162551-D801CDEA-C84F-428F-9FD0-F150378361DC

Q45162551-DDA1D043-D7BC-4242-856C-B044C6D3CBFE

Q45162551-FAD1E27E-3151-44A3-85AF-17E6AA0E87AE

P304

Q45162551-867E23A4-C669-49B9-B29F-DC9F2CF296AB

P31

Q45162551-2043C8E6-E87E-43AA-B471-867B7BD01B14

P356

Q45162551-6D51FFEC-D2B5-4D10-903C-0F55B3E52F51

P433

Q45162551-3EC21707-0059-4ECB-A3DA-F7DD80A07D12

P478

Q45162551-0078C230-415A-4D3A-8EA1-3F250CAF1D1C

P50

Q45162551-B224C8A7-A38C-4847-9ACC-17C2B14CAD4F

P577

Q45162551-F4408905-E618-451A-A54C-C8AA8DD4ECF1

P698

Q45162551-BBEC970E-ABAF-4E85-9227-3CAC0D33C1E8

P921

Q45162551-0FCEBA87-716B-4D8F-85ED-2265A9BC6856

P356

AJPHEART.01244.2003

P1433

American Journal of Physiology - Heart and Circulatory Physiology

P1476

Hypoxic postconditioning reduc ...... d intracellular Ca2+ overload.

@en

P2093

Faraz Kerendi

http://www.w3.org/2001/XMLSchema#string

Hajime Kin

http://www.w3.org/2001/XMLSchema#string

He-Ying Sun

http://www.w3.org/2001/XMLSchema#string

Jakob Vinten-Johansen

http://www.w3.org/2001/XMLSchema#string

Ning-Ping Wang

http://www.w3.org/2001/XMLSchema#string

Robert A Guyton

http://www.w3.org/2001/XMLSchema#string

Zhi-Qing Zhao

http://www.w3.org/2001/XMLSchema#string

P304

H1900-8

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1152/AJPHEART.01244.2003

http://www.w3.org/2001/XMLSchema#string

P433

4

http://www.w3.org/2001/XMLSchema#string

P478

288

http://www.w3.org/2001/XMLSchema#string

P50

Michael E Halkos

P577

2004-11-24T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P698

15563525

http://www.w3.org/2001/XMLSchema#string

P921