Inhibition of T cell costimulation abrogates airway hyperresponsiveness in a murine model
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The BTNL2 gene and sarcoidosis susceptibility in African Americans and WhitesContribution of nitric oxide synthases 1, 2, and 3 to airway hyperresponsiveness and inflammation in a murine model of asthmaPrevention of house dust mite induced allergic airways disease in mice through immune toleranceImmunomodulation by interference with co-stimulatory molecules: therapeutic perspectives in asthma.Immune mechanisms of childhood asthmaThe B7/CD28/CTLA4 T-cell activation pathway. Implications for inflammatory lung disease.CTLA4-Ig: a novel immunosuppressive agent.Costimulation blockade inhibits allergic sensitization but does not affect established allergy in a murine model of grass pollen allergy.Is Th1 the solution for Th2 in asthma?Soluble CD86 protein in serum samples of patients with asthma.Biology and therapeutic potential of the interleukin-4/interleukin-13 signaling pathway in asthma.CD48 is critically involved in allergic eosinophilic airway inflammation.Controlled release strategies for modulating immune responses to promote tissue regeneration(R)-albuterol decreases immune responses: role of activated T cellsRegulation of cytotoxic T lymphocyte antigen 4 by cyclic AMP.The costimulatory molecule SLAM is critical for pulmonary allergic responses.CD28 and ICOS play complementary non-overlapping roles in the development of Th2 immunity in vivo.Cathepsin S activity regulates antigen presentation and immunity.Intrinsic and extrinsic control of peripheral T-cell tolerance by costimulatory molecules of the CD28/ B7 familyExperimental advances in understanding allergic airway inflammation.PKC-theta-mediated signal delivery from the TCR/CD28 surface receptors.CTLA4-IgG reverses asthma manifestations in a mild but not in a more "severe" ongoing murine model.Divergence in NK cell and cyclic AMP regulation of T cell CD40L expression in asthmatic subjects.Respiratory syncytial virus infection prolongs methacholine-induced airway hyperresponsiveness in ovalbumin-sensitized mice.Toll-like receptor 4 or 2 agonists decrease allergic inflammation.Alterations in lung collectins in an adaptive allergic immune response.Requirement of CD28-CD86 costimulation for allergen-specific T cell proliferation and cytokine expression.The costimulatory molecules CD80, CD86 and OX40L are up-regulated in Aspergillus fumigatus sensitized mice.Effects of cyclosporin A and dinactin on T-cell proliferation, interleukin-5 production, and murine pulmonary inflammation.CD28 and CTLA4 coordinately regulate airway inflammatory cell recruitment and T-helper cell differentiation after inhaled allergenCD28 interactions with either CD80 or CD86 are sufficient to induce allergic airway inflammation in mice
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P2860
Inhibition of T cell costimulation abrogates airway hyperresponsiveness in a murine model
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on December 1996
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Inhibition of T cell costimulation abrogates airway hyperresponsiveness in a murine model
@en
Inhibition of T cell costimula ...... ponsiveness in a murine model.
@nl
type
label
Inhibition of T cell costimulation abrogates airway hyperresponsiveness in a murine model
@en
Inhibition of T cell costimula ...... ponsiveness in a murine model.
@nl
prefLabel
Inhibition of T cell costimulation abrogates airway hyperresponsiveness in a murine model
@en
Inhibition of T cell costimula ...... ponsiveness in a murine model.
@nl
P2093
P2860
P356
P1476
Inhibition of T cell costimulation abrogates airway hyperresponsiveness in a murine model
@en
P2093
D C Christiani
D L Perkins
G T De Sanctis
M Cernadas
P2860
P304
P356
10.1172/JCI119093
P407
P577
1996-12-01T00:00:00Z