Strand bias in oligonucleotide-mediated dystrophin gene editing.
about
Oligonucleotide-directed mutagenesis for precision gene editingGenetic correction of splice site mutation in purified and enriched myoblasts isolated from mdx5cv mice.Gene and cell-mediated therapies for muscular dystrophy.Regulation of Gene Editing Activity Directed by Single-Stranded Oligonucleotides and CRISPR/Cas9 Systems.High throughput screening in duchenne muscular dystrophy: from drug discovery to functional genomicsOligonucleotide transformation of yeast reveals mismatch repair complexes to be differentially active on DNA replication strands.Gene therapy progress and prospects: targeted gene repair.Proliferation of genetically modified human cells on electrospun nanofiber scaffolds.Electrospun fiber membranes enable proliferation of genetically modified cells.Read-through compound 13 restores dystrophin expression and improves muscle function in the mdx mouse model for Duchenne muscular dystrophyProgress and prospects: targeted gene alteration (TGA).Molecular-targeted therapy for Duchenne muscular dystrophy: progress and potential.A high-content, high-throughput siRNA screen identifies cyclin D2 as a potent regulator of muscle progenitor cell fusion and a target to enhance muscle regeneration.Gene therapy for muscular dystrophy: current progress and future prospects.Progress and prospects: oligonucleotide-directed gene modification in mouse embryonic stem cells: a route to therapeutic application.An update on targeted gene repair in mammalian cells: methods and mechanismsOligonucleotide-directed gene-editing technology: mechanisms and future prospects.New developments in the use of gene therapy to treat Duchenne muscular dystrophy.Emerging gene editing strategies for Duchenne muscular dystrophy targeting stem cells.Generation of a mouse mutant by oligonucleotide-mediated gene modification in ES cells.Enhanced gene repair mediated by methyl-CpG-modified single-stranded oligonucleotidesDNA breakage associated with targeted gene alteration directed by DNA oligonucleotides.Homologous recombination is required for AAV-mediated gene targeting.Strand bias influences the mechanism of gene editing directed by single-stranded DNA oligonucleotides.A Standard Methodology to Examine On-site Mutagenicity As a Function of Point Mutation Repair Catalyzed by CRISPR/Cas9 and SsODN in Human CellsEx vivo gene editing of the dystrophin gene in muscle stem cells mediated by peptide nucleic acid single stranded oligodeoxynucleotides induces stable expression of dystrophin in a mouse model for Duchenne muscular dystrophy.Genetic modification through oligonucleotide-mediated mutagenesis. A GMO regulatory challenge?
P2860
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P2860
Strand bias in oligonucleotide-mediated dystrophin gene editing.
description
2004 nî lūn-bûn
@nan
2004年の論文
@ja
2004年学术文章
@wuu
2004年学术文章
@zh-cn
2004年学术文章
@zh-hans
2004年学术文章
@zh-my
2004年学术文章
@zh-sg
2004年學術文章
@yue
2004年學術文章
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2004年學術文章
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name
Strand bias in oligonucleotide-mediated dystrophin gene editing.
@en
Strand bias in oligonucleotide-mediated dystrophin gene editing.
@nl
type
label
Strand bias in oligonucleotide-mediated dystrophin gene editing.
@en
Strand bias in oligonucleotide-mediated dystrophin gene editing.
@nl
prefLabel
Strand bias in oligonucleotide-mediated dystrophin gene editing.
@en
Strand bias in oligonucleotide-mediated dystrophin gene editing.
@nl
P2093
P356
P1476
Strand bias in oligonucleotide-mediated dystrophin gene editing.
@en
P2093
Carmen Bertoni
Glenn E Morris
Thomas A Rando
P304
P356
10.1093/HMG/DDI020
P577
2004-11-24T00:00:00Z