The IFN-independent response to virus particle entry provides a first line of antiviral defense that is independent of TLRs and retinoic acid-inducible gene I.
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Biology of Zika Virus Infection in Human Skin CellsChikungunya virus induces IPS-1-dependent innate immune activation and protein kinase R-independent translational shutoffProcessing of Genome 5′ Termini as a Strategy of Negative-Strand RNA Viruses to Avoid RIG-I-Dependent Interferon InductionHMGB proteins function as universal sentinels for nucleic-acid-mediated innate immune responsesReceptor-interacting protein homotypic interaction motif-dependent control of NF-kappa B activation via the DNA-dependent activator of IFN regulatory factorsHuman cytomegalovirus induces the interferon response via the DNA sensor ZBP1.Host responses in human skin after conventional intradermal injection or microneedle administration of virus-like-particle influenza vaccine.Cellular localization of the herpes simplex virus ICP0 protein dictates its ability to block IRF3-mediated innate immune responsesHuman neuronal cells possess functional cytoplasmic and TLR-mediated innate immune pathways influenced by phosphatidylinositol-3 kinase signaling.Recognition of virus infection and innate host responses to viral gene therapy vectors.Activation of the innate immune response against DENV in normal non-transformed human fibroblastsVaricella-zoster virus immediate-early protein 62 blocks interferon regulatory factor 3 (IRF3) phosphorylation at key serine residues: a novel mechanism of IRF3 inhibition among herpesvirusesInterferon regulatory factor 3-dependent pathways are critical for control of herpes simplex virus type 1 central nervous system infection.Trappin-2/elafin modulate innate immune responses of human endometrial epithelial cells to PolyI:CHerpes simplex virus glycoproteins gH/gL and gB bind Toll-like receptor 2, and soluble gH/gL is sufficient to activate NF-κBTrex1 regulates lysosomal biogenesis and interferon-independent activation of antiviral genesDistinct and overlapping roles of Nipah virus P gene products in modulating the human endothelial cell antiviral response.Lipopolysaccharide inhibits Sindbis virus-induced IP-10 release in human peripheral blood mononuclear cells.Type I and type III interferons drive redundant amplification loops to induce a transcriptional signature in influenza-infected airway epitheliaEarly induction of autophagy in human fibroblasts after infection with human cytomegalovirus or herpes simplex virus 1Innate and adaptive immune responses to herpes simplex virusThe epithelial αvβ3-integrin boosts the MYD88-dependent TLR2 signaling in response to viral and bacterial components.Functional Analysis of Chicken IRF7 in Response to dsRNA Analog Poly(I:C) by Integrating Overexpression and KnockdownType 1 IFN-independent activation of a subset of interferon stimulated genes in West Nile virus Eg101-infected mouse cells.Type I interferon production during herpes simplex virus infection is controlled by cell-type-specific viral recognition through Toll-like receptor 9, the mitochondrial antiviral signaling protein pathway, and novel recognition systems.Antibody-dependent enhancement of dengue virus infection inhibits RLR-mediated Type-I IFN-independent signalling through upregulation of cellular autophagy.Membrane Perturbation-Associated Ca2+ Signaling and Incoming Genome Sensing Are Required for the Host Response to Low-Level Enveloped Virus Particle EntryControl of TANK-binding kinase 1-mediated signaling by the gamma(1)34.5 protein of herpes simplex virus 1TANK-binding kinase-1 plays an important role during in vitro and in vivo type I IFN responses to DNA virus infections.A selective contribution of the RIG-I-like receptor pathway to type I interferon responses activated by cytosolic DNA.Ebola virus-like particles stimulate type I interferons and proinflammatory cytokine expression through the toll-like receptor and interferon signaling pathways.Recognition of herpes simplex viruses: toll-like receptors and beyondBunyaviruses and the type I interferon system.Membrane perturbation elicits an IRF3-dependent, interferon-independent antiviral response.Class A Scavenger Receptor-Mediated Double-Stranded RNA Internalization Is Independent of Innate Antiviral Signaling and Does Not Require Phosphatidylinositol 3-Kinase Activity.Type I interferon and NF-κB activation elicited by herpes simplex virus gH/gL via αvβ3 integrin in epithelial and neuronal cell lines.Innate Immune Mechanisms and Herpes Simplex Virus Infection and Disease.Sindbis virus induced phosphorylation of IRF3 in human embryonic kidney cells is not dependent on mTOR.Activation of the interferon response by human cytomegalovirus occurs via cytoplasmic double-stranded DNA but not glycoprotein B.Cell fusion-induced activation of interferon-stimulated genes is not required for restriction of a herpes simplex virus VP16/ICP0 mutant in heterokarya formed between permissive and restrictive cells.
P2860
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P2860
The IFN-independent response to virus particle entry provides a first line of antiviral defense that is independent of TLRs and retinoic acid-inducible gene I.
description
2006 nî lūn-bûn
@nan
2006年の論文
@ja
2006年学术文章
@wuu
2006年学术文章
@zh
2006年学术文章
@zh-cn
2006年学术文章
@zh-hans
2006年学术文章
@zh-my
2006年学术文章
@zh-sg
2006年學術文章
@yue
2006年學術文章
@zh-hant
name
The IFN-independent response t ...... etinoic acid-inducible gene I.
@en
The IFN-independent response t ...... etinoic acid-inducible gene I.
@nl
type
label
The IFN-independent response t ...... etinoic acid-inducible gene I.
@en
The IFN-independent response t ...... etinoic acid-inducible gene I.
@nl
prefLabel
The IFN-independent response t ...... etinoic acid-inducible gene I.
@en
The IFN-independent response t ...... etinoic acid-inducible gene I.
@nl
P2093
P1476
The IFN-independent response t ...... etinoic acid-inducible gene I.
@en
P2093
Derek T Cummings
Patrick Paladino
Ryan S Noyce
P304
P356
10.4049/JIMMUNOL.177.11.8008
P407
P577
2006-12-01T00:00:00Z