Hypoxic postconditioning reduces cardiomyocyte loss by inhibiting ROS generation and intracellular Ca2+ overload.
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Role of the MPTP in conditioning the heart - translatability and mechanismIschaemic postconditioning: cardiac protection after the eventHypoxemic reperfusion of ischemic states: an alternative approach for the attenuation of oxidative stress mediated reperfusion injuryThe compound Chinese medicine "Kang Fu Ling" protects against high power microwave-induced myocardial injuryNonanticoagulant heparin reduces myocyte Na+ and Ca2+ loading during simulated ischemia and decreases reperfusion injury.Calcium-sensing receptors regulate cardiomyocyte Ca2+ signaling via the sarcoplasmic reticulum-mitochondrion interface during hypoxia/reoxygenation.PLCδ1 protein rescues ischemia-reperfused heart by the regulation of calcium homeostasisEndogenous adenosine selectively modulates oxidant stress via the A1 receptor in ischemic hearts.Isoflurane protects cardiomyocytes and mitochondria by immediate and cytosol-independent action at reperfusion.Protective effects of early hypoxic post-conditioning in cultured cortical neurons.gp-91 mediates histone deacetylase inhibition-induced cardioprotection.Diazoxide, a K(ATP) channel opener, prevents ischemia-reperfusion injury in rodent pancreatic islets.Evaluation of cardiomyocyte hypoxia injury models for the pharmacological study in vitro.Potential therapeutic benefits of strategies directed to mitochondria.Mitochondrial K+ channels are involved in ischemic postconditioning in rat hearts.Protection of ghrelin postconditioning on hypoxia/reoxygenation in gastric epithelial cellsIschemic preconditioning attenuates mitochondrial localization of PTEN induced by ischemia-reperfusionIschemic postconditioning protects cardiomyocytes against ischemia/reperfusion injury by inducing MIP2Endogenous cardioprotection by ischaemic postconditioning and remote conditioning.Decreased autophagy: a major factor for cardiomyocyte death induced by β1-adrenoceptor autoantibodies.Protective effect of reduced glutathione C60 derivative against hydrogen peroxide-induced apoptosis in HEK 293T cells.Reactive oxygen species trigger ischemic and pharmacological postconditioning: in vivo and in vitro characterizationRole of vitamin D3 combined to alginates in preventing acid and oxidative injury in cultured gastric epithelial cells.Cardioprotection and mitochondrial S-nitrosation: effects of S-nitroso-2-mercaptopropionyl glycine (SNO-MPG) in cardiac ischemia-reperfusion injuryRealizing the clinical potential of ischemic preconditioning and postconditioning.Endogenous hydrogen sulfide mediates the cardioprotection induced by ischemic postconditioning in the early reperfusion phase.Curcumin promotes cardiac repair and ameliorates cardiac dysfunction following myocardial infarctionCYP1A in TCDD toxicity and in physiology-with particular reference to CYP dependent arachidonic acid metabolism and other endogenous substrates.Modulation of electron transport protects cardiac mitochondria and decreases myocardial injury during ischemia and reperfusion.Tanshinone IIA and Cryptotanshinone Prevent Mitochondrial Dysfunction in Hypoxia-Induced H9c2 Cells: Association to Mitochondrial ROS, Intracellular Nitric Oxide, and Calcium LevelsProtective effect of bioactive compounds from Lonicera japonica Thunb. against H2O2-induced cytotoxicity using neonatal rat cardiomyocytesPreconditioning and postconditioning: new strategies for cardioprotection.Protective effects of vitamin D3 on fimbrial cells exposed to catalytic iron damageIschemic postconditioning protects the heart against ischemia-reperfusion injury via neuronal nitric oxide synthase in the sarcoplasmic reticulum and mitochondriaThe paradigm of postconditioning to protect the heart.Stanniocalcin-1 Protects a Mouse Model from Renal Ischemia-Reperfusion Injury by Affecting ROS-Mediated Multiple Signaling PathwaysCardioprotection by ischemic postconditioning during surgical procedures.Protective ischaemia in patients: preconditioning and postconditioning.Critical role of mitochondrial ROS is dependent on their site of production on the electron transport chain in ischemic heartAcute hyperglycaemia enhances oxidative stress and aggravates myocardial ischaemia/reperfusion injury: role of thioredoxin-interacting protein.
P2860
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P2860
Hypoxic postconditioning reduces cardiomyocyte loss by inhibiting ROS generation and intracellular Ca2+ overload.
description
2004 nî lūn-bûn
@nan
2004年の論文
@ja
2004年学术文章
@wuu
2004年学术文章
@zh
2004年学术文章
@zh-cn
2004年学术文章
@zh-hans
2004年学术文章
@zh-my
2004年学术文章
@zh-sg
2004年學術文章
@yue
2004年學術文章
@zh-hant
name
Hypoxic postconditioning reduc ...... d intracellular Ca2+ overload.
@en
Hypoxic postconditioning reduc ...... d intracellular Ca2+ overload.
@nl
type
label
Hypoxic postconditioning reduc ...... d intracellular Ca2+ overload.
@en
Hypoxic postconditioning reduc ...... d intracellular Ca2+ overload.
@nl
prefLabel
Hypoxic postconditioning reduc ...... d intracellular Ca2+ overload.
@en
Hypoxic postconditioning reduc ...... d intracellular Ca2+ overload.
@nl
P2093
P1476
Hypoxic postconditioning reduc ...... d intracellular Ca2+ overload.
@en
P2093
Faraz Kerendi
Hajime Kin
He-Ying Sun
Jakob Vinten-Johansen
Ning-Ping Wang
Robert A Guyton
Zhi-Qing Zhao
P304
P356
10.1152/AJPHEART.01244.2003
P577
2004-11-24T00:00:00Z