Respiratory syncytial virus infection prolongs methacholine-induced airway hyperresponsiveness in ovalbumin-sensitized mice. - Wikidata - wikimedia - TriplyDB

Respiratory syncytial virus infection prolongs methacholine-induced airway hyperresponsiveness in ovalbumin-sensitized mice.

Respiratory syncytial virus infection prolongs methacholine-induced airway hyperresponsiveness in ovalbumin-sensitized mice.

http://www.wikidata.org/entity/Q45751730

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Hyperresponsiveness to inhaled but not intravenous methacholine during acute respiratory syncytial virus infection in mice Mouse models of acute exacerbations of allergic asthma CD8+ TCR transgenic strains expressing public versus private TCR targeting the respiratory syncytial virus K(d)M2(82-90) epitope demonstrate similar functional profiles.Respiratory syncytial virus infection and G and/or SH protein expression contribute to substance P, which mediates inflammation and enhanced pulmonary disease in BALB/c mice.Inducible expression of inflammatory chemokines in respiratory syncytial virus-infected mice: role of MIP-1alpha in lung pathology.Respiratory syncytial virus (RSV) G glycoprotein is not necessary for vaccine-enhanced disease induced by immunization with formalin-inactivated RSV.Respiratory syncytial virus reverses airway hyperresponsiveness to methacholine in ovalbumin-sensitized mice Adjunct corticosteroids in children hospitalized with community-acquired pneumonia New immune pathways from chronic post-viral lung disease Toward primary prevention of asthma. Reviewing the evidence for early-life respiratory viral infections as modifiable risk factors to prevent childhood asthma Respiratory syncytial virus and reactive airway disease Krüppel-like factor 6 regulates transforming growth factor-β gene expression during human respiratory syncytial virus infection.Differential immune responses and pulmonary pathophysiology are induced by two different strains of respiratory syncytial virus.Suppression of airway hyperresponsiveness induced by ovalbumin sensitisation and RSV infection with Y-27632, a Rho kinase inhibitor.Effect of respiratory syncytial virus infection on regulated on activation, normal T-cells expressed and secreted production in a murine model of asthma.Pathogenesis of respiratory syncytial virus infection in the murine model Epidemiologic, experimental, and clinical links between respiratory syncytial virus infection and asthma.Respiratory syncytial virus infection: from biology to therapy: a perspective.Respiratory syncytial virus infection modifies and accelerates pulmonary disease via DC activation and migration.IkappaB kinase is a critical regulator of chemokine expression and lung inflammation in respiratory syncytial virus infection.A chimeric A2 strain of respiratory syncytial virus (RSV) with the fusion protein of RSV strain line 19 exhibits enhanced viral load, mucus, and airway dysfunction Respiratory syncytial virus infection activates IL-13-producing group 2 innate lymphoid cells through thymic stromal lymphopoietin.Cell cycle arrest by transforming growth factor beta1 enhances replication of respiratory syncytial virus in lung epithelial cells Bugs and asthma: a different disease?IL-17A inhibits airway reactivity induced by respiratory syncytial virus infection during allergic airway inflammation.Influenza A virus infection and cigarette smoke impair bronchodilator responsiveness to β-adrenoceptor agonists in mouse lung Anti-respiratory syncytial virus (RSV) neutralizing antibody decreases lung inflammation, airway obstruction, and airway hyperresponsiveness in a murine RSV model Respiratory syncytial virus--a comprehensive review.The relationship between respiratory syncytial virus and asthma.Neonatal respiratory syncytial virus infection has an effect on lung inflammation and the CD4(+) CD25(+) T cell subpopulation during ovalbumin sensitization in adult mice.Respiratory syncytial virus infection differentiates airway dysfunction in the central and peripheral airways in OVA-sensitized mice.Cooperative effects of rhinovirus and TNF-{alpha} on airway epithelial cell chemokine expression.Dual effects of respiratory syncytial virus infections on airway inflammation by regulation of Th17/Treg responses in ovalbumin-challenged mice.Development of atopy by severe paramyxoviral infection in a mouse model.Heat Shock-Related Protein 20 Peptide Decreases Human Airway Constriction Downstream of β2-Adrenergic Receptor.Respiratory syncytial virus induces airway insensitivity to beta-agonists in BALB/c mice.The failure of interleukin-10-deficient mice to develop airway hyperresponsiveness is overcome by respiratory syncytial virus infection in allergen-sensitized/challenged mice.Respiratory syncytial virus infection reversed anti-asthma effect of neonatal Bacillus Calmette-Guerin vaccination in BALB/c mice.Selective cyclooxygenase-1 and -2 inhibitors each increase allergic inflammation and airway hyperresponsiveness in mice.Effects of primary and secondary low-grade respiratory syncytial virus infections in a murine model of asthma.

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P2860

Respiratory syncytial virus infection prolongs methacholine-induced airway hyperresponsiveness in ovalbumin-sensitized mice.

http://www.wikidata.org/entity/Q45751730

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Journal of Medical Virology

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P2860

Role of T lymphocyte subsets in the pathogenesis of primary infection and rechallenge with respiratory syncytial virus in mice.

Rhinovirus upper respiratory infection increases airway hyperreactivity and late asthmatic reactions.

Transfer of allergic airway responses with antigen-primed CD4+ but not CD8+ T cells in brown Norway rats.

Allergen-induced bronchial hyperreactivity and eosinophilic inflammation occur in the absence of IgE in a mouse model of asthma.

Allergic airway sensitization induces T cell activation but not airway hyperresponsiveness in B cell-deficient mice.

Interleukin 12 inhibits antigen-induced airway hyperresponsiveness, inflammation, and Th2 cytokine expression in mice.

Interleukin 4, but not interleukin 5 or eosinophils, is required in a murine model of acute airway hyperreactivity.

Community study of role of viral infections in exacerbations of asthma in 9-11 year old children.

Inhibition of T cell costimulation abrogates airway hyperresponsiveness in a murine model

Respiratory syncytial virus infection results in airway hyperresponsiveness and enhanced airway sensitization to allergen.

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