Reverse rate dependency is an intrinsic property of canine cardiac preparations. - Wikidata - wikimedia - TriplyDB

Reverse rate dependency is an intrinsic property of canine cardiac preparations.

Reverse rate dependency is an intrinsic property of canine cardiac preparations.

http://www.wikidata.org/entity/Q45952596

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Determinants of beat-to-beat variability of repolarization duration in the canine ventricular myocyte: a computational analysis Comprehensive analyses of ventricular myocyte models identify targets exhibiting favorable rate dependence Late sodium current contributes to the reverse rate-dependent effect of IKr inhibition on ventricular repolarization.Restitution slope is principally determined by steady-state action potential duration.Local control of β-adrenergic stimulation: Effects on ventricular myocyte electrophysiology and Ca(2+)-transient.Comparison of the IKr blockers moxifloxacin, dofetilide and E-4031 in five screening models of pro-arrhythmia reveals lack of specificity of isolated cardiomyocytes Role of action potential configuration and the contribution of C²⁺a and K⁺ currents to isoprenaline-induced changes in canine ventricular cells.Nonlinear dynamics in cardiology.Arrhythmic risk biomarkers for the assessment of drug cardiotoxicity: from experiments to computer simulations.Geometrical considerations in cardiac electrophysiology and arrhythmogenesis.Role of abnormal repolarization in the mechanism of cardiac arrhythmia.Ca2+-activated Cl- current is antiarrhythmic by reducing both spatial and temporal heterogeneity of cardiac repolarization.Action potential contour contributes to species differences in repolarization response to β-adrenergic stimulation.In Vitro and In Silico Risk Assessment in Acquired Long QT Syndrome: The Devil Is in the Details.Contribution of ion currents to beat-to-beat variability of action potential duration in canine ventricular myocytes.Rate-Dependent Role of IKur in Human Atrial Repolarization and Atrial Fibrillation Maintenance.Differential roles of two delayed rectifier potassium currents in regulation of ventricular action potential duration and arrhythmia susceptibility.Beat-to-beat variability of cardiac action potential duration: underlying mechanism and clinical implications.[Ca²⁺] i-induced augmentation of the inward rectifier potassium current (IK1) in canine and human ventricular myocardium.Slow Recovery of Excitability Increases Ventricular Fibrillation Risk as Identified by Emulation

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Reverse rate dependency is an intrinsic property of canine cardiac preparations.

http://www.wikidata.org/entity/Q45952596

description

2009 nî lūn-bûn

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2009年の論文

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2009年学术文章

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2009年学术文章

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2009年学术文章

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2009年学术文章

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2009年学术文章

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2009年学术文章

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2009年學術文章

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2009年學術文章

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name

Reverse rate dependency is an intrinsic property of canine cardiac preparations.

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Reverse rate dependency is an intrinsic property of canine cardiac preparations.

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type

label

Reverse rate dependency is an intrinsic property of canine cardiac preparations.

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Reverse rate dependency is an intrinsic property of canine cardiac preparations.

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prefLabel

Reverse rate dependency is an intrinsic property of canine cardiac preparations.

@en

Reverse rate dependency is an intrinsic property of canine cardiac preparations.

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Cardiovascular Research

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Reverse rate dependency is an intrinsic property of canine cardiac preparations

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András Varró

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Balázs Horváth

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Gábor Harmati

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János Magyar

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László Bárándi

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László Virág

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Norbert Szentandrássy

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Péter P Nánási

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Stefano Marangoni

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Tamás Bányász

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P2860

Measurement of current-voltage relations in the membrane of the giant axon of Loligo

Rate dependency of delayed rectifier currents during the guinea-pig ventricular action potential

Early afterdepolarizations in cardiac myocytes: mechanism and rate dependence.

Kinetics of onset of rate-dependent effects of Class I antiarrhythmic drugs are important in determining their effects on refractoriness in guinea-pig ventricle, and provide a theoretical basis for their subclassification.

Novel antiarrhythmic compounds with combined class IB and class III mode of action.

The combined electrophysiological effects of lignocaine and sotalol in canine isolated cardiac Purkinje fibres are rate-dependent

Veratridine modifies open sodium channels

Voltage-dependent gating of veratridine-modified Na channels.

Pathophysiology and pharmacology of the cardiac "late sodium current.".

Time- and voltage-dependent interactions of antiarrhythmic drugs with cardiac sodium channels.

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10.1093/CVR/CVP213

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2

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84

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26320565

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19556280

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