Altered expression of COX-1, COX-2, and mPGES in rats with nephrogenic and central diabetes insipidus.
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Bypassing vasopressin receptor signaling pathways in nephrogenic diabetes insipidusPhysiology and pathophysiology of cyclooxygenase-2 and prostaglandin E2 in the kidneyEffects of lithium on lipopolysaccharide-induced inflammation in rat primary glia cellsPotential role of purinergic signaling in lithium-induced nephrogenic diabetes insipidusRenal cortical regulation of COX-1 and functionally related products in early renovascular hypertension (rat).Mice lacking mPGES-1 are resistant to lithium-induced polyuria.GSK3beta mediates renal response to vasopressin by modulating adenylate cyclase activity.A maladaptive role for EP4 receptors in mouse mesangial cells.Glycogen synthase kinase 3α regulates urine concentrating mechanism in mice.Immunohistochemical Expressions of Main PGE(2) Biosynthesis-related Enzymes and PGE(2) Receptor in Rat NephrogenesisInhibition of glycogen synthase kinase-3β prevents NSAID-induced acute kidney injury.Renal Fibrosis mRNA Classifier: Validation in Experimental Lithium-Induced Interstitial Fibrosis in the Rat Kidney.Lithium: a versatile tool for understanding renal physiology.Glycogen synthase kinase-3 regulation of urinary concentrating abilityDysregulation of Renal Cyclooxygenase-2 in Rats with Lithium-induced Nephrogenic Diabetes Insipidus.Lithium induces microcysts and polyuria in adolescent rat kidney independent of cyclooxygenase-2.Cyclooxygenase metabolites in the kidney.Molecular mechanisms in lithium-associated renal disease: a systematic review.Relationship of Cell Proliferating Marker Expressions with PGE(2) Receptors in Regenerating Rat Renal Tubules after Cisplatin InjectionStimulation of UT-A1-mediated transepithelial urea flux in MDCK cells by lithium.V2 vasopressin receptor deficiency causes changes in expression and function of renal and hypothalamic components involved in electrolyte and water homeostasis.Hydrochlorothiazide attenuates lithium-induced nephrogenic diabetes insipidus independently of the sodium-chloride cotransporter.Selectivity of cyclooxygenase isoform activity and prostanoid production in normal and diseased Han:SPRD-cy rat kidneys.Amiloride restores renal medullary osmolytes in lithium-induced nephrogenic diabetes insipidus.Calcineurin inhibitor cyclosporine A activates renal Na-K-Cl cotransporters via local and systemic mechanisms.COX-2 activity transiently contributes to increased water and NaCl excretion in the polyuric phase after release of ureteral obstruction.Brain death increases COX-1 and COX-2 expression in the renal medulla in a pig model.Cisplatin decreases renal cyclooxygenase-2 expression and activity in rats.Lithium reduces aquaporin-2 transcription independent of prostaglandins.
P2860
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P2860
Altered expression of COX-1, COX-2, and mPGES in rats with nephrogenic and central diabetes insipidus.
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年学术文章
@wuu
2005年学术文章
@zh
2005年学术文章
@zh-cn
2005年学术文章
@zh-hans
2005年学术文章
@zh-my
2005年学术文章
@zh-sg
2005年學術文章
@yue
2005年學術文章
@zh-hant
name
Altered expression of COX-1, C ...... nd central diabetes insipidus.
@en
Altered expression of COX-1, C ...... nd central diabetes insipidus.
@nl
type
label
Altered expression of COX-1, C ...... nd central diabetes insipidus.
@en
Altered expression of COX-1, C ...... nd central diabetes insipidus.
@nl
prefLabel
Altered expression of COX-1, C ...... nd central diabetes insipidus.
@en
Altered expression of COX-1, C ...... nd central diabetes insipidus.
@nl
P2093
P2860
P1476
Altered expression of COX-1, C ...... nd central diabetes insipidus.
@en
P2093
Ciril Krzisnik
Jakob Nielsen
Jørgen Frøkiaer
Primoz Kotnik
Søren Nielsen
Tae-Hwan Kwon
P2860
P304
P356
10.1152/AJPRENAL.00114.2004
P577
2005-01-11T00:00:00Z